Cyclin D1 overexpression supports stable EBV infection in nasopharyngeal epithelial cells - Wikidata - awgldk - TriplyDB

Cyclin D1 overexpression supports stable EBV infection in nasopharyngeal epithelial cells

Cyclin D1 overexpression supports stable EBV infection in nasopharyngeal epithelial cells

http://www.wikidata.org/entity/Q36483613

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Regulation of the latent-lytic switch in Epstein-Barr virus miR-31 is consistently inactivated in EBV-associated nasopharyngeal carcinoma and contributes to its tumorigenesis PAK5-Egr1-MMP2 signaling controls the migration and invasion in breast cancer cell.Enhanced IL-6/IL-6R signaling promotes growth and malignant properties in EBV-infected premalignant and cancerous nasopharyngeal epithelial cells.Efficient immortalization of primary nasopharyngeal epithelial cells for EBV infection study.Telomerase activity impacts on Epstein-Barr virus infection of AGS cells.Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial Cells The Epstein-Barr Virus BART miRNA Cluster of the M81 Strain Modulates Multiple Functions in Primary B Cells.Overexpression of PIN1 Enhances Cancer Growth and Aggressiveness with Cyclin D1 Induction in EBV-Associated Nasopharyngeal Carcinoma.Epstein-Barr virus mRNA profiles and viral DNA methylation status in nasopharyngeal brushings from nasopharyngeal carcinoma patients reflect tumor origin Highly Efficient CRISPR/Cas9-Mediated Cloning and Functional Characterization of Gastric Cancer-Derived Epstein-Barr Virus Strains.Extremely stringent activation of p16INK4a prevents immortalization of uterine cervical epithelial cells without human papillomavirus oncogene expression.Modes of infection and oncogenesis by the Epstein-Barr virus.Epstein-Barr virus infection and persistence in nasopharyngeal epithelial cells.The role of Epstein-Barr virus in epithelial malignancies.The interplay of host genetic factors and Epstein-Barr virus in the development of nasopharyngeal carcinoma.The role of Epstein-Barr virus infection in the pathogenesis of nasopharyngeal carcinoma.Restricted TET2 Expression in Germinal Center Type B Cells Promotes Stringent Epstein-Barr Virus Latency.Epstein-Barr virus: more than 50 years old and still providing surprises.The Long and Complicated Relationship between Epstein-Barr Virus and Epithelial Cells.The interaction between human papillomavirus and other viruses.Epstein-Barr virus infection and nasopharyngeal carcinoma.Epstein-Barr Virus Rta-Mediated Accumulation of DNA Methylation Interferes with CTCF Binding in both Host and Viral Genomes.Pathogenesis: Infections causing gastric cancer.A new nasal cavity nursing methods application in patients with mechanical ventilation.Latent Membrane Protein 1 Is a Novel Determinant of Epstein-Barr Virus Genome Persistence and Reactivation.Epstein-Barr Virus Hijacks DNA Damage Response Transducers to Orchestrate Its Life Cycle.Modulation of the Tumor Microenvironment by Epstein-Barr virus Latent Membrane Protein-1 in Nasopharyngeal Carcinoma.EBV Infection and Glucose Metabolism in Nasopharyngeal Carcinoma.Epstein-Barr Virus Gene BARF1 Expression is Regulated by the Epithelial Differentiation Factor ΔNp63α in Undifferentiated Nasopharyngeal Carcinoma.New Insights from Elucidating the Role of LMP1 in Nasopharyngeal Carcinoma.Downregulation of PAK5 inhibits glioma cell migration and invasion potentially through the PAK5-Egr1-MMP2 signaling pathway.Preclinical evaluation of ribociclib and its synergistic effect in combination with alpelisib in non-keratinizing nasopharyngeal carcinoma.EBV based cancer prevention and therapy in nasopharyngeal carcinoma.Epstein-Barr virus and nasopharyngeal carcinoma Role of Epstein-Barr Virus in the Pathogenesis of Head and Neck Cancers and Its Potential as an Immunotherapeutic Target Establishment and characterization of new tumor xenografts and cancer cell lines from EBV-positive nasopharyngeal carcinoma

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Cyclin D1 overexpression supports stable EBV infection in nasopharyngeal epithelial cells

http://www.wikidata.org/entity/Q36483613

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2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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2012年學術文章

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name

Cyclin D1 overexpression supports stable EBV infection in nasopharyngeal epithelial cells

@ast

Cyclin D1 overexpression supports stable EBV infection in nasopharyngeal epithelial cells

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type

label

Cyclin D1 overexpression supports stable EBV infection in nasopharyngeal epithelial cells

@ast

Cyclin D1 overexpression supports stable EBV infection in nasopharyngeal epithelial cells

@en

prefLabel

Cyclin D1 overexpression supports stable EBV infection in nasopharyngeal epithelial cells

@ast

Cyclin D1 overexpression supports stable EBV infection in nasopharyngeal epithelial cells

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Cyclin D1 overexpression supports stable EBV infection in nasopharyngeal epithelial cells

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Annie Lai-Man Cheung

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Chi Man Tsang

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Honglin Chen

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Jaap Michiel Middeldorp

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Ka Fai To

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Kenzo Takada

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Kwok Wai Lo

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Maria Li Lung

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Musheng Zeng

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Pok Man Hau

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P2860

Deciphering the molecular genetic basis of NPC through molecular, cytogenetic, and epigenetic approaches

Epstein-Barr virus: 40 years on

Influenza A virus replication induces cell cycle arrest in G0/G1 phase

Murine coronavirus replication induces cell cycle arrest in G0/G1 phase.

Cyclin D1 downregulation is important for permanent cell cycle exit and initiation of differentiation induced by anchorage-deprivation in human keratinocytes.

Epstein-Barr virus nuclear antigens 3C and 3A maintain lymphoblastoid cell growth by repressing p16INK4A and p14ARF expression.

Terminal differentiation into plasma cells initiates the replicative cycle of Epstein-Barr virus in vivo.

Epstein-Barr virus infection in humans: from harmless to life endangering virus-lymphocyte interactions.

Epstein-Barr virus in the pathogenesis of NPC.

Genetic and epigenetic changes in nasopharyngeal carcinoma.

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