Fusion properties of cells persistently infected with human parainfluenza virus type 3: participation of hemagglutinin-neuraminidase in membrane fusion
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Entry of parainfluenza virus into cells as a target for interrupting childhood respiratory diseaseParainfluenza virusesUnity in diversity: shared mechanism of entry among paramyxovirusesAttenuation of Murray Valley encephalitis virus by site-directed mutagenesis of the hinge and putative receptor-binding regions of the envelope protein.Interaction between the Hemagglutinin-Neuraminidase and Fusion Glycoproteins of Human Parainfluenza Virus Type III Regulates Viral Growth In VivoThe anti-influenza virus agent 4-GU-DANA (zanamivir) inhibits cell fusion mediated by human parainfluenza virus and influenza virus HA.Perspective: emerging challenges in the treatment of influenza and parainfluenza in transplant patientsInhibition of Nipah virus infection in vivo: targeting an early stage of paramyxovirus fusion activation during viral entryInfluence of the human parainfluenza virus 3 attachment protein's neuraminidase activity on its capacity to activate the fusion protein.A single amino acid change in the Newcastle disease virus fusion protein alters the requirement for HN protein in fusionMechanism of interference mediated by human parainfluenza virus type 3 infection.Contribution of the human parainfluenza virus type 3 HN-receptor interaction to pathogenesis in vivo.Subacute sclerosing panencephalitis virus dominantly interferes with replication of wild-type measles virus in a mixed infection: implication for viral persistence.A recombinant sialidase fusion protein effectively inhibits human parainfluenza viral infection in vitro and in vivo.Viral entry inhibitors targeted to the membrane site of action.Inhibition of receptor binding stabilizes Newcastle disease virus HN and F protein-containing complexes.Triggering of human parainfluenza virus 3 fusion protein (F) by the hemagglutinin-neuraminidase (HN) protein: an HN mutation diminishes the rate of F activation and fusionInhibition of hendra virus fusion.The aberrant gene-end transcription signal of the matrix M gene of human parainfluenza virus type 3 downregulates fusion F protein expression and the F-specific antibody response in vivoElectron tomography imaging of surface glycoproteins on human parainfluenza virus 3: association of receptor binding and fusion proteins before receptor engagementThe hemagglutinin envelope protein of canine distemper virus (CDV) confers cell tropism as illustrated by CDV and measles virus complementation analysis.Localization of monoclonal antibody epitopes and functional domains in the hemagglutinin protein of measles virus.Quantitative measurement of paramyxovirus fusion: differences in requirements of glycoproteins between simian virus 5 and human parainfluenza virus 3 or Newcastle disease virus.Association of the parainfluenza virus fusion and hemagglutinin-neuraminidase glycoproteins on cell surfaces.Identification of a cytoplasmic Tyr-X-X-Leu motif essential for down regulation of the human cell receptor CD46 in persistent measles virus infectionFusion promotion by a paramyxovirus hemagglutinin-neuraminidase protein: pH modulation of receptor avidity of binding sites I and IIMolecular determinants of antiviral potency of paramyxovirus entry inhibitors.Cell fusion by the envelope glycoproteins of persistent measles viruses which caused lethal human brain disease.Identification of an amino acid that defines the fusogenicity of mumps virus.Hemagglutinin-neuraminidase enhances F protein-mediated membrane fusion of reconstituted Sendai virus envelopes with cellsFusion properties of cells infected with human parainfluenza virus type 3: receptor requirements for viral spread and virus-mediated membrane fusion.Functional interactions between the fusion protein and hemagglutinin-neuraminidase of human parainfluenza viruses.Biological activity of paramyxovirus fusion proteins: factors influencing formation of syncytia.Role of thiol/disulfide exchange in newcastle disease virus entryKinetic dependence of paramyxovirus entry inhibition.Mutated form of the Newcastle disease virus hemagglutinin-neuraminidase interacts with the homologous fusion protein despite deficiencies in both receptor recognition and fusion promotion.Quantitative comparison of human parainfluenza virus hemagglutinin-neuraminidase receptor binding and receptor cleavageFixation of oligosaccharides to a surface may increase the susceptibility to human parainfluenza virus 1, 2, or 3 hemagglutinin-neuraminidase.Full conversion of the hemagglutinin-neuraminidase specificity of the parainfluenza virus 5 fusion protein by replacement of 21 amino acids in its head region with those of the simian virus 41 fusion protein.Recovery and characterization of a chimeric rinderpest virus with the glycoproteins of peste-des-petits-ruminants virus: homologous F and H proteins are required for virus viability.
P2860
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P2860
Fusion properties of cells persistently infected with human parainfluenza virus type 3: participation of hemagglutinin-neuraminidase in membrane fusion
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Fusion properties of cells per ...... uraminidase in membrane fusion
@ast
Fusion properties of cells per ...... uraminidase in membrane fusion
@en
type
label
Fusion properties of cells per ...... uraminidase in membrane fusion
@ast
Fusion properties of cells per ...... uraminidase in membrane fusion
@en
prefLabel
Fusion properties of cells per ...... uraminidase in membrane fusion
@ast
Fusion properties of cells per ...... uraminidase in membrane fusion
@en
P2860
P1433
P1476
Fusion properties of cells per ...... uraminidase in membrane fusion
@en
P2093
P2860
P304
P407
P577
1991-06-01T00:00:00Z