ROS-induced ATF3 causes susceptibility to secondary infections during sepsis-associated immunosuppression. - Wikidata - awgldk - TriplyDB

ROS-induced ATF3 causes susceptibility to secondary infections during sepsis-associated immunosuppression.

ROS-induced ATF3 causes susceptibility to secondary infections during sepsis-associated immunosuppression.

http://www.wikidata.org/entity/Q36564435

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Immune responsive gene 1 (IRG1) promotes endotoxin tolerance by increasing A20 expression in macrophages through reactive oxygen species Nrf2 as a master regulator of tissue damage control and disease tolerance to infection Redox control of microglial function: molecular mechanisms and functional significance Roles nrf2 plays in myeloid cells and related disorders The Drosophila MAPK p38c regulates oxidative stress and lipid homeostasis in the intestine Hydrogen gas presents a promising therapeutic strategy for sepsis.Oxidant-induced activation of cGMP-dependent protein kinase Iα mediates neuropathic pain after peripheral nerve injury.Neuroprotective effects of sulforaphane on cholinergic neurons in mice with Alzheimer's disease-like lesions.Activating transcription factor 3-mediated chemo-intervention with cancer chemokines in a noncanonical pathway under endoplasmic reticulum stress Sustained oxidative stress causes late acute renal failure via duplex regulation on p38 MAPK and Akt phosphorylation in severely burned rats.Endoplasmic reticulum stress-dependent activation of ATF3 mediates the late phase of ischemic preconditioning Exposure to diesel exhaust particle extracts (DEPe) impairs some polarization markers and functions of human macrophages through activation of AhR and Nrf2.IL-4 abrogates T(H)17 cell-mediated inflammation by selective silencing of IL-23 in antigen-presenting cells ATF3 protects pulmonary resident cells from acute and ventilator-induced lung injury by preventing Nrf2 degradation.Macrophages sense and kill bacteria through carbon monoxide-dependent inflammasome activation.Astaxanthin attenuates early acute kidney injury following severe burns in rats by ameliorating oxidative stress and mitochondrial-related apoptosis Loss of ATF3 promotes Akt activation and prostate cancer development in a Pten knockout mouse model The common stress responsive transcription factor ATF3 binds genomic sites enriched with p300 and H3K27ac for transcriptional regulation.Function of GRIM-19, a mitochondrial respiratory chain complex I protein, in innate immunity.Indole-3-carbinol and its N-alkoxy derivatives preferentially target ERα-positive breast cancer cells Superoxide Dismutase 1 Protects Hepatocytes from Type I Interferon-Driven Oxidative Damage Macrophages and Iron Metabolism.Suppression of inflammation and acute lung injury by Miz1 via repression of C/EBP-δ.Transcription factor ATF3 links host adaptive response to breast cancer metastasis.Redox control of inflammation in macrophages.Integrative transcriptome analysis reveals dysregulation of canonical cancer molecular pathways in placenta leading to preeclampsia ATF3 Protects against LPS-Induced Inflammation in Mice via Inhibiting HMGB1 Expression.Activated Transcription Factor 3 in Association with Histone Deacetylase 6 Negatively Regulates MicroRNA 199a2 Transcription by Chromatin Remodeling and Reduces Endothelin-1 Expression.Antioxidant activity of sestrin 2 controls neuropathic pain after peripheral nerve injury Gene expression analysis of TREM1 and GRK2 in polymorphonuclear leukocytes as the surrogate biomarkers of acute bacterial infections Are reactive oxygen species always detrimental to pathogens?ATF3 acts as a rheostat to control JNK signalling during intestinal regeneration.Bimodal immune activation in psoriasis.Immunopathogenesis of abdominal sepsis.Cross-talk between HPA-axis-increased glucocorticoids and mitochondrial stress determines immune responses and clinical manifestations of patients with sepsis.DJ-1/PARK7 Impairs Bacterial Clearance in Sepsis.Involvement of ATF3 in the negative regulation of iNOS expression and NO production in activated macrophages.TLR4 mediates pneumolysin-induced ATF3 expression through the JNK/p38 pathway in Streptococcus pneumoniae-infected RAW 264.7 cells.Streptococcus pneumoniae-Induced Oxidative Stress in Lung Epithelial Cells Depends on Pneumococcal Autolysis and Is Reversible by Resveratrol.In situ electroactive and antioxidant supramolecular hydrogel based on cyclodextrin/copolymer inclusion for tissue engineering repair.

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ROS-induced ATF3 causes susceptibility to secondary infections during sepsis-associated immunosuppression.

http://www.wikidata.org/entity/Q36564435

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ROS-induced ATF3 causes suscep ...... -associated immunosuppression.

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Nature Medicine

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Anna Teske

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Bernd Echtenacher

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Claus Krenn

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Florian Woelbing

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Ji-Hyeon Park

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Jürgen Brück

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Kamran Ghoreschi

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Kerstin Fuchs

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Kyu-Han Kim

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Kyu-Won Kim

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P2860

Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

Definitions for sepsis and organ failure and guidelines for the use of innovative therapies in sepsis. The ACCP/SCCM Consensus Conference Committee. American College of Chest Physicians/Society of Critical Care Medicine

Innate immune recognition

Passive immunization against cachectin/tumor necrosis factor protects mice from lethal effect of endotoxin

An important function of Nrf2 in combating oxidative stress: detoxification of acetaminophen

Characterization of tumor necrosis factor-deficient mice

The epidemiology of sepsis in the United States from 1979 through 2000

The pathophysiology and treatment of sepsis

Expression of stress-response ATF3 is mediated by Nrf2 in astrocytes.

ATF3 and stress responses.

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P304

128-134

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10.1038/NM.2557

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1

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18

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Konrad Hoetzenecker

Emmanuella Guenova

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2011-12-18T00:00:00Z

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51897890

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22179317

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immunosuppression

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3555699

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