EGFR Signaling Enhances Aerobic Glycolysis in Triple-Negative Breast Cancer Cells to Promote Tumor Growth and Immune Escape.
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Current advances in biomarkers for targeted therapy in triple-negative breast cancerKey Players in Choline Metabolic Reprograming in Triple-Negative Breast CancerThe Human Kinome Targeted by FDA Approved Multi-Target Drugs and Combination Products: A Comparative Study from the Drug-Target Interaction Network PerspectiveReversing EGFR Mediated Immunoescape by Targeted Monoclonal Antibody TherapyRacial disparity in metabolic regulation of cancer.Palmitoylation: a protein S-acylation with implications for breast cancer.Prognostic Significance of the Metabolic Marker Hexokinase-2 in Various Solid Tumors: A Meta-Analysis.Extracellular PKM2 induces cancer proliferation by activating the EGFR signaling pathway.Spheroid cancer stem cells display reprogrammed metabolism and obtain energy by actively running the tricarboxylic acid (TCA) cycleHarnessing the power of proteomics for identification of oncogenic, druggable signalling pathways in cancer.The BRCA1ness signature is associated significantly with response to PARP inhibitor treatment versus control in the I-SPY 2 randomized neoadjuvant setting.Comparison of FDA Approved Kinase Targets to Clinical Trial Ones: Insights from Their System Profiles and Drug-Target Interaction Networks.Acceleration of pancreatic tumorigenesis under immunosuppressive microenvironment induced by Reg3g overexpressionFructose-1,6-bisphosphate couples glycolytic flux to activation of Ras.Plasma TuM2-PK correlates with tumor size, CRP and CA 15-3 in metastatic breast carcinomas; short versus long term follow up study of the Egyptian breast cancer patients.Prognostic significance of metabolic enzyme pyruvate kinase M2 in breast cancer: A meta-analysis.Emerging functions of the EGFR in cancer.Stress hormones promote EGFR inhibitor resistance in NSCLC: Implications for combinations with β-blockers.Metabolic advantages and vulnerabilities in brain metastases.Metabolic Footprints and Molecular Subtypes in Breast Cancer.The relevance of tyrosine kinase inhibitors for global metabolic pathways in cancer.Immunotherapeutic interventions of Triple Negative Breast Cancer.Pan-cancer deconvolution of tumour composition using DNA methylationConstruction and analysis of mRNA, miRNA, lncRNA, and TF regulatory networks reveal the key genes associated with prostate cancerMetabolic Targeting of Breast Cancer Cells With the 2-Deoxy-D-Glucose and the Mitochondrial Bioenergetics Inhibitor MDIVI-1EGFR may participate in immune evasion through regulation of B7‑H5 expression in non‑small cell lung carcinoma
P2860
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P2860
EGFR Signaling Enhances Aerobic Glycolysis in Triple-Negative Breast Cancer Cells to Promote Tumor Growth and Immune Escape.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
EGFR Signaling Enhances Aerobi ...... umor Growth and Immune Escape.
@ast
EGFR Signaling Enhances Aerobi ...... umor Growth and Immune Escape.
@en
type
label
EGFR Signaling Enhances Aerobi ...... umor Growth and Immune Escape.
@ast
EGFR Signaling Enhances Aerobi ...... umor Growth and Immune Escape.
@en
prefLabel
EGFR Signaling Enhances Aerobi ...... umor Growth and Immune Escape.
@ast
EGFR Signaling Enhances Aerobi ...... umor Growth and Immune Escape.
@en
P2093
P2860
P1433
P1476
EGFR Signaling Enhances Aerobi ...... umor Growth and Immune Escape.
@en
P2093
Aysegul Sahin
Chia-Wei Li
Chung-Hsuan Chen
Daniel Raftery
Danijel Djukovic
Gabriel N Hortobagyi
Heng-Huan Lee
Hung-Ling Wang
Jennifer L Hsu
P2860
P304
P356
10.1158/0008-5472.CAN-15-2478
P407
P577
2016-01-12T00:00:00Z