Distinguishing diabetic nephropathy from other causes of glomerulosclerosis: an update.
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A Systems Biology Overview on Human Diabetic Nephropathy: From Genetic Susceptibility to Post-Transcriptional and Post-Translational ModificationsHistological changes of kidney in diabetic nephropathyRedox Signaling in Diabetic Nephropathy: Hypertrophy versus Death Choices in Mesangial Cells and PodocytesRenal endothelial dysfunction in diabetic nephropathyAS101 prevents diabetic nephropathy progression and mesangial cell dysfunction: regulation of the AKT downstream pathwayGenetic variation in the matrix metalloproteinase genes and diabetic nephropathy in type 1 diabetes.Laminin α1 regulates age-related mesangial cell proliferation and mesangial matrix accumulation through the TGF-β pathwayA Comparison of Food-grade Folium mori ( Sāng Yè) Extract and 1-Deoxynojirimycin for Glycemic Control and Renal Function in Streptozotocin-induced Diabetic Rats.Prevention of diabetic nephropathy in Ins2(+/)⁻(AkitaJ) mice by the mitochondria-targeted therapy MitoQ.Fluorofenidone offers improved renoprotection at early interventions during the course of diabetic nephropathy in db/db mice via multiple pathwaysSimultaneous onset of steroid resistant nephrotic syndrome and IDDM in two young children.A modest decrease in endothelial NOS in mice comparable to that associated with human NOS3 variants exacerbates diabetic nephropathyFrom fibrosis to sclerosis: mechanisms of glomerulosclerosis in diabetic nephropathy.G-CSF prevents progression of diabetic nephropathy in ratIdiopathic nodular glomerulosclerosis in a never-smoking, normotensive, non-obese, normal-glucose-tolerant middle-aged womanSalutary effects of a novel oxidative stress modulator on adenine-induced chronic progressive tubulointerstitial nephropathyNew insights into the mechanisms of fibrosis and sclerosis in diabetic nephropathy.Heat-processed Panax ginseng and diabetic renal damage: active components and action mechanism.Reversal of Early Diabetic Nephropathy by Islet Transplantation under the Kidney Capsule in a Rat Model.Exercise and Beta-Glucan Consumption (Saccharomyces cerevisiae) Improve the Metabolic Profile and Reduce the Atherogenic Index in Type 2 Diabetic Rats (HFD/STZ).Glomerulonephropathy in aged captive Key Largo woodrats (Neotoma floridana smalli).Diabetic nephropathy: Treatment with phosphodiesterase type 5 inhibitors.Crosstalk between advanced glycation end products (AGEs)-receptor RAGE axis and dipeptidyl peptidase-4-incretin system in diabetic vascular complications.Hyperoside pre-treatment prevents glomerular basement membrane damage in diabetic nephropathy by inhibiting podocyte heparanase expression.Pentoxifylline for Diabetic Nephropathy: an Important Opportunity to Re-purpose an Old Drug?Critical role of serum response factor in podocyte epithelial-mesenchymal transition of diabetic nephropathy.Diabetic kidney disease: a role for advanced glycation end-product receptor 1 (AGE-R1)?Classification and Differential Diagnosis of Diabetic Nephropathy.Diabetic nephropathy: Time to withhold development and progression - A review.The VEGF-A inhibitor sFLT-1 improves renal function by reducing endothelial activation and inflammation in a mouse model of type 1 diabetes.Fcγ receptor deficiency attenuates diabetic nephropathy.Insulin signaling to the glomerular podocyte is critical for normal kidney function.Pathology and Prognosis of Type 2 Diabetes Mellitus with Renal Involvement.Validation of the Maryland Aggregate Pathology Index (MAPI), a pre-implantation scoring system that predicts graft outcome.Triptolide Suppresses Glomerular Mesangial Cell Proliferation in Diabetic Nephropathy Is Associated with Inhibition of PDK1/Akt/mTOR Pathway.Lithium reduces blood glucose levels, but aggravates albuminuria in BTBR-ob/ob mice.The mechanical challenges and cytoskeletal impairments in focal segmental glomerulosclerosis.Disparate phospho-Smad2 levels in advanced type 2 diabetes patients with diabetic nephropathy and early experimental db/db mouse model.Cannabinoid-1 receptor deletion in podocytes mitigates both glomerular and tubular dysfunction in a mouse model of diabetic nephropathy.Serum response factor induces endothelial-mesenchymal transition in glomerular endothelial cells to aggravate proteinuria in diabetic nephropathy.
P2860
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P2860
Distinguishing diabetic nephropathy from other causes of glomerulosclerosis: an update.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Distinguishing diabetic nephropathy from other causes of glomerulosclerosis: an update.
@ast
Distinguishing diabetic nephropathy from other causes of glomerulosclerosis: an update.
@en
type
label
Distinguishing diabetic nephropathy from other causes of glomerulosclerosis: an update.
@ast
Distinguishing diabetic nephropathy from other causes of glomerulosclerosis: an update.
@en
prefLabel
Distinguishing diabetic nephropathy from other causes of glomerulosclerosis: an update.
@ast
Distinguishing diabetic nephropathy from other causes of glomerulosclerosis: an update.
@en
P2860
P356
P1476
Distinguishing diabetic nephropathy from other causes of glomerulosclerosis: an update.
@en
P2093
A M Herzenberg
K O Alsaad
P2860
P356
10.1136/JCP.2005.035592
P407
P577
2007-01-01T00:00:00Z