Targeted deletion of AP-2alpha leads to disruption in corneal epithelial cell integrity and defects in the corneal stroma
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The keratocyte: corneal stromal cell with variable repair phenotypesCell autonomous roles for AP-2alpha in lens vesicle separation and maintenance of the lens epithelial cell phenotypeAP-2alpha knockout mice exhibit optic cup patterning defects and failure of optic stalk morphogenesisBasonuclin-null mutation impairs homeostasis and wound repair in mouse corneal epitheliumConditional deletion of activating protein 2alpha (AP-2alpha) in the developing retina demonstrates non-cell-autonomous roles for AP-2alpha in optic cup developmentConditional deletion of the mouse Klf4 gene results in corneal epithelial fragility, stromal edema, and loss of conjunctival goblet cellsRybp, a polycomb complex-associated protein, is required for mouse eye development.Mouse conjunctival forniceal gene expression during postnatal development and its regulation by Kruppel-like factor 4.Regulation of corneal epithelial barrier function by Kruppel-like transcription factor 4.Critical role of Klf5 in regulating gene expression during post-eyelid opening maturation of mouse corneas.Identification of candidate Klf4 target genes reveals the molecular basis of the diverse regulatory roles of Klf4 in the mouse cornea.Hemizygous Le-Cre transgenic mice have severe eye abnormalities on some genetic backgrounds in the absence of LoxP sitesAP-2β Is a Downstream Effector of PITX2 Required to Specify Endothelium and Establish Angiogenic Privilege During Corneal Development.Matrix Metalloproteinases as Mediators of Primary and Secondary CataractsOverlapping expression patterns and redundant roles for AP-2 transcription factors in the developing mammalian retina.Conditional deletion of AP-2β in mouse cranial neural crest results in anterior segment dysgenesis and early-onset glaucoma.Conditional deletion of Cited2 results in defective corneal epithelial morphogenesis and maintenance.Ocular surface development and gene expression.Conditional disruption of mouse Klf5 results in defective eyelids with malformed meibomian glands, abnormal cornea and loss of conjunctival goblet cells.Regulation of mouse lens maturation and gene expression by Krüppel-like factor 4AP-2ε Expression in Developing Retina: Contributing to the Molecular Diversity of Amacrine Cells.
P2860
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P2860
Targeted deletion of AP-2alpha leads to disruption in corneal epithelial cell integrity and defects in the corneal stroma
description
2005 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2005
@ast
im Oktober 2005 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2005/10/01)
@sk
vědecký článek publikovaný v roce 2005
@cs
wetenschappelijk artikel (gepubliceerd op 2005/10/01)
@nl
наукова стаття, опублікована в жовтні 2005
@uk
مقالة علمية (نشرت في أكتوبر 2005)
@ar
name
Targeted deletion of AP-2alpha ...... defects in the corneal stroma
@ast
Targeted deletion of AP-2alpha ...... defects in the corneal stroma
@en
Targeted deletion of AP-2alpha ...... defects in the corneal stroma
@nl
type
label
Targeted deletion of AP-2alpha ...... defects in the corneal stroma
@ast
Targeted deletion of AP-2alpha ...... defects in the corneal stroma
@en
Targeted deletion of AP-2alpha ...... defects in the corneal stroma
@nl
prefLabel
Targeted deletion of AP-2alpha ...... defects in the corneal stroma
@ast
Targeted deletion of AP-2alpha ...... defects in the corneal stroma
@en
Targeted deletion of AP-2alpha ...... defects in the corneal stroma
@nl
P2093
P2860
P356
P1476
Targeted deletion of AP-2alpha ...... defects in the corneal stroma
@en
P2093
Dhruva J. Dwivedi
Giuseppe F. Pontoriero
Judith A. West-Mays
Shelley Sullivan
Trevor Williams
P2860
P304
P356
10.1167/IOVS.05-0028
P407
P577
2005-10-01T00:00:00Z