Peptide-mediated desmoglein 3 crosslinking prevents pemphigus vulgaris autoantibody-induced skin blistering
about
Adducin is required for desmosomal cohesion in keratinocytes.Mechanisms of Autoantibody-Induced Pathology.Immature truncated O-glycophenotype of cancer directly induces oncogenic featuresDesmosomes: regulators of cellular signaling and adhesion in epidermal health and disease.F-actin binding protein, anillin, regulates integrity of intercellular junctions in human epithelial cells.Setting the target for pemphigus vulgaris therapy.p38 MAPK Signaling in Pemphigus: Implications for Skin AutoimmunityPlakophilins, desmogleins, and pemphigus: the tail wagging the dog.Desmoglein 2 regulates the intestinal epithelial barrier via p38 mitogen-activated protein kinaseMechanisms of Disease: Pemphigus and Bullous Pemphigoid.Desmoglein 2 compensates for desmoglein 3 but does not control cell adhesion via regulation of p38 mitogen-activated protein kinase in keratinocytes.MAPKAP kinase 2 (MK2)-dependent and -independent models of blister formation in pemphigus vulgaris.Crosstalk between Signaling Pathways in Pemphigus: A Role for Endoplasmic Reticulum Stress in p38 Mitogen-Activated Protein Kinase Activation?Different signaling patterns contribute to loss of keratinocyte cohesion dependent on autoantibody profile in pemphigus.Peptide mimetics of immunoglobulin A (IgA) and FcαRI block IgA-induced human neutrophil activation and migration.Pemphigus: a Comprehensive Review on Pathogenesis, Clinical Presentation and Novel Therapeutic Approaches.Pemphigus-A Disease of Desmosome Dysfunction Caused by Multiple Mechanisms.Atomic Force Microscopy Provides New Mechanistic Insights into the Pathogenesis of Pemphigus.Keratins Regulate p38MAPK-Dependent Desmoglein Binding Properties in Pemphigus.Autoantibody Signaling in Pemphigus Vulgaris: Development of an Integrated Model.Keratin Retraction and Desmoglein3 Internalization Independently Contribute to Autoantibody-Induced Cell Dissociation in Pemphigus Vulgaris.Immunoadsorption of Desmoglein-3-Specific IgG Abolishes the Blister-Inducing Capacity of Pemphigus Vulgaris IgG in Neonatal Mice
P2860
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P2860
Peptide-mediated desmoglein 3 crosslinking prevents pemphigus vulgaris autoantibody-induced skin blistering
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Peptide-mediated desmoglein 3 ...... tibody-induced skin blistering
@ast
Peptide-mediated desmoglein 3 ...... tibody-induced skin blistering
@en
type
label
Peptide-mediated desmoglein 3 ...... tibody-induced skin blistering
@ast
Peptide-mediated desmoglein 3 ...... tibody-induced skin blistering
@en
prefLabel
Peptide-mediated desmoglein 3 ...... tibody-induced skin blistering
@ast
Peptide-mediated desmoglein 3 ...... tibody-induced skin blistering
@en
P2093
P2860
P356
P1476
Peptide-mediated desmoglein 3 ...... tibody-induced skin blistering
@en
P2093
Bettina Kempf
Carina Dehner
Enno Schmidt
Eva Hartlieb
Gregory S Harms
Mariya Radeva
Martin Gliem
Vera Rötzer
Volker Spindler
P2860
P304
P356
10.1172/JCI60139
P407
P50
P577
2013-01-09T00:00:00Z