Administration of the Nrf2-ARE activators sulforaphane and carnosic acid attenuates 4-hydroxy-2-nonenal-induced mitochondrial dysfunction ex vivo.
about
Carnosic acid protects against acetaminophen-induced hepatotoxicity by potentiating Nrf2-mediated antioxidant capacity in miceTBI and sex: crucial role of progesterone protecting the brain in an omega-3 deficient condition.Lipid peroxidation: production, metabolism, and signaling mechanisms of malondialdehyde and 4-hydroxy-2-nonenalTemporal and spatial dynamics of nrf2-antioxidant response elements mediated gene targets in cortex and hippocampus after controlled cortical impact traumatic brain injury in mice.Efficacy of some non-conventional herbal medications (sulforaphane, tanshinone IIA, and tetramethylpyrazine) in inducing neuroprotection in comparison with interleukin-10 after spinal cord injury: A meta-analysis.K(ATP) channel opener diazoxide prevents neurodegeneration: a new mechanism of action via antioxidative pathway activationNrf2-ARE activator carnosic acid decreases mitochondrial dysfunction, oxidative damage and neuronal cytoskeletal degradation following traumatic brain injury in mice.Long-Term Consequences of Traumatic Brain Injury: Current Status of Potential Mechanisms of Injury and Neurological OutcomesEnhanced 4-hydroxynonenal resistance in KEAP1 silenced human colon cancer cellsSulforaphane decreases endothelial cell apoptosis in fuchs endothelial corneal dystrophy: a novel treatment.Anti-neuropathic effects of Rosmarinus officinalis L. terpenoid fraction: relevance of nicotinic receptors.Evidence to support mitochondrial neuroprotection, in severe traumatic brain injury.The emerging role of Nrf2 in mitochondrial functionThe impact of oxidative stress and inflammation on RPE degeneration in non-neovascular AMD.Lipid peroxidation biomarkers in adolescents with or at high-risk for bipolar disorder.Carnosic acid induces autophagic cell death through inhibition of the Akt/mTOR pathway in human hepatoma cells.In vitro assessment of the indirect antioxidant activity of Sulforaphane in redox imbalance vanadium-induced.Sulforaphane prevents quinolinic acid-induced mitochondrial dysfunction in rat striatum.Myricetin ameliorates brain injury and neurological deficits via Nrf2 activation after experimental stroke in middle-aged rats.Carnosic Acid as a Promising Agent in Protecting Mitochondria of Brain Cells.Chronic traumatic encephalopathy-integration of canonical traumatic brain injury secondary injury mechanisms with tau pathology.Carnosic acid attenuates RANKL-induced oxidative stress and osteoclastogenesis via induction of Nrf2 and suppression of NF-κB and MAPK signalling.
P2860
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P2860
Administration of the Nrf2-ARE activators sulforaphane and carnosic acid attenuates 4-hydroxy-2-nonenal-induced mitochondrial dysfunction ex vivo.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Administration of the Nrf2-ARE ...... chondrial dysfunction ex vivo.
@ast
Administration of the Nrf2-ARE ...... chondrial dysfunction ex vivo.
@en
type
label
Administration of the Nrf2-ARE ...... chondrial dysfunction ex vivo.
@ast
Administration of the Nrf2-ARE ...... chondrial dysfunction ex vivo.
@en
prefLabel
Administration of the Nrf2-ARE ...... chondrial dysfunction ex vivo.
@ast
Administration of the Nrf2-ARE ...... chondrial dysfunction ex vivo.
@en
P2093
P2860
P1476
Administration of the Nrf2-ARE ...... chondrial dysfunction ex vivo.
@en
P2093
Darren M Miller
Edward D Hall
Indrapal N Singh
Juan A Wang
P2860
P356
10.1016/J.FREERADBIOMED.2012.12.011
P577
2012-12-27T00:00:00Z