Status Epilepticus Enhances Depotentiation after Fully Established LTP in an NMDAR-Dependent but GluN2B-Independent Manner
about
Functional Metaplasticity of Hippocampal Schaffer Collateral-CA1 Synapses Is Reversed in Chronically Epileptic Rats.NMDAR-independent hippocampal long-term depression impairment after status epilepticus in a lithium-pilocarpine model of temporal lobe epilepsy.Cardiac Arrest Induces Ischemic Long-Term Potentiation of Hippocampal CA1 Neurons That Occludes Physiological Long-Term Potentiation.
P2860
Status Epilepticus Enhances Depotentiation after Fully Established LTP in an NMDAR-Dependent but GluN2B-Independent Manner
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Status Epilepticus Enhances De ...... but GluN2B-Independent Manner
@ast
Status Epilepticus Enhances De ...... but GluN2B-Independent Manner
@en
type
label
Status Epilepticus Enhances De ...... but GluN2B-Independent Manner
@ast
Status Epilepticus Enhances De ...... but GluN2B-Independent Manner
@en
prefLabel
Status Epilepticus Enhances De ...... but GluN2B-Independent Manner
@ast
Status Epilepticus Enhances De ...... but GluN2B-Independent Manner
@en
P2093
P2860
P356
P1433
P1476
Status Epilepticus Enhances De ...... but GluN2B-Independent Manner
@en
P2093
Timo Kirschstein
Tursonjan Tokay
Xiati Guli
P2860
P304
P356
10.1155/2016/6592038
P577
2016-01-03T00:00:00Z