Chronic Treatment with the AMPK Agonist AICAR Prevents Skeletal Muscle Pathology but Fails to Improve Clinical Outcome in a Mouse Model of Severe Spinal Muscular Atrophy.
about
Functional characterization of AMP-activated protein kinase signaling in tumorigenesis.Exercise as a Positive Modulator of Brain Function.Chronic 5-Aminoimidazole-4-Carboxamide-1-β-d-Ribofuranoside Treatment Induces Phenotypic Changes in Skeletal Muscle, but Does Not Improve Disease Outcomes in the R6/2 Mouse Model of Huntington's Disease.
P2860
Chronic Treatment with the AMPK Agonist AICAR Prevents Skeletal Muscle Pathology but Fails to Improve Clinical Outcome in a Mouse Model of Severe Spinal Muscular Atrophy.
description
2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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name
Chronic Treatment with the AMP ...... evere Spinal Muscular Atrophy.
@ast
Chronic Treatment with the AMP ...... evere Spinal Muscular Atrophy.
@en
type
label
Chronic Treatment with the AMP ...... evere Spinal Muscular Atrophy.
@ast
Chronic Treatment with the AMP ...... evere Spinal Muscular Atrophy.
@en
prefLabel
Chronic Treatment with the AMP ...... evere Spinal Muscular Atrophy.
@ast
Chronic Treatment with the AMP ...... evere Spinal Muscular Atrophy.
@en
P2093
P2860
P1433
P1476
Chronic Treatment with the AMP ...... evere Spinal Muscular Atrophy.
@en
P2093
Clàudia Cerveró
Lídia Piedrafita
Neus Montull
Olga Tarabal
P2860
P2888
P304
P356
10.1007/S13311-015-0399-X
P577
2015-11-18T00:00:00Z