Sepsis-induced organ failure is mediated by different pathways in the kidney and liver: acute renal failure is dependent on MyD88 but not renal cell apoptosis.
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TLR2, TLR4 and the MYD88 signaling pathway are crucial for neutrophil migration in acute kidney injury induced by sepsisSynchronous recruitment of epigenetic modifiers to endotoxin synergistically activated Tnf-α gene in acute kidney injuryElectroacupuncture Ameliorates Acute Renal Injury in Lipopolysaccharide-Stimulated Rabbits via Induction of HO-1 through the PI3K/Akt/Nrf2 PathwaysSepsis: in search of cure.Animal models of sepsis and sepsis-induced kidney injuryEndotoxin uptake by S1 proximal tubular segment causes oxidative stress in the downstream S2 segment.Dominant role of the MyD88-dependent signaling pathway in mediating early endotoxin-induced murine ileusToll-like receptor 2 deficiency results in impaired antibody responses and septic shock during Borrelia hermsii infection.Oxidative stress and modification of renal vascular permeability are associated with acute kidney injury during P. berghei ANKA infection.A novel 1,2-benzenediamine derivative FC-99 suppresses TLR3 expression and ameliorates disease symptoms in a mouse model of sepsisCirculating microRNA-223 serum levels do not predict sepsis or survival in patients with critical illness.Sirt1 deletion leads to enhanced inflammation and aggravates endotoxin-induced acute kidney injuryPGC-1α promotes recovery after acute kidney injury during systemic inflammation in mice.Tubular cross talk in acute kidney injury: a story of sense and sensibilityApoptosis and acute kidney injurySeptic acute kidney injury: molecular mechanisms and the importance of stratification and targeting therapy.Liver proteomics for therapeutic drug discovery: inhibition of the cyclophilin receptor CD147 attenuates sepsis-induced acute renal failureChloroquine and inhibition of Toll-like receptor 9 protect from sepsis-induced acute kidney injury.TLR4 signaling mediates inflammation and tissue injury in nephrotoxicity.AP214, an analogue of alpha-melanocyte-stimulating hormone, ameliorates sepsis-induced acute kidney injury and mortality.Lipopolysaccharide clearance, bacterial clearance, and systemic inflammatory responses are regulated by cell type-specific functions of TLR4 during sepsis.Could Biomarkers Direct Therapy for the Septic Patient?Methyl-2-acetamidoacrylate, an ethyl pyruvate analog, decreases sepsis-induced acute kidney injury in mice.Pre-existing renal disease promotes sepsis-induced acute kidney injury and worsens outcomeRole of Mitochondrial DNA in Septic AKI via Toll-Like Receptor 9.Renal hypoxia and dysoxia after reperfusion of the ischemic kidney.Mitochondrial mechanisms of sepsis-induced organ failure.Reduced production of creatinine limits its use as marker of kidney injury in sepsisBRG1 increases transcription of proinflammatory genes in renal ischemia.Class B scavenger receptor types I and II and CD36 targeting improves sepsis survival and acute outcomes in mice.Pathophysiology of acute kidney injuryAcute kidney injury: current perspectives.New roles for innate immune response in acute and chronic kidney injuries.Pharmacological targets in the renal peritubular microenvironment: implications for therapy for sepsis-induced acute kidney injury.Animal models of renal dysfunction: acute kidney injury.Acute toll-like receptor 4 activation impairs rat renal microvascular autoregulatory behaviour.Physiological aspects of Toll-like receptor 4 activation in sepsis-induced acute kidney injury.Insight into the beneficial immunomodulatory mechanism of the sevoflurane metabolite hexafluoro-2-propanol in a rat model of endotoxaemia.Delayed Mitogen-Activated Protein Kinase/Extracellular Signal-Regulated Kinase Inhibition by Trametinib Attenuates Systemic Inflammatory Responses and Multiple Organ Injury in Murine Sepsis.Cl-Amidine Prevents Histone 3 Citrullination and Neutrophil Extracellular Trap Formation, and Improves Survival in a Murine Sepsis Model.
P2860
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P2860
Sepsis-induced organ failure is mediated by different pathways in the kidney and liver: acute renal failure is dependent on MyD88 but not renal cell apoptosis.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Sepsis-induced organ failure i ...... but not renal cell apoptosis.
@ast
Sepsis-induced organ failure i ...... but not renal cell apoptosis.
@en
type
label
Sepsis-induced organ failure i ...... but not renal cell apoptosis.
@ast
Sepsis-induced organ failure i ...... but not renal cell apoptosis.
@en
prefLabel
Sepsis-induced organ failure i ...... but not renal cell apoptosis.
@ast
Sepsis-induced organ failure i ...... but not renal cell apoptosis.
@en
P2093
P2860
P356
P1433
P1476
Sepsis-induced organ failure i ...... 8 but not renal cell apoptosis
@en
P2093
P2860
P2888
P304
P356
10.1038/SJ.KI.5000165
P407
P577
2006-03-01T00:00:00Z