Chronic hypoxia suppresses pregnancy-induced upregulation of large-conductance Ca2+-activated K+ channel activity in uterine arteries
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Oxidative Stress and Maxi Calcium-Activated Potassium (BK) ChannelsGestational hypoxia up-regulates protein kinase C and inhibits calcium-activated potassium channels in ovine uterine arteries.Higher estrogen levels during pregnancy in Andean than European residents of high altitude suggest differences in aromatase activityEpigenetic upregulation of large-conductance Ca2+-activated K+ channel expression in uterine vascular adaptation to pregnancyWestern blot analysis of BK channel β1-subunit expression should be interpreted cautiously when using commercially available antibodies.Chronic hypoxia during gestation enhances uterine arterial myogenic tone via heightened oxidative stress.Uterine artery blood flow, fetal hypoxia and fetal growth.Gestational hypoxia increases reactive oxygen species and inhibits steroid hormone-mediated upregulation of Ca(2+)-activated K(+) channel function in uterine arteries.Hypoxia Represses ER-α Expression and Inhibits Estrogen-Induced Regulation of Ca2+-Activated K+ Channel Activity and Myogenic Tone in Ovine Uterine Arteries: Causal Role of DNA MethylationChronic hypoxia during gestation causes epigenetic repression of the estrogen receptor-α gene in ovine uterine arteries via heightened promoter methylation.Developmental acceleration of bradykinin-dependent relaxation by prenatal chronic hypoxia impedes normal development after birthChronic hypoxia inhibits pregnancy-induced upregulation of SKCa channel expression and function in uterine arteries.Gestational hypoxia induces preeclampsia-like symptoms via heightened endothelin-1 signaling in pregnant rats.Chronic Hypobaric Hypoxia Modulates Primary Cilia Differently in Adult and Fetal Ovine Kidneys.Pregnancy Reprograms Large-Conductance Ca2+-Activated K+ Channel in Uterine Arteries: Roles of Ten-Eleven Translocation Methylcytosine Dioxygenase 1-Mediated Active Demethylation.Direct effect of chronic hypoxia in suppressing large conductance Ca(2+)-activated K(+) channel activity in ovine uterine arteries via increasing oxidative stress.Chronic hypoxia upregulates DNA methyltransferase and represses large conductance Ca2+-activated K+ channel function in ovine uterine arteries.MicroRNA-210 Targets Ten-Eleven Translocation Methylcytosine Dioxygenase 1 and Suppresses Pregnancy-Mediated Adaptation of Large Conductance Ca2+-Activated K+ Channel Expression and Function in Ovine Uterine Arteries.Hypoxia, AMPK activation and uterine artery vasoreactivity.Long-term hypoxia uncouples Ca and eNOS in bradykinin-mediated pulmonary arterial relaxation
P2860
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P2860
Chronic hypoxia suppresses pregnancy-induced upregulation of large-conductance Ca2+-activated K+ channel activity in uterine arteries
description
2012 nî lūn-bûn
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2012年の論文
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2012年論文
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2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
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2012年论文
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2012年论文
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2012年论文
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name
Chronic hypoxia suppresses pre ...... l activity in uterine arteries
@ast
Chronic hypoxia suppresses pre ...... l activity in uterine arteries
@en
type
label
Chronic hypoxia suppresses pre ...... l activity in uterine arteries
@ast
Chronic hypoxia suppresses pre ...... l activity in uterine arteries
@en
prefLabel
Chronic hypoxia suppresses pre ...... l activity in uterine arteries
@ast
Chronic hypoxia suppresses pre ...... l activity in uterine arteries
@en
P2093
P2860
P1433
P1476
Chronic hypoxia suppresses pre ...... l activity in uterine arteries
@en
P2093
Daliao Xiao
Lubo Zhang
Ronghui Zhu
Sean M Wilson
Shumei Yang
Xiang-Qun Hu
Xiaohui Huang
P2860
P304
P356
10.1161/HYPERTENSIONAHA.112.196097
P407
P577
2012-06-04T00:00:00Z