Circulating Growth Differentiation Factor 11/8 Levels Decline With Age. - Wikidata - awgldk - TriplyDB

Circulating Growth Differentiation Factor 11/8 Levels Decline With Age.

Circulating Growth Differentiation Factor 11/8 Levels Decline With Age.

http://www.wikidata.org/entity/Q36566423

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Extracellular RNA in aging Molecular studies of exercise, skeletal muscle, and ageing When stem cells grow old: phenotypes and mechanisms of stem cell aging Role of circulating factors in cardiac aging.Structural basis for potency differences between GDF8 and GDF11.Activin A more prominently regulates muscle mass in primates than does GDF8.The Role of the Molecular Clock in Skeletal Muscle and What It Is Teaching Us About Muscle-Bone Crosstalk.Biochemistry and Biology of GDF11 and Myostatin: Similarities, Differences, and Questions for Future Investigation Is Growth Differentiation Factor 11 a Realistic Therapeutic for Aging-Dependent Muscle Defects?Supraphysiological levels of GDF11 induce striated muscle atrophy Serum Levels of Growth Differentiation Factor 11 Are Independently Associated with Low Hemoglobin Values in Hemodialysis Patients Quantification of GDF11 and Myostatin in Human Aging and Cardiovascular Disease.GDF11/BMP11 activates both smad1/5/8 and smad2/3 signals but shows no significant effect on proliferation and migration of human umbilical vein endothelial cells.Molecular circuitry of stem cell fate in skeletal muscle regeneration, ageing and disease.GDF11 decreases bone mass by stimulating osteoclastogenesis and inhibiting osteoblast differentiation.GDF11 improves tubular regeneration after acute kidney injury in elderly mice.GDF11 administration does not extend lifespan in a mouse model of premature aging.Growth Differentiation Factor 11 does not Mitigate the Lethal Effects of Total-Abdominal Irradiation.Systemic Problems: A perspective on stem cell aging and rejuvenation.Quantitation of circulating GDF-11 and β2-MG in aged patients with age-related impairment in cognitive function.Exogenous GDF11 induces cardiac and skeletal muscle dysfunction and wasting.Interorgan Communication Pathways in Physiology: Focus on Drosophila.Pathways for salvage and protection of the heart under stress: novel routes for cardiac rejuvenation.Rejuvenating Strategies for Stem Cell-Based Therapies in Aging.The brain, sirtuins, and ageing.Treatment with rGDF11 does not improve the dystrophic muscle pathology of mdx mice Circulating Concentrations of Growth Differentiation Factor 11 Are Heritable and Correlate With Life Span.GDF11 Protects against Endothelial Injury and Reduces Atherosclerotic Lesion Formation in Apolipoprotein E-Null Mice.Questions and Answers About Myostatin, GDF11, and the Aging Heart.Getting Old through the Blood: Circulating Molecules in Aging and Senescence of Cardiovascular Regenerative Cells.Role of growth differentiation factor 11 in development, physiology and disease.Tumor-Suppressor Inactivation of GDF11 Occurs by Precursor Sequestration in Triple-Negative Breast Cancer.* The Impact of Age on Skeletal Muscle Progenitor Cell Survival and Fate After Injury.Targeted myocardial delivery of GDF11 gene rejuvenates the aged mouse heart and enhances myocardial regeneration after ischemia-reperfusion injury.Myostatin propeptide mutation of the hypermuscular Compact mice decreases the formation of myostatin and improves insulin sensitivity.Muscle Stem Cells: A Model System for Adult Stem Cell Biology.Endoplasmic Reticulum Stress Induces Myostatin High Molecular Weight Aggregates and Impairs Mature Myostatin Secretion.Crystal structure of human GDF11.Neonatal Systemic AAV-Mediated Gene Delivery of GDF11 Inhibits Skeletal Muscle Growth.GDF11 Modulates Ca2+-Dependent Smad2/3 Signaling to Prevent Cardiomyocyte Hypertrophy.

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P2860

Circulating Growth Differentiation Factor 11/8 Levels Decline With Age.

http://www.wikidata.org/entity/Q36566423

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2015 nî lūn-bûn

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Circulating Growth Differentiation Factor 11/8 Levels Decline With Age.

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Circulating Growth Differentiation Factor 11/8 Levels Decline With Age.

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Circulating Growth Differentiation Factor 11/8 Levels Decline With Age.

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Circulating Growth Differentiation Factor 11/8 Levels Decline With Age.

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CIRCRESAHA.115.307521

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Circulation Research

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Circulating Growth Differentiation Factor 11/8 Levels Decline With Age

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Amy J Wagers

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Ana Vujic

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Aysu Uygur

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Emilia Gonzalez

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James R Pancoast

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Jill Goldstein

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Miook Cho

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Rachel M Tandias

http://www.w3.org/2001/XMLSchema#string

Richard T Lee

http://www.w3.org/2001/XMLSchema#string

Tommaso Poggioli

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P2860

Regulation of myostatin activity and muscle growth

Genetic deletion of myostatin from the heart prevents skeletal muscle atrophy in heart failure

Autoregulation of neurogenesis by GDF11

Regulation of skeletal muscle mass in mice by a new TGF-beta superfamily member

Restoring systemic GDF11 levels reverses age-related dysfunction in mouse skeletal muscle

GDF11 Increases with Age and Inhibits Skeletal Muscle Regeneration

GDF11 does not rescue aging-related pathological hypertrophy

Growth differentiation factor 11 is a circulating factor that reverses age-related cardiac hypertrophy

GDF11 modulates NGN3+ islet progenitor cell number and promotes beta-cell differentiation in pancreas development

Molecular aging and rejuvenation of human muscle stem cells.

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29-37

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scholarly article

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10.1161/CIRCRESAHA.115.307521

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1

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118

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Francesco Loffredo

Thomas B. Thompson

Claudio Macias-Trevino

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2015-10-21T00:00:00Z

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26489925

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4748736

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