AP214, an analogue of alpha-melanocyte-stimulating hormone, ameliorates sepsis-induced acute kidney injury and mortality. - Wikidata - awgldk - TriplyDB

AP214, an analogue of alpha-melanocyte-stimulating hormone, ameliorates sepsis-induced acute kidney injury and mortality.

AP214, an analogue of alpha-melanocyte-stimulating hormone, ameliorates sepsis-induced acute kidney injury and mortality.

http://www.wikidata.org/entity/Q36681505

about

The melanocortin MC(1) receptor agonist BMS-470539 inhibits leucocyte trafficking in the inflamed vasculature Synchronous recruitment of epigenetic modifiers to endotoxin synergistically activated Tnf-α gene in acute kidney injury Effects of continuous erythropoietin receptor activator in sepsis-induced acute kidney injury and multi-organ dysfunction Animal models of sepsis and sepsis-induced kidney injury Melanocortin-1 Receptor Polymorphisms and the Risk of Complicated Sepsis After Trauma: A Candidate Gene Association Study.The melanocortin agonist AP214 exerts anti-inflammatory and proresolving properties Hemodynamic changes in the kidney in a pediatric rat model of sepsis-induced acute kidney injury.Sirt1 deletion leads to enhanced inflammation and aggravates endotoxin-induced acute kidney injury Calpastatin controls polymicrobial sepsis by limiting procoagulant microparticle release High-dose ascorbate with low-dose amphotericin B attenuates severity of disease in a model of the reappearance of candidemia during sepsis in the mouse.Cecal ligation puncture procedure.Development of oxidative stress in the peritubular capillary microenvironment mediates sepsis-induced renal microcirculatory failure and acute kidney injury.Chronic kidney disease worsens sepsis and sepsis-induced acute kidney injury by releasing High Mobility Group Box Protein-1 Development of intestinal ischemia/reperfusion-induced acute kidney injury in rats with or without chronic kidney disease: Cytokine/chemokine response and effect of α-melanocyte-stimulating hormone.Experimental evidence that preexisting chronic kidney disease is a risk factor for acute kidney injury Sesamol ameliorates hypotension by modulating cytokines and PPAR-gamma in systemic inflammatory response Adrenocorticotropic hormone ameliorates acute kidney injury by steroidogenic-dependent and -independent mechanisms A two-hit mechanism for sepsis-induced impairment of renal tubule function Curbing Inflammation through Endogenous Pathways: Focus on Melanocortin Peptides Use of Biotelemetry to Define Physiology-Based Deterioration Thresholds in a Murine Cecal Ligation and Puncture Model of Sepsis.Methyl-2-acetamidoacrylate, an ethyl pyruvate analog, decreases sepsis-induced acute kidney injury in mice.Pre-existing renal disease promotes sepsis-induced acute kidney injury and worsens outcome Reduced production of creatinine limits its use as marker of kidney injury in sepsis ABT-719 for the Prevention of Acute Kidney Injury in Patients Undergoing High-Risk Cardiac Surgery: A Randomized Phase 2b Clinical Trial.Class B scavenger receptor types I and II and CD36 targeting improves sepsis survival and acute outcomes in mice.Renoprotective approaches and strategies in acute kidney injury Neuropeptide receptors as potential drug targets in the treatment of inflammatory conditions Leveraging melanocortin pathways to treat glomerular diseases.Clinical and Experimental Sepsis Impairs CD8 T-Cell-Mediated Immunity.Mediators of inflammation in acute kidney injury.Osthole protects sepsis-induced acute kidney injury via down-regulating NF-κB signal pathway.Challenges and advances in the treatment of AKI.Pharmacological targets in the renal peritubular microenvironment: implications for therapy for sepsis-induced acute kidney injury.Direct and indirect antimicrobial activities of neuropeptides and their therapeutic potential.Designing drugs that combat kidney damage.Recent early clinical drug development for acute kidney injury.Alpha-melanocyte stimulating hormone ameliorates disease activity in an induced murine lupus-like model.Enhanced Cationic Charge is a Key Factor in Promoting Staphylocidal Activity of α-Melanocyte Stimulating Hormone via Selective Lipid Affinity.Inhibition of NF-kappaB ameliorates sepsis-induced downregulation of aquaporin-2/V2 receptor expression and acute renal failure in vivo.Neuropeptide downregulation in sepsis.

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AP214, an analogue of alpha-melanocyte-stimulating hormone, ameliorates sepsis-induced acute kidney injury and mortality.

http://www.wikidata.org/entity/Q36681505

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P2860

Exocrine gland dysfunction in MC5-R-deficient mice: evidence for coordinated regulation of exocrine gland function by melanocortin peptides

The epidemiology of sepsis in the United States from 1979 through 2000

The pathophysiology and treatment of sepsis

Molecular control of immune/inflammatory responses: interactions between nuclear factor-kappa B and steroid receptor-signaling pathways.

Evidence of autocrine modulation of macrophage nitric oxide synthase by alpha-melanocyte-stimulating hormone.

Effect of treatment with low doses of hydrocortisone and fludrocortisone on mortality in patients with septic shock.

In vivo delivery of caspase-8 or Fas siRNA improves the survival of septic mice.

Animal models of sepsis: setting the stage.

Sepsis-induced organ failure is mediated by different pathways in the kidney and liver: acute renal failure is dependent on MyD88 but not renal cell apoptosis.

Simvastatin improves sepsis-induced mortality and acute kidney injury via renal vascular effects

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