The role of vesicular stomatitis virus matrix protein in inhibition of host-directed gene expression is genetically separable from its function in virus assembly.
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The cell-rounding activity of the vesicular stomatitis virus matrix protein is due to the induction of cell deathResistance to Rhabdoviridae Infection and Subversion of Antiviral ResponsesVesicular stomatitis virus as a flexible platform for oncolytic virotherapy against cancerRhabdovirus Matrix Protein Structures Reveal a Novel Mode of Self-AssociationCryo-EM Model of the Bullet-Shaped Vesicular Stomatitis VirusUnderstanding and altering cell tropism of vesicular stomatitis virusThe matrix protein of vesicular stomatitis virus inhibits nucleocytoplasmic transport when it is in the nucleus and associated with nuclear pore complexes.Combining Oncolytic Virotherapy with p53 Tumor Suppressor Gene TherapyVesicular stomatitis viruses expressing wild-type or mutant M proteins activate apoptosis through distinct pathways.Protection against lethal vaccinia virus challenge by using an attenuated matrix protein mutant vesicular stomatitis virus vaccine vector expressing poxvirus antigens.Effect of vesicular stomatitis virus matrix protein on transcription directed by host RNA polymerases I, II, and III.Infectious hematopoietic necrosis virus matrix protein inhibits host-directed gene expression and induces morphological changes of apoptosis in cell culturesInhibition of host transcription by vesicular stomatitis virus involves a novel mechanism that is independent of phosphorylation of TATA-binding protein (TBP) or association of TBP with TBP-associated factor subunits.Matrix protein and another viral component contribute to induction of apoptosis in cells infected with vesicular stomatitis virus.Cytopathogenesis and inhibition of host gene expression by RNA viruses.Interplay between innate immunity and negative-strand RNA viruses: towards a rational modelComplexes of vesicular stomatitis virus matrix protein with host Rae1 and Nup98 involved in inhibition of host transcription.Ability of the matrix protein of vesicular stomatitis virus to suppress beta interferon gene expression is genetically correlated with the inhibition of host RNA and protein synthesis.Contrasting effects of matrix protein on apoptosis in HeLa and BHK cells infected with vesicular stomatitis virus are due to inhibition of host gene expression.Oncolytic vesicular stomatitis virus induces apoptosis in U87 glioblastoma cells by a type II death receptor mechanism and induces cell death and tumor clearance in vivoMutations in the glycoprotein of vesicular stomatitis virus affect cytopathogenicity: potential for oncolytic virotherapy.Impact of Vesicular Stomatitis Virus M Proteins on Different Cellular FunctionsSuppression of the Sendai virus M protein through a novel short interfering RNA approach inhibits viral particle production but does not affect viral RNA synthesisOncolytic vesicular stomatitis virus induces apoptosis via signaling through PKR, Fas, and Daxx.Inducible and conditional inhibition of human immunodeficiency virus proviral expression by vesicular stomatitis virus matrix protein.The vesicular stomatitis virus matrix protein inhibits transcription from the human beta interferon promoterAnalysis of Venezuelan equine encephalitis virus capsid protein function in the inhibition of cellular transcriptionCytopathogenesis of vesicular stomatitis virus is regulated by the PSAP motif of M protein in a species-dependent manner.Effect of vesicular stomatitis virus matrix protein on host-directed translation in vivoPlasma membrane microdomains containing vesicular stomatitis virus M protein are separate from microdomains containing G protein and nucleocapsidsIn vitro and in vivo replication of influenza A H1N1 WSN33 viruses with different M1 proteins.hnRNPs Relocalize to the cytoplasm following infection with vesicular stomatitis virus.Vesicular stomatitis virus matrix protein mutations that affect association with host membranes and viral nucleocapsidsSuppression of antiviral innate immunity by sunitinib enhances oncolytic virotherapyInterferon response and viral evasion by members of the family rhabdoviridae.Role of Viral Hemorrhagic Septicemia Virus Matrix (M) Protein in Suppressing Host Transcription.Amalgamating oncolytic viruses to enhance their safety, consolidate their killing mechanisms, and accelerate their spreadResistance of pancreatic cancer cells to oncolytic vesicular stomatitis virus: role of type I interferon signaling.Mutations in the PPPY motif of vesicular stomatitis virus matrix protein reduce virus budding by inhibiting a late step in virion releaseIdentification of two additional translation products from the matrix (M) gene that contribute to vesicular stomatitis virus cytopathology
P2860
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P2860
The role of vesicular stomatitis virus matrix protein in inhibition of host-directed gene expression is genetically separable from its function in virus assembly.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
The role of vesicular stomatit ...... ts function in virus assembly.
@ast
The role of vesicular stomatit ...... ts function in virus assembly.
@en
type
label
The role of vesicular stomatit ...... ts function in virus assembly.
@ast
The role of vesicular stomatit ...... ts function in virus assembly.
@en
prefLabel
The role of vesicular stomatit ...... ts function in virus assembly.
@ast
The role of vesicular stomatit ...... ts function in virus assembly.
@en
P2093
P2860
P1433
P1476
The role of vesicular stomatit ...... ts function in virus assembly.
@en
P2093
P2860
P304
P407
P577
1993-08-01T00:00:00Z