Sensitive and specific assays for C3 nephritic factors clarify mechanisms underlying complement dysregulation - Wikidata - awgldk - TriplyDB

Sensitive and specific assays for C3 nephritic factors clarify mechanisms underlying complement dysregulation

Sensitive and specific assays for C3 nephritic factors clarify mechanisms underlying complement dysregulation

http://www.wikidata.org/entity/Q36718519

about

Reclassification of membranoproliferative glomerulonephritis: Identification of a new GN: C3GN Complement System Part II: Role in Immunity C5 inhibition prevents renal failure in a mouse model of lethal C3 glomerulopathy.A humanized antibody that regulates the alternative pathway convertase: potential for therapy of renal disease associated with nephritic factors Functional Characterization of Autoantibodies against Complement Component C3 in Patients with Lupus Nephritis.Autoantibody stabilization of the classical pathway C3 convertase leading to C3 deficiency and Neisserial sepsis: C4 nephritic factor revisited.Insights into the Effects of Complement Factor H on the Assembly and Decay of the Alternative Pathway C3 Proconvertase and C3 Convertase.Testing the Activity of Complement Convertases in Serum/Plasma for Diagnosis of C4NeF-Mediated C3 Glomerulonephritis.C3 glomerulopathy: consensus report.Complement diagnostics: concepts, indications, and practical guidelines.Making sense of the spectrum of glomerular disease associated with complement dysregulation.Recent advances in the understanding of immune-mediated nephrotic syndrome: diagnostic and prognostic implications.Endothelial cells: source, barrier, and target of defensive mediators.Complement regulation and kidney diseases: recent knowledge of the double-edged roles of complement activation in nephrology.Recovery of renal function succeeding stem cell transplant: a case of C3 Glomerulonephiritis secondary to monoclonal gammopathy Serum properdin consumption as a biomarker of C5 convertase dysregulation in C3 glomerulopathy.Differences in clinical findings, pathology, and outcomes between C3 glomerulonephritis and membranoproliferative glomerulonephritis.Gain of function mutant of complement factor B K323E mimics pathogenic C3NeF autoantibodies in convertase assays.Cluster Analysis Identifies Distinct Pathogenetic Patterns in C3 Glomerulopathies/Immune Complex-Mediated Membranoproliferative GN.Overactivity of Alternative Pathway Convertases in Patients With Complement-Mediated Renal Diseases.C3 Glomerulopathy.A novel method for direct measurement of complement convertases activity in human serum.Interpretation of Serological Complement Biomarkers in Disease

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Sensitive and specific assays for C3 nephritic factors clarify mechanisms underlying complement dysregulation

http://www.wikidata.org/entity/Q36718519

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

Sensitive and specific assays ...... lying complement dysregulation

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Sensitive and specific assays ...... lying complement dysregulation

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type

label

Sensitive and specific assays ...... lying complement dysregulation

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Sensitive and specific assays ...... lying complement dysregulation

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Sensitive and specific assays ...... lying complement dysregulation

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Sensitive and specific assays ...... lying complement dysregulation

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Kidney International

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Sensitive and specific assays ...... lying complement dysregulation

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Claire L Harris

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Danielle Paixão-Cavalcante

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Lennart Truedsson

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Lillemor Skattum

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Patricia C Giclas

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Santiago Rodríguez de Córdoba

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Timothy H Goodship

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P2860

Control of the amplification convertase of complement by the plasma protein beta1H

Complement. First of two parts

Translational mini-review series on complement factor H: renal diseases associated with complement factor H: novel insights from humans and animals.

Autoantibodies in haemolytic uraemic syndrome (HUS).

Lessons from functional and structural analyses of disease-associated genetic variants in the complement alternative pathway.

Clinical features and metabolic and autoimmune derangements in acquired partial lipodystrophy: report of 35 cases and review of the literature.

Common polymorphisms in C3, factor B, and factor H collaborate to determine systemic complement activity and disease risk

Complement components and their autoantibodies.

Causes of alternative pathway dysregulation in dense deposit disease.

Properdin: binding to C3b and stabilization of the C3b-dependent C3 convertase

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1084-1092

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scholarly article

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10.1038/KI.2012.250

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10

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82

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M López-Trascasa

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2012-08-01T00:00:00Z

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230598998

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22854646

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3608896

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