Flightless-I regulates proinflammatory caspases by selectively modulating intracellular localization and caspase activity.
about
Retinoic acid-induced gene-1 (RIG-I) associates with the actin cytoskeleton via caspase activation and recruitment domain-dependent interactionsCytoskeletal Regulation of Inflammation and Its Impact on Skin Blistering Disease Epidermolysis Bullosa AcquisitaThe Roles of RNase-L in Antimicrobial Immunity and the Cytoskeleton-Associated Innate ResponseThe cytoskeleton in cell-autonomous immunity: structural determinants of host defenceCaspase-1 activation via Rho GTPases: a common theme in mucosal infections?The Jekyll and Hyde functions of caspases.Caspase-11 controls interleukin-1β release through degradation of TRPC1.Flightless I interacts with NMMIIA to promote cell extension formation, which enables collagen remodeling.Glutaredoxin 1 protects dopaminergic cells by increased protein glutathionylation in experimental Parkinson's disease.Lysosomal secretion of Flightless I upon injury has the potential to alter inflammationCytoskeletal protein Flightless I inhibits apoptosis, enhances tumor cell invasion and promotes cutaneous squamous cell carcinoma progressionThe influence of Flightless I on Toll-like-receptor-mediated inflammation in a murine model of diabetic wound healing.The Yersinia virulence effector YopM binds caspase-1 to arrest inflammasome assembly and processing.LRRFIP2 negatively regulates NLRP3 inflammasome activation in macrophages by promoting Flightless-I-mediated caspase-1 inhibition.Dynamic expression of epidermal caspase 8 simulates a wound healing response.Identification of Flightless-I as a substrate of the cytokine-independent survival kinase CISK.Modulation of innate immune responses by Yersinia type III secretion system translocators and effectors.The Roles of Lysosomes in Inflammation and Autoimmune Diseases.Enzymatically Inactive Procaspase 1 stabilizes the ASC Pyroptosome and Supports Pyroptosome Spreading during Cell DivisionEvasion and interference: intracellular pathogens modulate caspase-dependent inflammatory responses.Nucleoredoxin negatively regulates Toll-like receptor 4 signaling via recruitment of flightless-I to myeloid differentiation primary response gene (88).PKR stirs up inflammasomes.Attenuation of flightless I improves wound healing and enhances angiogenesis in a murine model of type 1 diabetes.A Membrane Potential- and Calpain-Dependent Reversal of Caspase-1 Inhibition Regulates Canonical NLRP3 Inflammasome
P2860
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P2860
Flightless-I regulates proinflammatory caspases by selectively modulating intracellular localization and caspase activity.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Flightless-I regulates proinfl ...... lization and caspase activity.
@ast
Flightless-I regulates proinfl ...... lization and caspase activity.
@en
type
label
Flightless-I regulates proinfl ...... lization and caspase activity.
@ast
Flightless-I regulates proinfl ...... lization and caspase activity.
@en
prefLabel
Flightless-I regulates proinfl ...... lization and caspase activity.
@ast
Flightless-I regulates proinfl ...... lization and caspase activity.
@en
P2093
P2860
P356
P1476
Flightless-I regulates proinfl ...... lization and caspase activity.
@en
P2093
Helen L Yin
Junying Yuan
P2860
P304
P356
10.1083/JCB.200711082
P407
P577
2008-04-14T00:00:00Z