Starvation after AgRP neuron ablation is independent of melanocortin signaling. - Wikidata - awgldk - TriplyDB

Starvation after AgRP neuron ablation is independent of melanocortin signaling.

Starvation after AgRP neuron ablation is independent of melanocortin signaling.

http://www.wikidata.org/entity/Q36497492

about

60 YEARS OF POMC: Regulation of feeding and energy homeostasis by α-MSH Plasticity of the Melanocortin System: Determinants and Possible Consequences on Food Intake Melanocortin neurons: Multiple routes to regulation of metabolism.Optogenetic dissection of neural circuits underlying emotional valence and motivated behaviors.Arcuate nucleus proopiomelanocortin neurons mediate the acute anorectic actions of leukemia inhibitory factor via gp130.Neuronal control of peripheral insulin sensitivity and glucose metabolism Minireview: Metabolism of female reproduction: regulatory mechanisms and clinical implications The tyrosine kinase receptor Tyro3 enhances lifespan and neuropeptide Y (Npy) neuron survival in the mouse anorexia (anx) mutation.AGRP neurons are sufficient to orchestrate feeding behavior rapidly and without training.Ablation of Sim1 neurons causes obesity through hyperphagia and reduced energy expenditure.Hypothalamic survival circuits: blueprints for purposive behaviors Loss of agouti-related peptide does not significantly impact the phenotype of murine POMC deficiency.The use of animal models to decipher physiological and neurobiological alterations of anorexia nervosa patients.Ablation of neurons expressing agouti-related protein, but not melanin concentrating hormone, in leptin-deficient mice restores metabolic functions and fertility.Agouti-related protein segments outside of the receptor binding core are required for enhanced short- and long-term feeding stimulation The melanocortin-4 receptor: physiology, pharmacology, and pathophysiology.Hypothalamic AgRP-neurons control peripheral substrate utilization and nutrient partitioning Arcuate AgRP neurons and the regulation of energy balance Action of Neurotransmitter: A Key to Unlock the AgRP Neuron Feeding Circuit.RETRACTED: Glucose regulates the effects of leptin on hypothalamic POMC neurons.New inducible genetic method reveals critical roles of GABA in the control of feeding and metabolism.Revisiting the Ventral Medial Nucleus of the Hypothalamus: The Roles of SF-1 Neurons in Energy Homeostasis.Gad1 mRNA as a reliable indicator of altered GABA release from orexigenic neurons in the hypothalamus.Integration of sensory information via central thermoregulatory leptin targets Ablation of neurons expressing agouti-related protein activates fos and gliosis in postsynaptic target regions.Coordinated regulation of hepatic energy stores by leptin and hypothalamic agouti-related protein Cross-talk between reproduction and energy homeostasis: central impact of estrogens, leptin and kisspeptin signaling The membrane estrogen receptor ligand STX rapidly enhances GABAergic signaling in NPY/AgRP neurons: role in mediating the anorexigenic effects of 17β-estradiol.The role of the Agouti-Related Protein in energy balance regulation.Hyperphagia and obesity produced by arcuate injection of NPY-saporin do not require upregulation of lateral hypothalamic orexigenic peptide genes Loss of GABAergic signaling by AgRP neurons to the parabrachial nucleus leads to starvation Deciphering a neuronal circuit that mediates appetite.GABAergic signaling by AgRP neurons prevents anorexia via a melanocortin-independent mechanism.Expanding neurotransmitters in the hypothalamic neurocircuitry for energy balance regulation.Limitations in anti-obesity drug development: the critical role of hunger-promoting neurons.Metabolic transceivers: in tune with the central melanocortin system.Unraveling the central proopiomelanocortin neural circuits.Molecular and cellular regulation of hypothalamic melanocortin neurons controlling food intake and energy metabolism.Pro-Opiomelanocortin (POMC) Neurones, POMC-Derived Peptides, Melanocortin Receptors and Obesity: How Understanding of this System has Changed Over the Last Decade.Making sense of the sensory regulation of hunger neurons.

