Defects in ex vivo and in vivo growth and sensitivity to osmotic stress of group A Streptococcus caused by interruption of response regulator gene vicR.
about
Functional analysis of the group A streptococcal luxS/AI-2 system in metabolism, adaptation to stress and interaction with host cellsStress Physiology of Lactic Acid BacteriaATP forms a stable complex with the essential histidine kinase WalK (YycG) domainComparative genomic characterization of Francisella tularensis strains belonging to low and high virulence subspeciesBenfang Lei's research on heme acquisition in Gram-positive pathogens and bacterial pathogenesisYvqE and CovRS of Group A Streptococcus Play a Pivotal Role in Viability and Phenotypic Adaptations to Multiple Environmental StressesStructure and mechanism of the essential two-component signal-transduction system WalKR in Staphylococcus aureus.Identification of Lactobacillus sakei genes induced during meat fermentation and their role in survival and growth.Extensive adaptive changes occur in the transcriptome of Streptococcus agalactiae (group B streptococcus) in response to incubation with human blood.Challenges of antibacterial discoveryKinetic characterization of the WalRKSpn (VicRK) two-component system of Streptococcus pneumoniae: dependence of WalKSpn (VicK) phosphatase activity on its PAS domain.Thiazolidione derivatives targeting the histidine kinase YycG are effective against both planktonic and biofilm-associated Staphylococcus epidermidis.A novel STK1-targeted small-molecule as an "antibiotic resistance breaker" against multidrug-resistant Staphylococcus aureus.IgG Endopeptidase SeMac does not Inhibit Opsonophagocytosis of Streptococcus equi Subspecies equi by Horse Polymorphonuclear Leukocytes.Essential Genes in the Core Genome of the Human Pathogen Streptococcus pyogenes.Localization and cellular amounts of the WalRKJ (VicRKX) two-component regulatory system proteins in serotype 2 Streptococcus pneumoniae.Streptococcus pyogenes Ser/Thr kinase-regulated cell wall hydrolase is a cell division plane-recognizing and chain-forming virulence factor.Two Spx regulators modulate stress tolerance and virulence in Streptococcus suis serotype 2Differential regulation of iron- and manganese-specific MtsABC and heme-specific HtsABC transporters by the metalloregulator MtsR of group A Streptococcus.Contribution of eukaryotic-type serine/threonine kinase to stress response and virulence of Streptococcus suis.Serine/threonine protein kinase Stk is required for virulence, stress response, and penicillin tolerance in Streptococcus pyogenes.Serotype M3 and M28 Group A Streptococci Have Distinct Capacities to Evade Neutrophil and TNF-α Responses and to Invade Soft Tissues.A Neutralizing Monoclonal IgG1 Antibody of Platelet-Activating Factor Acetylhydrolase SsE Protects Mice against Lethal Subcutaneous Group A Streptococcus InfectionActive and passive immunizations with the streptococcal esterase Sse protect mice against subcutaneous infection with group A streptococciThe Mga Regulon but Not Deoxyribonuclease Sda1 of Invasive M1T1 Group A Streptococcus Contributes to In Vivo Selection of CovRS Mutations and Resistance to Innate Immune Killing Mechanisms.Regulation of bacterial virulence gene expression by cell envelope stress responses.Regulation of inhibition of neutrophil infiltration by the two-component regulatory system CovRS in subcutaneous murine infection with group A streptococcusThe Two-Component Regulatory System VicRK is Important to Virulence of Streptococcus equi Subspecies equi.Characterization of streptococcal platelet-activating factor acetylhydrolase variants that are involved in innate immune evasion.Post-translational modifications are enriched within protein functional groups important to bacterial adaptation within a deep-sea hydrothermal vent environmentStreptococcus adherence and colonization.The secreted esterase of group a streptococcus is important for invasive skin infection and dissemination in miceThe sagA/pel locus does not regulate the expression of the M protein of the M1T1 lineage of group A Streptococcus.Neutrophils select hypervirulent CovRS mutants of M1T1 group A Streptococcus during subcutaneous infection of miceNatural disruption of two regulatory networks in serotype M3 group A Streptococcus isolates contributes to the virulence factor profile of this hypervirulent serotype.Insights into Streptococcus pyogenes pathogenesis from transcriptome studies.The Streptococcus pyogenes orphan protein tyrosine phosphatase, SP-PTP, possesses dual specificity and essential virulence regulatory functions.Understanding the Streptococcus mutans Cid/Lrg System through CidB Function.Involvement of WalK (VicK) phosphatase activity in setting WalR (VicR) response regulator phosphorylation level and limiting cross-talk in Streptococcus pneumoniae D39 cells.Regulation of bacteriocin production and cell death by the VicRK signaling system in Streptococcus mutans.
