Genetic basis of the impaired renal myogenic response in FHH rats - Wikidata - awgldk - TriplyDB

Genetic basis of the impaired renal myogenic response in FHH rats

Genetic basis of the impaired renal myogenic response in FHH rats

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Renal autoregulation in health and disease Role of 20-HETE in the impaired myogenic and TGF responses of the Af-Art of Dahl salt-sensitive rats.Zinc-finger nuclease knockout of dual-specificity protein phosphatase-5 enhances the myogenic response and autoregulation of cerebral blood flow in FHH.1BN rats.Bleeding Diathesis in Fawn Hooded Rats-Possible Implications for Invasive Procedures and Refinement Strategies.Elevated Aminopeptidase P Attenuates Cerebral Arterial Responses to Bradykinin in Fawn-Hooded Hypertensive Rats.Fluorescence dilution technique for measurement of albumin reflection coefficient in isolated glomeruli.Comparison of the Development Diabetic Induced Renal Disease in Strains of Goto-Kakizaki Rats Elevated K+ channel activity opposes vasoconstrictor response to serotonin in cerebral arteries of the Fawn Hooded Hypertensive rat.Enhanced large conductance K+ channel activity contributes to the impaired myogenic response in the cerebral vasculature of Fawn Hooded Hypertensive rats.Rat models of cardiometabolic diseases: baseline clinical chemistries, and rationale for their use in examining air pollution health effects.Knockdown of Add3 impairs the myogenic response of renal afferent arterioles and middle cerebral arteries.Impaired myogenic response of the afferent arteriole contributes to the increased susceptibility to renal disease in Milan normotensive rats.The real culprit behind diabetic nephropathy: impaired renal autoregulation?P2 receptors in renal autoregulation.Progression of hypertension and kidney disease in aging fawn-hooded rats is mediated by enhanced influence of renin-angiotensin system and suppression of nitric oxide system and epoxyeicosanoids.Cerebral Autoregulation in Hypertension and Ischemic Stroke: A Mini Review

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Genetic basis of the impaired renal myogenic response in FHH rats

http://www.wikidata.org/entity/Q36699831

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2012 nî lūn-bûn

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Genetic basis of the impaired renal myogenic response in FHH rats

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Genetic basis of the impaired renal myogenic response in FHH rats

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Genetic basis of the impaired renal myogenic response in FHH rats

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Genetic basis of the impaired renal myogenic response in FHH rats

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Genetic basis of the impaired renal myogenic response in FHH rats

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AJPRENAL.00404.2012

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American Journal of Physiology - Renal Physiology

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Genetic basis of the impaired renal myogenic response in FHH rats

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Allison Sarkis

http://www.w3.org/2001/XMLSchema#string

Fan Fan

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Howard J Jacob

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Jan Michael Williams

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Jozef Lazar

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Malikarjuna Pabbidi

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Marilyn Burke

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Richard J Roman

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Ruisheng Liu

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Ying Ge

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P2860

RBM20, a gene for hereditary cardiomyopathy, regulates titin splicing

Shoc2 is targeted to late endosomes and required for Erk1/2 activation in EGF-stimulated cells

Evidence for an interaction between adducin and Na(+)-K(+)-ATPase: relation to genetic hypertension

35H, a sequence isolated as a protein kinase C binding protein, is a novel member of the adducin family

Human non-synonymous SNPs: server and survey

The Rat Genome Database, update 2007--easing the path from disease to data and back again.

Signaling mechanisms underlying the vascular myogenic response.

Targeted deletion of the gamma-adducin gene (Add3) in mice reveals differences in alpha-adducin interactions in erythroid and nonerythroid cells.

Temporal characterization of the development of renal injury in FHH rats and FHH.1BN congenic strains.

γ-Adducin stimulates the thiazide-sensitive NaCl cotransporter

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10.1152/AJPRENAL.00404.2012

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5

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