P2860

Q26768703-64DAA53E-FEB8-4B3F-8871-8C4B52B9297B Q26781434-A7539D61-4B31-4A2E-A916-016618360D10 Q30234481-EE6C8108-14E2-4A49-A718-6EAE125CDEDB Q30434815-D0E8326D-05EF-4C47-A465-19C4CA232879 Q33634360-26AA4986-EE19-416C-925D-6A4AF5AC8519 Q33640082-6DAF87D5-2A32-410E-889E-D3DBD85DEC78 Q33701382-CCC84283-5267-43BA-8C70-CD0A21AAEE12 Q33746241-27DA34E5-1ADF-40D7-BA75-9F6D08D9ECC7 Q34157730-B5E8E1E6-5D2D-42D0-8984-1EC325522CFC Q34257527-7D1C6AF0-E8AB-4F08-8AF8-811312F36B8E Q35012837-150140E2-4B76-4B58-943A-EB6340DC1540 Q35108528-8682DFCA-C116-49C9-819F-C2C4A5396D78 Q35622131-B593945D-A1D2-46E0-9922-239DC10B11BB Q35779008-0AF3ABA1-D4B1-4E2D-88CE-6F661C9FC174 Q35783645-AC1D3711-FC8E-4C3D-82C3-EF20C9E37B50 Q36003378-E2970EDA-38CB-4E9F-8AB3-59012AB98AF1 Q36408527-063E632B-85DE-4AFE-8BAD-83FA57C0CD0E Q36490506-413E5C91-32B4-4B01-BD88-2D1E0E425E4D Q36547100-6750BE90-676B-4506-A22B-A2340FFCD9A0 Q36743443-06A20D2E-8B3F-4B18-BD78-059A975EACFE Q36770507-42A1B978-83F9-49EB-9143-AAE7A6E042B3 Q36821976-95AF796E-8D2D-4D75-B9A8-F447EBF26770 Q36911779-FD7D3D8B-DB34-4A24-A168-6EC730D23121 Q36930525-8070D590-F17C-479E-8A14-BF92EA970819 Q37001927-660D94F1-A7A6-46A9-81FF-A315CD08809F Q37020215-4A8AAC3B-05D5-4F73-AD6A-97EE135481A0 Q37122845-A6E9435F-C12F-4C7B-B50A-DBCA9665E9A9 Q37142394-A6ADB400-F60C-4307-B87F-EC4FDB9E064C Q37159385-E6571DFF-17E9-4679-A0A1-6CDD4F89574A Q37264091-D0EBFAC8-8922-4A28-93A0-556549C44651 Q37311618-4AEEF2D4-BC2C-4BBE-A9B9-2A444D31ECD5 Q37724740-8D7598D1-1984-4A19-B23E-E4A8AE2B7AE6 Q37826478-FF4A1B78-AB6A-4D4F-8D9F-DA199B7621E2 Q37953326-72242C04-5533-40A0-9030-1ACB2B644A0C Q38031743-B8A2387B-AB53-493B-AF26-0DA51A1DC980 Q38062727-13205A7A-4062-4F3E-B6EF-3B59429A6662 Q38084559-DB2BDBB9-1E6C-4B22-81BE-A417CF2F9B36 Q38204626-4415BEB6-271F-4AF4-871C-6408F6246637 Q38420594-119A927A-B986-456A-B53D-2C4C681ACB22 Q38742674-C7B894E5-CB31-4C9B-86F7-F75EEC475D1A

P2860

Starvation after AgRP neuron ablation is independent of melanocortin signaling.

http://www.wikidata.org/entity/Q36497492

description

2008 nî lūn-bûn

@nan

2008年の論文

@ja

2008年学术文章

@wuu

2008年学术文章

@zh-cn

2008年学术文章

@zh-hans

2008年学术文章

@zh-my

2008年学术文章

@zh-sg

2008年學術文章

@yue

2008年學術文章

@zh

2008年學術文章

@zh-hant

name

Starvation after AgRP neuron ablation is independent of melanocortin signaling.

@ast

Starvation after AgRP neuron ablation is independent of melanocortin signaling.

@en

type

label

Starvation after AgRP neuron ablation is independent of melanocortin signaling.

@ast

Starvation after AgRP neuron ablation is independent of melanocortin signaling.