P2860
Q21263070-1A70D7C3-1F12-4B21-AFF3-0D5D66A5F8BAQ26741272-C86D0B30-A1E8-42C9-A40F-F0AF03512BCFQ27670593-0A604D4D-7078-43B1-ACC0-208A696B18ECQ28475535-358ADF48-12B5-458A-AFFD-3D371FFC1D25Q28740655-D636B7BF-A541-404A-A647-BD01BAA4B742Q29248567-F7D9418D-297C-44C1-A52A-ADAB271E4058Q30387565-E5CF69E7-6ECB-4E4B-B411-D3D8819CBF68Q33274423-149ED853-08A3-4D62-9D5D-947486E4E718Q33366465-80A8382E-F1EB-4913-A531-DF5A33D66B2BQ33793074-C2B0AC1A-7658-44AE-83D5-6DEFA748DC89Q33825947-B696041E-F4B5-431A-A9FF-05CF26656E84Q33828707-B7056A64-FC52-4E20-A1D0-83FDB4B5E538Q33894853-99E67B32-A06F-4C81-9807-0B598427E7F8Q33912968-D47D16CA-A33F-4C0D-965D-82232B75311DQ34044076-DE865B2C-9D7B-4165-A33C-CB0276A00216Q34119216-AA430656-CBC2-466E-96F9-13A231B53ADFQ34155390-304E08EF-EF10-4223-8557-374CF0E8361BQ34270233-C105AA14-0142-4F98-9889-B98C3345316DQ35073751-E5051515-B8EB-49E8-B2F9-15F4D2E3FF7DQ35123135-EF82C9C0-F88C-4B4F-9D9A-B8EF29CCBD3FQ35273191-2DE92D54-DC80-45E8-A5EC-701BEC56F5AEQ35654539-11B61A5B-FE4D-46F5-AAC4-B00DCA4777C9Q35745520-2B1531C9-98BD-4BE4-B730-8953FEFFB3B0Q35913574-8715D93F-79FB-424A-BB4B-80593A1D28CCQ36137999-152668EA-EAB1-4FFB-9354-BFB9D15ECA35Q36147683-DF39D157-0CA4-41BB-ACFD-D64ED42E4B00Q36646612-7CE593C3-7D7E-4247-9431-8DD3341891A6Q36993866-01F1F4D0-F394-4723-9AA0-547ED6C06D67Q37123729-E87643A1-323F-488C-AF88-A8A1EDE5128BQ37233542-0C6C88B1-A9DA-4B9A-A671-495C23681E20Q37333704-7E874EF7-5B1D-4A9F-B2CB-BA836E00B75DQ37451078-3C054641-5962-4DC3-9596-0A1F3DA1E98BQ37586990-302F034A-7067-4542-B241-A316D81D70EAQ37713261-F74F3164-707B-42C5-A618-5C1126A277CBQ37713316-5CA4C229-5D8D-4A40-9A03-2D0DC26B4697Q37816404-02425E50-01A5-4AAC-B74C-6C7D4C67DF53Q38879201-E22393AD-58B1-4CB6-B35B-58E25D687ADDQ39493799-BFDDCF70-146B-40B6-8B3D-CFF344642C5AQ40204226-B07077D2-2559-4041-9A47-D0DF744605A1Q42138973-68276EDB-8D4F-4C5B-BB69-2DC4F6722C95
P2860
Defects in ex vivo and in vivo growth and sensitivity to osmotic stress of group A Streptococcus caused by interruption of response regulator gene vicR.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Defects in ex vivo and in vivo ...... response regulator gene vicR.
@ast
Defects in ex vivo and in vivo ...... response regulator gene vicR.
@en
type
label
Defects in ex vivo and in vivo ...... response regulator gene vicR.
@ast
Defects in ex vivo and in vivo ...... response regulator gene vicR.
@en
prefLabel
Defects in ex vivo and in vivo ...... response regulator gene vicR.
@ast
Defects in ex vivo and in vivo ...... response regulator gene vicR.
@en
P2093
P2860
P356
P1433
P1476
Defects in ex vivo and in vivo ...... response regulator gene vicR.
@en
P2093
Benfang Lei
Daniel W Siemsen
Jinlian Zhang
Julie L Elser
Mark T Quinn
Mengyao Liu
Michael J McClure
Tracey S Hanks
P2860
P304
P356
10.1099/MIC.0.28706-0
P577
2006-04-01T00:00:00Z