Exposure to ambient particulate matter induces a NASH-like phenotype and impairs hepatic glucose metabolism in an animal model. - Wikidata - awgldk - TriplyDB

Exposure to ambient particulate matter induces a NASH-like phenotype and impairs hepatic glucose metabolism in an animal model.

Exposure to ambient particulate matter induces a NASH-like phenotype and impairs hepatic glucose metabolism in an animal model.

http://www.wikidata.org/entity/Q36481144

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Influencing Factors of Thermogenic Adipose Tissue Activity Insulin resistance and environmental pollutants: experimental evidence and future perspectives Petroleum coke in the urban environment: a review of potential health effects YKL-40 is a Protective Biomarker for Fatty Liver in World Trade Center Particulate Matter-Exposed Firefighters Repeated ozone exposure exacerbates insulin resistance and activates innate immune response in genetically susceptible mice The role of air pollutants in initiating liver disease Exaggerated effects of particulate matter air pollution in genetic type II diabetes mellitus.Environmental pollution: a tangible risk for NAFLD pathogenesis Extreme Air Pollution Conditions Adversely Affect Blood Pressure and Insulin Resistance: The Air Pollution and Cardiometabolic Disease Study.Association of Long-Term Near-Highway Exposure to Ultrafine Particles with Cardiovascular Diseases, Diabetes and Hypertension Environmental endocrine disruption of energy metabolism and cardiovascular risk.Modeling progressive non-alcoholic fatty liver disease in the laboratory mouse.Repeated intratracheal instillation of PM10 induces lipid reshaping in lung parenchyma and in extra-pulmonary tissues Role of MicroRNAs in Hepatocellular Carcinoma Central IKKβ inhibition prevents air pollution mediated peripheral inflammation and exaggeration of type II diabetes.Air pollution as a risk factor for type 2 diabetes.Diabetes mellitus is associated with hepatocellular carcinoma: a retrospective case-control study in hepatitis endemic area.Is particle pollution in outdoor air associated with metabolic control in type 2 diabetes?Air Pollution and Serum Glucose Levels: A Population-Based Study.Air-Pollution and Cardiometabolic Diseases (AIRCMD): a prospective study investigating the impact of air pollution exposure and propensity for type II diabetes Association of Roadway Proximity with Fasting Plasma Glucose and Metabolic Risk Factors for Cardiovascular Disease in a Cross-Sectional Study of Cardiac Catheterization Patients.Air pollution and type 2 diabetes: mechanistic insights In Vivo Exposures to Particulate Matter Collected from Saudi Arabia or Nickel Chloride Display Similar Dysregulation of Metabolic Syndrome Genes.Inhaled ozone (O3)-induces changes in serum metabolomic and liver transcriptomic profiles in rats.Ambient Air Pollutants Have Adverse Effects on Insulin and Glucose Homeostasis in Mexican Americans Exposure to fine airborne particulate matter induces macrophage infiltration, unfolded protein response, and lipid deposition in white adipose tissue Sparstolonin B attenuates early liver inflammation in experimental NASH by modulating TLR4 trafficking in lipid rafts via NADPH oxidase activation A healthier approach to clinical trials evaluating resveratrol for primary prevention of age‐related diseases in healthy populations.Exposure to fine airborne particulate matters induces hepatic fibrosis in murine models.Plasma selenium levels and nonalcoholic fatty liver disease in Chinese adults: a cross-sectional analysis.Air pollution-mediated susceptibility to inflammation and insulin resistance: influence of CCR2 pathways in mice.Association between soil heavy metals and fatty liver disease in men in Taiwan: a cross sectional study The hijacking of cellular signaling and the diabetes epidemic: mechanisms of environmental disruption of insulin action and glucose homeostasis.Particulate Air pollution mediated effects on insulin resistance in mice are independent of CCR2 Toxicological and epidemiological studies of cardiovascular effects of ambient air fine particulate matter (PM2.5) and its chemical components: coherence and public health implications.Ambient air pollution: an emerging risk factor for diabetes mellitus.Endocrine disruptors and obesity.Traffic-related air pollution associations with cytokeratin-18, a marker of hepatocellular apoptosis, in an overweight and obese paediatric population.Polluted Pathways: Mechanisms of Metabolic Disruption by Endocrine Disrupting Chemicals.Effects of air pollution exposure on glucose metabolism in Los Angeles minority children.

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Exposure to ambient particulate matter induces a NASH-like phenotype and impairs hepatic glucose metabolism in an animal model.

http://www.wikidata.org/entity/Q36481144

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Exposure to ambient particulat ...... metabolism in an animal model.

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Exposure to ambient particulat ...... metabolism in an animal model.

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J.JHEP.2012.08.009

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Journal of Hepatology

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Exposure to ambient particulat ...... metabolism in an animal model.

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Aixia Wang

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Chunbin Zhang

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Kezhong Zhang

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Lawrence I Grossman

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Lung Chi Chen

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Qinghua Sun

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Sanjay Rajagopalan

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Xiaohua Xu

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Xuebao Zhang

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Ze Zheng

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P2860

Protein kinase C Theta inhibits insulin signaling by phosphorylating IRS1 at Ser(1101)

Negative regulation of Toll-like receptor 4 signaling by the Toll-like receptor homolog RP105

Signal transduction by the c-Jun N-terminal kinase (JNK)--from inflammation to development

Introduction to NF-kappaB: players, pathways, perspectives

SP600125, an anthrapyrazolone inhibitor of Jun N-terminal kinase

Airborne particulate matter selectively activates endoplasmic reticulum stress response in the lung and liver tissues

Traffic-related air pollution and incident type 2 diabetes: results from the SALIA cohort study

Steatohepatitis: a tale of two "hits"?

The ratio of aspartate aminotransferase to alanine aminotransferase: potential value in differentiating nonalcoholic steatohepatitis from alcoholic liver disease.

Kupffer cell activation by ambient air particulate matter exposure may exacerbate non-alcoholic fatty liver disease.

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