IL-6 trans-signaling promotes pancreatitis-associated lung injury and lethality. - Wikidata - awgldk - TriplyDB

IL-6 trans-signaling promotes pancreatitis-associated lung injury and lethality.

IL-6 trans-signaling promotes pancreatitis-associated lung injury and lethality.

http://www.wikidata.org/entity/Q36638151

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Cell death and DAMPs in acute pancreatitis Externalized decondensed neutrophil chromatin occludes pancreatic ducts and drives pancreatitis Advances in Biomedical Imaging, Bioengineering, and Related Technologies for the Development of Biomarkers of Pancreatic Disease: Summary of a National Institute of Diabetes and Digestive and Kidney Diseases and National Institute of Biomedical Imag Inhibitors of ORAI1 Prevent Cytosolic Calcium-Associated Injury of Human Pancreatic Acinar Cells and Acute Pancreatitis in 3 Mouse Models Tocilizumab-induced pancreatitis: case report and review of data from the FDA Adverse Event Reporting System.Inhibition of Jak/STAT signaling reduces the activation of pancreatic stellate cells in vitro and limits caerulein-induced chronic pancreatitis in vivo.Inflammation and Epithelial-Mesenchymal Transition in Pancreatic Ductal Adenocarcinoma: Fighting Against Multiple Opponents Interleukin-6 in Allogeneic Stem Cell Transplantation: Its Possible Importance for Immunoregulation and As a Therapeutic Target.Combined administration of a sedative dose sevoflurane and 60% oxygen reduces inflammatory responses to sepsis in animals and in human PMBCs.Blockade of IL-6 Trans signaling attenuates pulmonary fibrosis Kynurenine-3-monooxygenase inhibition prevents multiple organ failure in rodent models of acute pancreatitis.Study on acute recent stage pancreatitis.Regeneration and repair of the exocrine pancreas.Effects of the mitochondria-targeted antioxidant mitoquinone in murine acute pancreatitis Exposure to Radiocontrast Agents Induces Pancreatic Inflammation by Activation of Nuclear Factor-κB, Calcium Signaling, and Calcineurin.Complement Component 5 Mediates Development of Fibrosis, via Activation of Stellate Cells, in 2 Mouse Models of Chronic Pancreatitis Inflammation in acute and chronic pancreatitis.Alternatively activated macrophages promote pancreatic fibrosis in chronic pancreatitis.Vagal nerve stimulation blocks interleukin 6-dependent synaptic hyperexcitability induced by lipopolysaccharide-induced acute stress in the rodent prefrontal cortex.CXCR2 inhibition suppresses acute and chronic pancreatic inflammation Targeting STAT3 signaling reduces immunosuppressive myeloid cells in head and neck squamous cell carcinoma Mechanism of mitochondrial permeability transition pore induction and damage in the pancreas: inhibition prevents acute pancreatitis by protecting production of ATP.One compound of saponins from Disocorea zingiberensis protected against experimental acute pancreatitis by preventing mitochondria-mediated necrosis.New insights into the pathogenesis of pancreatitis.Pathogenic mechanisms of pancreatitis.Early phase of acute pancreatitis: Assessment and management.IL-6 biology: implications for clinical targeting in rheumatic disease.ADAM-family metalloproteinases in lung inflammation: potential therapeutic targets.Local and Systemic Expression of Immunomodulatory Factors in Chronic Pancreatitis.Do Animal Models of Acute Pancreatitis Reproduce Human Disease?Pathophysiological mechanisms in acute pancreatitis: Current understanding.The Role of Plant-derived Products in Pancreatitis: Experimental and Clinical Evidence.Continuous venovenous hemofiltration decreases mortality and ameliorates acute lung injury in canine model of severe salt water drowning.The Soluble Interleukin 6 Receptor: Advanced Therapeutic Options in Inflammation.High attenuation areas on chest computed tomography in community-dwelling adults: the MESA study.Innate Lymphoid Cells Are the Predominant Source of IL-17A during the Early Pathogenesis of Acute Respiratory Distress Syndrome.Diabetes increases pancreatitis induced systemic inflammation but has little effect on inflammation and cell death in the lung.Traditional Chinese medicine, Qing Ying Tang, ameliorates the severity of acute lung injury induced by severe acute pancreatitis in rats via the upregulation of aquaporin-1.Human thrombin-derived host defense peptides inhibit neutrophil recruitment and tissue injury in severe acute pancreatitis.Valproic acid attenuates lipopolysaccharide-induced acute lung injury in mice.

P2860

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P2860

IL-6 trans-signaling promotes pancreatitis-associated lung injury and lethality.

http://www.wikidata.org/entity/Q36638151

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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2013年论文

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2013年论文

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name

IL-6 trans-signaling promotes pancreatitis-associated lung injury and lethality.

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IL-6 trans-signaling promotes pancreatitis-associated lung injury and lethality.

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type

label

IL-6 trans-signaling promotes pancreatitis-associated lung injury and lethality.

@ast

IL-6 trans-signaling promotes pancreatitis-associated lung injury and lethality.

@en

prefLabel

IL-6 trans-signaling promotes pancreatitis-associated lung injury and lethality.

@ast

IL-6 trans-signaling promotes pancreatitis-associated lung injury and lethality.

@en

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Journal of Clinical Investigation

P1476

IL-6 trans-signaling promotes pancreatitis-associated lung injury and lethality.

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P2093

Akihiko Yoshimura

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Björn Rabe

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Dieter Saur

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Gregor Weirich

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Hong Zhang

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Liang Song

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Magdalena U Kurkowski

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Marina Lesina

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Matthias Treiber

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Patrick Neuhöfer

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P2860

Hereditary pancreatitis is caused by a mutation in the cationic trypsinogen gene

Therapeutic strategies for the clinical blockade of IL-6/gp130 signaling

Mutations in the gene encoding the serine protease inhibitor, Kazal type 1 are associated with chronic pancreatitis

Soluble gp130 is the natural inhibitor of soluble interleukin-6 receptor transsignaling responses

STATs in cancer inflammation and immunity: a leading role for STAT3

The pathophysiology and treatment of sepsis

Selective chemical probe inhibitor of Stat3, identified through structure-based virtual screening, induces antitumor activity.

Sepsis and the systemic inflammatory response syndrome.

Hepatic acute-phase proteins control innate immune responses during infection by promoting myeloid-derived suppressor cell function.

Role of interleukin-6 in acute pancreatitis. Comparison with C-reactive protein and phospholipase A.

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1019-1031

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10.1172/JCI64931

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3

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123

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Joseph P Mizgerd

Henrik Thorlacius

Stefan Rose-John

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2013-02-15T00:00:00Z

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235681810

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23426178

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3582130

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