about
Neurotoxic species of misfolded SOD1G93A recognized by antibodies against the P2X4 subunit of the ATP receptor accumulate in damaged neurons of transgenic animal models of amyotrophic lateral sclerosis.Accumulation of misfolded SOD1 in dorsal root ganglion degenerating proprioceptive sensory neurons of transgenic mice with amyotrophic lateral sclerosisIncreased intramuscular nerve branching and inhibition of programmed cell death of chick embryo motoneurons by immunoglobulins from patients with motoneuron disease.Antibodies against c-Jun N-terminal peptide cross-react with neo-epitopes emerging after caspase-mediated proteolysis during apoptosis.Chronic Treatment with the AMPK Agonist AICAR Prevents Skeletal Muscle Pathology but Fails to Improve Clinical Outcome in a Mouse Model of Severe Spinal Muscular Atrophy.Caspase-independent type III programmed cell death in chronic lymphocytic leukemia: the key role of the F-actin cytoskeleton.7-Bromoindirubin-3'-oxime uncovers a serine protease-mediated paradigm of necrotic cell death.Survival and death of mature avian motoneurons in organotypic slice culture: trophic requirements for survival and different types of degeneration.Drp1 mediates caspase-independent type III cell death in normal and leukemic cellsPhylogenetic polymorphism on lectin binding to junctional and non-junctional basal lamina at the vertebrate neuromuscular junction.Receptors to agglutinin from Dolichus biflorus (DBA) at the synaptic basal lamina of rat neuromuscular junction. A histochemical study during development and denervation.Lithium prevents excitotoxic cell death of motoneurons in organotypic slice cultures of spinal cord.Neuregulin-1 is concentrated in the postsynaptic subsurface cistern of C-bouton inputs to α-motoneurons and altered during motoneuron diseases.Chronic treatment with lithium does not improve neuromuscular phenotype in a mouse model of severe spinal muscular atrophy.Rescue of developing spinal motoneurons from programmed cell death by the GABA(A) agonist muscimol acts by blockade of neuromuscular activity and increased intramuscular nerve branching.Calcitonin gene-related peptide in rat spinal cord motoneurons: subcellular distribution and changes induced by axotomy.Protein retention in the endoplasmic reticulum, blockade of programmed cell death and autophagy selectively occur in spinal cord motoneurons after glutamate receptor-mediated injury.Adverse effects of a SOD1-peptide immunotherapy on SOD1 G93A mouse slow model of amyotrophic lateral sclerosis.Excitotoxic motoneuron disease in chick embryo evolves with autophagic neurodegeneration and deregulation of neuromuscular innervation.Binding of beta-bungarotoxin to Torpedo electric organ synaptosomes. A high resolution autoradiographic study.Mechanisms involved in spinal cord central synapse loss in a mouse model of spinal muscular atrophy.Structural changes at pure cholinergic synaptosomes during the transmitter release induced by A-23187 inTorpedo marmorataDefective Neuromuscular Junction Organization and Postnatal Myogenesis in Mice With Severe Spinal Muscular AtrophyIntramuscular nerve sprouting induced by CNTF is associated with increases in CGRP content in mouse motor nerve terminalsEvidence for calcium regulation of spinal cord motoneuron death in the chick embryo in vivoTreatment with digestive agents reveals several glycoconjugates specifically associated with rat neuromuscular junctionAbsence of histochemical immunoreactivity to calcitonin gene-related peptide (CGRP) in spinal cord motoneurons from (+)-tubocurarine-treated chick embryosThe carbohydrate N-acetylglucosamine is involved in the guidance of neurites from chick ciliary ganglion neurons through the extracellular matrix of rat skeletal muscle fiberEffects of ouabain and electrical stimulation on the fine structure of nerve endings in the electric organ of Torpedo marmorataPrevention by lamotrigine, MK-801 and N omega-nitro-L-arginine methyl ester of motoneuron cell death after neonatal axotomyStrong P2X4 purinergic receptor-like immunoreactivity is selectively associated with degenerating neurons in transgenic rodent models of amyotrophic lateral sclerosisIonic dependence of adenosine uptake by isolated nerve endings from Torpedo electric organ
P50
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Josep E Esquerda
@ast
Josep E Esquerda
@en
Josep E Esquerda
@es
Josep E Esquerda
@nl
type
label
Josep E Esquerda
@ast
Josep E Esquerda
@en
Josep E Esquerda
@es
Josep E Esquerda
@nl
prefLabel
Josep E Esquerda
@ast
Josep E Esquerda
@en
Josep E Esquerda
@es
Josep E Esquerda
@nl
P106
P1153
7004174985
P21
P31
P496
0000-0003-1413-2103