AP-2β Is a Downstream Effector of PITX2 Required to Specify Endothelium and Establish Angiogenic Privilege During Corneal Development.
about
Genetic dissection of anterior segment dysgenesis caused by a Col4a1 mutation in mouse.Conditional deletion of AP-2β in mouse cranial neural crest results in anterior segment dysgenesis and early-onset glaucoma.Foxc1 and Foxc2 in the Neural Crest Are Required for Ocular Anterior Segment Development.Restricted Presence of POU6F2 in Human Corneal Endothelial Cells Uncovered by Extension of the Promoter-level Expression Atlas.PITX2 deficiency and associated human disease: insights from the zebrafish model.
P2860
AP-2β Is a Downstream Effector of PITX2 Required to Specify Endothelium and Establish Angiogenic Privilege During Corneal Development.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
AP-2β Is a Downstream Effector ...... ge During Corneal Development.
@ast
AP-2β Is a Downstream Effector ...... ge During Corneal Development.
@en
type
label
AP-2β Is a Downstream Effector ...... ge During Corneal Development.
@ast
AP-2β Is a Downstream Effector ...... ge During Corneal Development.
@en
prefLabel
AP-2β Is a Downstream Effector ...... ge During Corneal Development.
@ast
AP-2β Is a Downstream Effector ...... ge During Corneal Development.
@en
P2093
P2860
P356
P1476
AP-2β Is a Downstream Effector ...... ege During Corneal Development
@en
P2093
Judith West-Mays
Lisheng Chen
Philip J Gage
Trevor Williams
Vanessa Martino
P2860
P304
P356
10.1167/IOVS.15-18103
P407
P577
2016-03-01T00:00:00Z