Flow cytometric characterization of the response of Fanconi's anemia cells to mitomycin C treatment.
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The FANCJ/MutLalpha interaction is required for correction of the cross-link response in FA-J cellsFanconi anemia proteins FANCA, FANCC, and FANCG/XRCC9 interact in a functional nuclear complexDiagnosis of Fanconi Anemia: Chromosomal Breakage AnalysisThe Fanconi anaemia/BRCA pathwayFanconi anemia proteins localize to chromatin and the nuclear matrix in a DNA damage- and cell cycle-regulated manner.FANCG is phosphorylated at serines 383 and 387 during mitosisFunctional activity of the fanconi anemia protein FAA requires FAC binding and nuclear localization.Inherited and inducible chromosomal instability: a fragile bridge between genome integrity mechanisms and tumourigenesis.Congenital bone marrow failure syndromes.CCAAT/enhancer binding protein delta (C/EBPdelta, CEBPD)-mediated nuclear import of FANCD2 by IPO4 augments cellular response to DNA damage.Phenotypic correction of Fanconi anemia in human hematopoietic cells with a recombinant adeno-associated virus vector.Disrupted Signaling through the Fanconi Anemia Pathway Leads to Dysfunctional Hematopoietic Stem Cell Biology: Underlying Mechanisms and Potential Therapeutic Strategies.Diagnosis of Fanconi Anemia: Mutation Analysis by Multiplex Ligation-Dependent Probe Amplification and PCR-Based Sanger SequencingThe fanconi anemia pathway requires FAA phosphorylation and FAA/FAC nuclear accumulation.Fanconi anemia proteins, DNA interstrand crosslink repair pathways, and cancer therapy.Abnormal response to DNA crosslinking agents of Fanconi anemia fibroblasts can be corrected by transfection with normal human DNA.DNA replication is required To elicit cellular responses to psoralen-induced DNA interstrand cross-links.Subtyping analysis of Fanconi anemia by immunoblotting and retroviral gene transfer.Effectiveness of isolated liver perfusion with mitomycin C in the treatment of liver tumours of rat colorectal cancer.Diagnostic Overlap between Fanconi Anemia and the Cohesinopathies: Roberts Syndrome and Warsaw Breakage SyndromeUSP1 deubiquitinase maintains phosphorylated CHK1 by limiting its DDB1-dependent degradation.
P2860
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P2860
Flow cytometric characterization of the response of Fanconi's anemia cells to mitomycin C treatment.
description
1982 nî lūn-bûn
@nan
1982年の論文
@ja
1982年論文
@yue
1982年論文
@zh-hant
1982年論文
@zh-hk
1982年論文
@zh-mo
1982年論文
@zh-tw
1982年论文
@wuu
1982年论文
@zh
1982年论文
@zh-cn
name
Flow cytometric characterizati ...... ells to mitomycin C treatment.
@ast
Flow cytometric characterizati ...... ells to mitomycin C treatment.
@en
type
label
Flow cytometric characterizati ...... ells to mitomycin C treatment.
@ast
Flow cytometric characterizati ...... ells to mitomycin C treatment.
@en
prefLabel
Flow cytometric characterizati ...... ells to mitomycin C treatment.
@ast
Flow cytometric characterizati ...... ells to mitomycin C treatment.
@en
P2093
P2860
P356
P1433
P1476
Flow cytometric characterizati ...... ells to mitomycin C treatment.
@en
P2093
P2860
P304
P356
10.1002/CYTO.990020505
P577
1982-03-01T00:00:00Z