Prenatal nicotine exposure increases heart susceptibility to ischemia/reperfusion injury in adult offspring. - Wikidata - awgldk - TriplyDB

Prenatal nicotine exposure increases heart susceptibility to ischemia/reperfusion injury in adult offspring.

Prenatal nicotine exposure increases heart susceptibility to ischemia/reperfusion injury in adult offspring.

http://www.wikidata.org/entity/Q36647715

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Is there evidence for potential harm of electronic cigarette use in pregnancy?Primary graft dysfunction: lessons learned about the first 72 h after lung transplantation Developmental consequences of fetal exposure to drugs: what we know and what we still must learn Repeated exposure to methamphetamine induces sex-dependent hypersensitivity to ischemic injury in the adult rat heart.The oxygen-rich postnatal environment induces cardiomyocyte cell-cycle arrest through DNA damage response Long-term consequences of fetal and neonatal nicotine exposure: a critical review Prenatal exposure to nicotine in pregnant rat increased inflammatory marker in newborn rat.Perinatal nicotine exposure increases angiotensin II receptor-mediated vascular contractility in adult offspring.Foetal nicotine exposure causes PKCε gene repression by promoter methylation in rat hearts.Hypoxia and fetal heart development.Antenatal nicotine induces heightened oxidative stress and vascular dysfunction in rat offspring Epigenetic mechanisms in developmental programming of adult disease.Prenatal nicotine increases matrix metalloproteinase 2 (MMP-2) expression in fetal guinea pig hearts Protective Effect of Antenatal Antioxidant on Nicotine-Induced Heart Ischemia-Sensitive Phenotype in Rat Offspring Norepinephrine causes epigenetic repression of PKCε gene in rodent hearts by activating Nox1-dependent reactive oxygen species production.Developmental nicotine exposure results in programming of alveolar simplification and interstitial pulmonary fibrosis in adult male rats.Antenatal Antioxidant Prevents Nicotine-Mediated Hypertensive Response in Rat Adult Offspring Fetal hypoxia results in programming of aberrant angiotensin ii receptor expression patterns and kidney development Prenatal gender-related nicotine exposure increases blood pressure response to angiotensin II in adult offspring.Promoter methylation of Egr-1 site contributes to fetal hypoxia-mediated PKCε gene repression in the developing heart Estrogen normalizes perinatal nicotine-induced hypertensive responses in adult female rat offspring.Clinical risk factors for primary graft dysfunction after lung transplantation Perinatal nicotine exposure alters AT 1 and AT 2 receptor expression pattern in the brain of fetal and offspring rats.Prenatal cocaine exposure abolished ischemic preconditioning-induced protection in adult male rat hearts: role of PKCepsilon.The effect of fetal and neonatal nicotine exposure on renal development of AT(1) and AT(2) receptors.Prenatal hypoxia causes a sex-dependent increase in heart susceptibility to ischemia and reperfusion injury in adult male offspring: role of protein kinase C epsilon.Direct effect of cocaine on epigenetic regulation of PKCepsilon gene repression in the fetal rat heart.Inhibition of miRNA-210 reverses nicotine-induced brain hypoxic-ischemic injury in neonatal rats.Inhibition of Gata4 and Tbx5 by Nicotine-Mediated DNA Methylation in Myocardial Differentiation.Using Zebrafish to Implement a Course-Based Undergraduate Research Experience to Study Teratogenesis in Two Biology Laboratory Courses.Prenatal methamphetamine differentially alters myocardial sensitivity to ischemic injury in male and female adult hearts.Downregulation of miR-133 via MAPK/ERK signaling pathway involved in nicotine-induced cardiomyocyte apoptosis.Expression Changes of Apoptotic Genes in Tissues from Mice Exposed to Nicotine Role of DNA methylation in perinatal nicotine-induced development of heart ischemia-sensitive phenotype in rat offspring.Neonatal Lipopolysaccharide Exposure Gender-Dependently Increases Heart Susceptibility to Ischemia/Reperfusion Injury in Male Rats.Mesenchymal stem cells from preterm to term newborns undergo a significant switch from anaerobic glycolysis to the oxidative phosphorylation.Maternal nicotine exposure leads to decreased cardiac protein disulfide isomerase and impaired mitochondrial function in male rat offspring.Postnatal Cardiovascular Consequences in the Offspring of Pregnant Rats Exposed to Smoking and Smoking Cessation Pharmacotherapies.Does cardiac development provide heart research with novel therapeutic approaches?

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P2860

Prenatal nicotine exposure increases heart susceptibility to ischemia/reperfusion injury in adult offspring.

http://www.wikidata.org/entity/Q36647715

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Maryam Rejali

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Qin Xue

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Shumei Yang

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P2860

Increased particulate partitioning of PKC epsilon reverses susceptibility of phospholamban knockout hearts to ischemic injury

Fetal nicotine or cocaine exposure: which one is worse?

Ischemic preconditioning translocates PKC-delta and -epsilon, which mediate functional protection in isolated rat heart

Estrogen deficiency decreases ischemic tolerance in the aged rat heart: Roles of PKCdelta, PKCepsilon, Akt, and GSK3beta

Heart-targeted overexpression of caspase3 in mice increases infarct size and depresses cardiac function.

Apoptosis and heart failure: A critical review of the literature.

Prenatal cocaine exposure increases apoptosis of neonatal rat heart and heart susceptibility to ischemia-reperfusion injury in 1-month-old rat.

Intrauterine undernutrition--renal and vascular origin of hypertension.

Opposing roles of delta and epsilonPKC in cardiac ischemia and reperfusion: targeting the apoptotic machinery.

Prenatal adverse effects of nicotine on the developing brain.

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reperfusion injury

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