Cellular adaptations to hypoxia and acidosis during somatic evolution of breast cancer - Wikidata - awgldk - TriplyDB

Cellular adaptations to hypoxia and acidosis during somatic evolution of breast cancer

Cellular adaptations to hypoxia and acidosis during somatic evolution of breast cancer

http://www.wikidata.org/entity/Q36611104

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Is there a role for carbohydrate restriction in the treatment and prevention of cancer?Mechanisms of bone metastases of breast cancer Breast Cancer Metabolism and Mitochondrial Activity: The Possibility of Chemoprevention with Metformin Voltage-gated sodium channels and cancer: is excitability their primary role?Integrated PK-PD and agent-based modeling in oncology Evolution of the cancer genome Recapitulation of tumor heterogeneity and molecular signatures in a 3D brain cancer model with decreased sensitivity to histone deacetylase inhibition Can we negotiate with a tumor?Matriptase autoactivation is tightly regulated by the cellular chemical environments Acid gradient across plasma membrane can drive phosphate bond synthesis in cancer cells: acidic tumor milieu as a potential energy source Leveraging Hypoxia-Activated Prodrugs to Prevent Drug Resistance in Solid Tumors Vignettes from the field of mathematical biology: the application of mathematics to biology and medicine Patient-calibrated agent-based modelling of ductal carcinoma in situ (DCIS): from microscopic measurements to macroscopic predictions of clinical progression.Emergence of spatial structure in the tumor microenvironment due to the Warburg effect Metabolomic Dynamic Analysis of Hypoxia in MDA-MB-231 and the Comparison with Inferred Metabolites from Transcriptomics Data.Modelling acidosis and the cell cycle in multicellular tumour spheroids.Hyperglycemia, a neglected factor during cancer progression Exosomal Proteome Profiling: A Potential Multi-Marker Cellular Phenotyping Tool to Characterize Hypoxia-Induced Radiation Resistance in Breast Cancer.Characterization of monocarboxylate transporters (MCTs) expression in soft tissue sarcomas: distinct prognostic impact of MCT1 sub-cellular localization.A 2D mechanistic model of breast ductal carcinoma in situ (DCIS) morphology and progression.Episodic, transient systemic acidosis delays evolution of the malignant phenotype: Possible mechanism for cancer prevention by increased physical activity Cancer models, genomic instability and somatic cellular Darwinian evolution MR-visible lipids and the tumor microenvironment Relationships between hypoxia markers and the leptin system, estrogen receptors in human primary and metastatic breast cancer: effects of preoperative chemotherapy.Non-canonical HIF-2α function drives autonomous breast cancer cell growth via an AREG-EGFR/ErbB4 autocrine loop.Three-dimensional multispecies nonlinear tumor growth-II: Tumor invasion and angiogenesis Transketolase-like 1 expression is modulated during colorectal cancer progression and metastasis formation Nonlinear modelling of cancer: bridging the gap between cells and tumours Anti-tumour/metastasis effects of the potassium-sparing diuretic amiloride: an orally active anti-cancer drug waiting for its call-of-duty?Adaptation to hypoxia and acidosis in carcinogenesis and tumor progression Hypoxia-adaptation involves mitochondrial metabolic depression and decreased ROS leakage An Adaptive Multigrid Algorithm for Simulating Solid Tumor Growth Using Mixture Models Evolution of Tumor Metabolism might Reflect Carcinogenesis as a Reverse Evolution process (Dismantling of Multicellularity).Simulating cancer growth with multiscale agent-based modeling Role of hypoxia and EGF on expression, activity, localization and phosphorylation of carbonic anhydrase IX in MDA-MB-231 breast cancer cells.Tumor metabolism, cancer cell transporters, and microenvironmental resistance.Diabetogenic glucose and insulin concentrations modulate transcriptome and protein levels involved in tumour cell migration, adhesion and proliferation.Microenvironmental acidosis in carcinogenesis and metastases: new strategies in prevention and therapy.Hybrid models of tumor growth.Catabolism of exogenous lactate reveals it as a legitimate metabolic substrate in breast cancer.

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Cellular adaptations to hypoxia and acidosis during somatic evolution of breast cancer

http://www.wikidata.org/entity/Q36611104

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2007年论文

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2007年论文

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Cellular adaptations to hypoxia and acidosis during somatic evolution of breast cancer

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Cellular adaptations to hypoxia and acidosis during somatic evolution of breast cancer

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Cellular adaptations to hypoxia and acidosis during somatic evolution of breast cancer

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Cellular adaptations to hypoxia and acidosis during somatic evolution of breast cancer

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Cellular adaptations to hypoxia and acidosis during somatic evolution of breast cancer

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Cellular adaptations to hypoxia and acidosis during somatic evolution of breast cancer

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British Journal of Cancer

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Cellular adaptations to hypoxia and acidosis during somatic evolution of breast cancer

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J Averill

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K Smallbone

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L Worrall

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P K Maini

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R A Gatenby

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R B Nagle

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P2860

Expression of the hypoxia-inducible and tumor-associated carbonic anhydrases in ductal carcinoma in situ of the breast

A genetic model for colorectal tumorigenesis

Why do cancers have high aerobic glycolysis?

The role of acidity in solid tumour growth and invasion

Positron emission tomography scanning: current and future applications.

Molecular imaging of cancer with positron emission tomography.

Fatty acid oxidation is a dominant bioenergetic pathway in prostate cancer.

In vivo positron-emission tomography imaging of progression and transformation in a mouse model of mammary neoplasia.

The histological structure of some human lung cancers and the possible implications for radiotherapy.

An acidic environment leads to p53 dependent induction of apoptosis in human adenoma and carcinoma cell lines: implications for clonal selection during colorectal carcinogenesis.

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Robert J Gillies

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