Cystic fibrosis enters the proteomics scene: new answers to old questions.
about
Disruption of cytokeratin-8 interaction with F508del-CFTR corrects its functional defectStructures of a minimal human CFTR first nucleotide-binding domain as a monomer, head-to-tail homodimer, and pathogenic mutantUnderstanding protein kinase CK2 mis-regulation upon F508del CFTR expressionProteomics in protein misfolding diseases.Peroxiredoxin 6 fails to limit phospholipid peroxidation in lung from Cftr-knockout mice subjected to oxidative challenge.Eicosanoid release is increased by membrane destabilization and CFTR inhibition in Calu-3 cellsA novel lipidomic strategy reveals plasma phospholipid signatures associated with respiratory disease severity in cystic fibrosis patients.Airway proteins involved in bacterial clearance susceptible to cathepsin G proteolysis.Lipid mapping of colonic mucosa by cluster TOF-SIMS imaging and multivariate analysis in cftr knockout mice.Emergent properties of proteostasis in managing cystic fibrosis.Plasma lipidomics reveals potential prognostic signatures within a cohort of cystic fibrosis patients.Discovery of novel potent ΔF508-CFTR correctors that target the nucleotide binding domain.Quantitative proteomics reveals an altered cystic fibrosis in vitro bronchial epithelial secretomeOmics approaches in cystic fibrosis research: a focus on oxylipin profiling in airway secretions.Chemical and biological folding contribute to temperature-sensitive DeltaF508 CFTR traffickingCystic fibrosis as a bowel cancer syndrome and the potential role of CK2.Proteomics of lung cell biology and pulmonary disease.Misfolded proteins and neurodegeneration: role of non-native cytochrome c in cell death.Proteomics investigation of human platelets in healthy donors and cystic fibrosis patients by shotgun nUPLC-MSE and 2DE: a comparative study.Proteomic and ionomic profiling reveals significant alterations of protein expression and calcium homeostasis in cystic fibrosis cells.Structural models of CFTR-AMPK and CFTR-PKA interactions: R-domain flexibility is a key factor in CFTR regulation.Interactions between an inflammatory response to infection and protein trafficking pathways favor correction of defective protein trafficking in Cystic Fibrosis.
P2860
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P2860
Cystic fibrosis enters the proteomics scene: new answers to old questions.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Cystic fibrosis enters the proteomics scene: new answers to old questions.
@ast
Cystic fibrosis enters the proteomics scene: new answers to old questions.
@en
type
label
Cystic fibrosis enters the proteomics scene: new answers to old questions.
@ast
Cystic fibrosis enters the proteomics scene: new answers to old questions.
@en
prefLabel
Cystic fibrosis enters the proteomics scene: new answers to old questions.
@ast
Cystic fibrosis enters the proteomics scene: new answers to old questions.
@en
P2860
P356
P1433
P1476
Cystic fibrosis enters the proteomics scene: new answers to old questions
@en
P2093
Aleksander Edelman
Franck Brouillard
P2860
P304
P356
10.1002/PMIC.200600028
P50
P577
2006-07-01T00:00:00Z