Persistent neuroinflammatory effects of serial exposure to stress and methamphetamine on the blood-brain barrier.
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Methamphetamine effects on blood-brain barrier structure and functionImpact of methamphetamine on infection and immunityNeuroimmune basis of methamphetamine toxicityRecent advances in methamphetamine neurotoxicity mechanisms and its molecular pathophysiologyAmphetamine- and methamphetamine-induced hyperthermia: Implications of the effects produced in brain vasculature and peripheral organs to forebrain neurotoxicityBroken or maladaptive? Altered trajectories in neuroinflammation and behavior after early life adversityDisruption of the Glutamate-Glutamine Cycle Involving Astrocytes in an Animal Model of Depression for Males and Females3,4-Methylenedioxypyrovalerone prevents while methylone enhances methamphetamine-induced damage to dopamine nerve endings: β-ketoamphetamine modulation of neurotoxicity by the dopamine transporter.IFN-γ differentially modulates memory-related processes under basal and chronic stressor conditions.Commentary: Methamphetamine mediates immune dysregulation in a murine model of chronic viral infection.Restraint Stress-Induced Morphological Changes at the Blood-Brain Barrier in Adult Rats.Cyclooxygenase activity contributes to the monoaminergic damage caused by serial exposure to stress and methamphetamineVoluntary exercise protects against methamphetamine-induced oxidative stress in brain microvasculature and disruption of the blood-brain barrier.Stress does not increase blood-brain barrier permeability in mice.Peripheral ammonia and blood brain barrier structure and function after methamphetamine.Development of in vivo drug-induced neurotoxicity models.Acetyl-L-Carnitine Prevents Methamphetamine-Induced Structural Damage on Endothelial Cells via ILK-Related MMP-9 Activity.Role of microglia in methamphetamine-induced neurotoxicity.The neurobiological mechanisms of physical exercise in methamphetamine addiction.The Effects of Histone Deacetylase Inhibition on the Levels of Cerebral Cytokines in an Animal Model of Mania Induced by Dextroamphetamine.Methamphetamine-Induced Brain Injury and Alcohol Drinking.Ulinastatin decreases permeability of blood--brain barrier by inhibiting expression of MMP-9 and t-PA in postoperative aged rats.Lithium modulates the production of peripheral and cerebral cytokines in an animal model of mania induced by dextroamphetamine.
P2860
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P2860
Persistent neuroinflammatory effects of serial exposure to stress and methamphetamine on the blood-brain barrier.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
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2012年论文
@zh-cn
name
Persistent neuroinflammatory e ...... ne on the blood-brain barrier.
@ast
Persistent neuroinflammatory e ...... ne on the blood-brain barrier.
@en
type
label
Persistent neuroinflammatory e ...... ne on the blood-brain barrier.
@ast
Persistent neuroinflammatory e ...... ne on the blood-brain barrier.
@en
prefLabel
Persistent neuroinflammatory e ...... ne on the blood-brain barrier.
@ast
Persistent neuroinflammatory e ...... ne on the blood-brain barrier.
@en
P2860
P1476
Persistent neuroinflammatory e ...... ne on the blood-brain barrier.
@en
P2093
Bryan K Yamamoto
Nicole A Northrop
P2860
P2888
P304
P356
10.1007/S11481-012-9391-Y
P577
2012-07-26T00:00:00Z