Functions of BCL-X L at the Interface between Cell Death and Metabolism - Wikidata - awgldk - TriplyDB

Functions of BCL-X L at the Interface between Cell Death and Metabolism

Functions of BCL-X L at the Interface between Cell Death and Metabolism

http://www.wikidata.org/entity/Q36702748

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Mitochondrial dysfunction in amyotrophic lateral sclerosis - a valid pharmacological target?Identification of miRNA-7 by genome-wide analysis as a critical sensitizer for TRAIL-induced apoptosis in glioblastoma cells.Inherited thrombocytopenia: novel insights into megakaryocyte maturation, proplatelet formation and platelet lifespan.Oral Administration of Tualang and Manuka Honeys Modulates Breast Cancer Progression in Sprague-Dawley Rats Model.A let-7b binding site SNP in the 3'-UTR of the Bcl-xL gene enhances resistance to 5-fluorouracil and doxorubicin in breast cancer cells.Cytotoxicity of Eupatorium cannabinum L. ethanolic extract against colon cancer cells and interactions with Bisphenol A and Doxorubicin.PKD1 protein is involved in reactive oxygen species-mediated mitochondrial depolarization in cooperation with protein kinase Cδ (PKCδ).Group VIA Phospholipase A2 (iPLA2β) Modulates Bcl-x 5'-Splice Site Selection and Suppresses Anti-apoptotic Bcl-x(L) in β-Cells.The BH4 domain of anti-apoptotic Bcl-XL, but not that of the related Bcl-2, limits the voltage-dependent anion channel 1 (VDAC1)-mediated transfer of pro-apoptotic Ca2+ signals to mitochondria.Midbrain dopamine neurons in Parkinson's disease exhibit a dysregulated miRNA and target-gene network Antagonism between granulocytic maturation and deacetylase inhibitor-induced apoptosis in acute promyelocytic leukaemia cells Calcium-independent phospholipases A2 and their roles in biological processes and diseases.Knockout of Vdac1 activates hypoxia-inducible factor through reactive oxygen species generation and induces tumor growth by promoting metabolic reprogramming and inflammation.Dynamics of the BH3-Only Protein Binding Interface of Bcl-xL.Bcl-xL promotes metastasis independent of its anti-apoptotic activity.Molecular and Computational Studies on Apoptotic Pathway Regulator, Bcl-2 Gene from Breast Cancer Cell Line MCF-7.Application potential of toll-like receptors in cancer immunotherapy: Systematic review.Structural biology of the Bcl-2 family and its mimicry by viral proteins Tumor necrosis factor α-induced protein 8-like 1 promotes apoptosis by regulating B-cell leukemia/lymphoma-2 family proteins in RAW264.7 cells.Competition through dimerization between antiapoptotic and proapoptotic HS-1-associated protein X-1 (Hax-1).Mitochondrial dysfunction and mitophagy: the beginning and end to diabetic nephropathy?Modulation of alternative splicing by anticancer drugs.Bcl-2 proteins and calcium signaling: complexity beneath the surface.Acetogenins from Annona muricata as potential inhibitors of antiapoptotic proteins: a molecular modeling study Bisphenol A disrupts transcription and decreases viability in aging vascular endothelial cells In vivo inhibition of the mitochondrial H+-ATP synthase in neurons promotes metabolic preconditioning.Neural Stem Cell Transplantation Is Associated with Inhibition of Apoptosis, Bcl-xL Upregulation, and Recovery of Neurological Function in a Rat Model of Traumatic Brain Injury.Vascular mechanotransduction data in a rodent model of diabetes: Pressure-induced regulation of SHP2 and associated signaling in the rat inferior vena cava.BCL-XL binds and antagonizes RASSF6 tumor suppressor to suppress p53 expression.The T-cell fingerprint of MALT1 paracaspase revealed by selective inhibition.Signaling events associated with gonadotropin releasing hormone-agonist-induced hormonal castration and its reversal in canines.Evidence for anti-apoptotic roles of proteasome activator 28γ via inhibiting caspase activity.Highly efficient genome editing via CRISPR-Cas9 in human pluripotent stem cells is achieved by transient BCL-XL overexpression

P2860

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P2860

Functions of BCL-X L at the Interface between Cell Death and Metabolism

http://www.wikidata.org/entity/Q36702748

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

@zh-tw

2013年论文

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2013年论文

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2013年论文

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name

Functions of BCL-X L at the Interface between Cell Death and Metabolism

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Functions of BCL-X L at the Interface between Cell Death and Metabolism

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Functions of BCL-X L at the Interface between Cell Death and Metabolism

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Functions of BCL-X L at the Interface between Cell Death and Metabolism

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Functions of BCL-X L at the Interface between Cell Death and Metabolism

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Functions of BCL-X L at the Interface between Cell Death and Metabolism

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International Journal of Cell Biology

P1476

Functions of BCL-X L at the Interface between Cell Death and Metabolism

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P2093

Delphine Lissa

http://www.w3.org/2001/XMLSchema#string

Judith Michels

http://www.w3.org/2001/XMLSchema#string

Maria Castedo

http://www.w3.org/2001/XMLSchema#string

P2860

BNIP3 and genetic control of necrosis-like cell death through the mitochondrial permeability transition pore

p53 has a direct apoptogenic role at the mitochondria

Bcl-2 and Bcl-XL regulate proinflammatory caspase-1 activation by interaction with NALP1

Mixed lineage kinase domain-like protein mediates necrosis signaling downstream of RIP3 kinase

The mitochondrial phosphatase PGAM5 functions at the convergence point of multiple necrotic death pathways

Functional and physical interaction between Bcl-X(L) and a BH3-like domain in Beclin-1

Direct activation of Bax by p53 mediates mitochondrial membrane permeabilization and apoptosis

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

Bad, a heterodimeric partner for Bcl-XL and Bcl-2, displaces Bax and promotes cell death

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705294

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scholarly article

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10.1155/2013/705294

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2013

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Lorenzo Galluzzi

Laura Senovilla

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2013-02-28T00:00:00Z

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236084950

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23533418

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3603586

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