@en

prefLabel

Starvation after AgRP neuron ablation is independent of melanocortin signaling.

@ast

Starvation after AgRP neuron ablation is independent of melanocortin signaling.

@en

P1433

Q36497492-56F24C4C-EF13-44CD-8DDD-9978A0FE2F03

P1476

Q36497492-CC0BF1A1-F8F6-48DD-B44F-C21CA34EE47A

P2093

Q36497492-053FFB09-3B31-48CD-9E0F-0F9D00D1A5DC

Q36497492-4D6E1F04-59AE-4078-962F-2B459A526F36

Q36497492-89341209-D11D-4A54-98C0-60B7E2585F57

P2860

Q36497492-093C0D56-A5EE-4B29-8ED3-6007A7EACEDB

Q36497492-0E7590FA-5C73-41A2-8066-F1C6DE2DF4BE

Q36497492-1024F94C-D030-4A7E-9133-4ADC74BFAF30

Q36497492-140EFF5C-5B36-48F8-930E-7BBBF0F2C6A8

Q36497492-20522076-B9C3-4ADD-B999-4BBDA73A7014

Q36497492-2AA700EE-CE00-4BA5-8B46-CA9EF2C8B050

Q36497492-329EF24E-DCC0-4FC5-A55B-90AE239C30AF

Q36497492-3441500F-AC05-4B7C-8BFE-9889F0B7AF4E

Q36497492-39688040-E4A2-4D0C-85A4-BA26FA3622D5

Q36497492-3A753BB8-111B-46F4-8F17-128AC4FB6233

P304

Q36497492-5DC1ED72-739A-43D7-98FE-D54591EFEEFC

P31

Q36497492-81552A4E-EACD-4CB3-8A26-51D8D13711B5

P356

Q36497492-80676E89-E5E6-4C70-942F-CCBDC4B511E1

P407

Q36497492-2143413E-231A-44C6-A7CC-87E20ED2532B

P433

Q36497492-B3AB6296-349F-4CC3-AFAD-51937DB7FB67

P478

Q36497492-E3FF5236-C215-45BF-8660-83FE2B184F3A

P50

Q36497492-B3C7AB70-1D89-4208-88EF-721941A2F0E4

P577

Q36497492-B4FFDABB-AE9B-4683-B812-940F24DFF061

P5875

Q36497492-EC677D23-6D15-4536-964E-95335B174806

P698

Q36497492-2F8A4277-1F57-4415-8A1B-2D58776FB95B

P932

Q36497492-48743C49-6E02-4DF6-85DC-BFE16DD21C8D

P356

PNAS.0712062105

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

Starvation after AgRP neuron ablation is independent of melanocortin signaling.

@en

P2093

Maureen P Howell

http://www.w3.org/2001/XMLSchema#string

Qi Wu

http://www.w3.org/2001/XMLSchema#string

Richard D Palmiter

http://www.w3.org/2001/XMLSchema#string

P2860

Positional cloning of the mouse obese gene and its human homologue

Antagonism of central melanocortin receptors in vitro and in vivo by agouti-related protein

Severe early-onset obesity, adrenal insufficiency and red hair pigmentation caused by POMC mutations in humans

Neither agouti-related protein nor neuropeptide Y is critically required for the regulation of energy homeostasis in mice

Neuropeptide Y in normal eating and in genetic and dietary-induced obesity

Genome-wide atlas of gene expression in the adult mouse brain

Targeted disruption of the melanocortin-4 receptor results in obesity in mice

Cardiovascular response, feeding behavior and locomotor activity in mice lacking the NPY Y1 receptor

Central nervous system control of food intake and body weight

Leptin activates anorexigenic POMC neurons through a neural network in the arcuate nucleus

P304

2687-2692

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1073/PNAS.0712062105

http://www.w3.org/2001/XMLSchema#string

P407

P433

7

http://www.w3.org/2001/XMLSchema#string

P478

105

http://www.w3.org/2001/XMLSchema#string

P50

P577

2008-02-13T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

5580600

http://www.w3.org/2001/XMLSchema#string

P698

18272480

http://www.w3.org/2001/XMLSchema#string

P932

2268197

http://www.w3.org/2001/XMLSchema#string