Epitope specificity determines pathogenicity and detectability in ANCA-associated vasculitis. - Wikidata - awgldk - TriplyDB

Epitope specificity determines pathogenicity and detectability in ANCA-associated vasculitis.

Epitope specificity determines pathogenicity and detectability in ANCA-associated vasculitis.

http://www.wikidata.org/entity/Q36733435

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The Prevalence and Management of Pauci-Immune Glomerulonephritis and Vasculitis in Western Countries Overview of the Pathogenesis of ANCA-Associated Vasculitis Neutrophil Extracellular Traps in ANCA-Associated Vasculitis Neutrophil-Mediated Regulation of Innate and Adaptive Immunity: The Role of Myeloperoxidase Anti-neutrophil cytoplasmic autoantibody pathogenicity revisited: pathogenic versus non-pathogenic anti-neutrophil cytoplasmic autoantibody Updates in ANCA-associated vasculitis Mechanisms of Autoantibody-Induced Pathology.ANCA as a predictor of relapse: useful in patients with renal involvement but not in patients with nonrenal disease Linking classification and therapeutic management of vasculitides.Detection of Anti-Pentraxin-3 Autoantibodies in ANCA-Associated Vasculitis.Systemic Lupus Erythematosus and Antineutrophil Cytoplasmic Antibody-Associated Vasculitis Overlap Syndrome in Patients With Biopsy-Proven Glomerulonephritis.Clinical and Immunologic Characteristics of Patients With ANCA-Associated Glomerulonephritis Combined With Membranous Nephropathy: A Retrospective Cohort Study in a Single Chinese Center Myeloperoxidase Peptide-Based Nasal Tolerance in Experimental ANCA-Associated GN.Autoantibodies against Linear Epitopes of Myeloperoxidase in Anti-Glomerular Basement Membrane Disease.Diagnosing ANCA-associated vasculitis in ANCA positive patients: A retrospective analysis on the role of clinical symptoms and the ANCA titre.Myeloperoxidase anti-neutrophil cytoplasmic antibody affinity is associated with the formation of neutrophil extracellular traps in the kidney and vasculitis activity in myeloperoxidase anti-neutrophil cytoplasmic antibody-associated microscopic pol Autoantibodies to hLAMP-2 in ANCA-negative pauci-immune focal necrotizing GN Clinicopathologic Characteristics and Outcomes of Lupus Nephritis With Antineutrophil Cytoplasmic Antibody: A Retrospective Study.Anti-glomerular basement membrane disease: Case series from a tertiary center in North India.Granuloma in ANCA-associated vasculitides: another reason to distinguish between syndromes?The pathogenic potential of autoreactive antibodies in rheumatoid arthritis.B cell-mediated pathogenesis of ANCA-mediated vasculitis.Key advances in the clinical approach to ANCA-associated vasculitis.Pathogenesis of antineutrophil cytoplasmic autoantibody-mediated disease.Pathogenesis of anti-neutrophil cytoplasmic antibody-associated vasculitis: challenges and solutions 2014.The kidneys and ANCA-associated vasculitis: from pathogenesis to diagnosis.A historical study of American patients with anti-neutrophil cytoplasmic antibody negative pauci-immune glomerulonephritis.Multicolor flow cytometric analysis of TLR2 and TLR9 expression and function in NK cells from patients with ANCA-associated vasculitis.Defining Glomerular Disease in Mechanistic Terms: Implementing an Integrative Biology Approach in Nephrology.Review: Neutrophils as Invigorated Targets in Rheumatic Diseases Anti-pentraxin antibodies in autoimmune systemic diseases: Focus on anti-pentraxin-3 autoantibodies.Position paper: Revised 2017 international consensus on testing of ANCAs in granulomatosis with polyangiitis and microscopic polyangiitis.Myeloperoxidase-antineutrophil cytoplasmic antibody causes different renal diseases by immune-complex formation and pauci-immune mechanism: A case report.Deglycosylation influences the oxidation activity and antigenicity of myeloperoxidase.Histone modification signature at myeloperoxidase and proteinase 3 in patients with anti-neutrophil cytoplasmic autoantibody-associated vasculitis.Current and novel biomarkers in anti-neutrophil cytoplasm-associated vasculitis.Cathelicidin antimicrobial peptide as a serologic marker and potential pathogenic factor in antineutrophil cytoplasmic antibody-associated vasculitis.Anti-ADAMTS13 IgG autoantibodies present in healthy individuals share linear epitopes with those in patients with thrombotic thrombocytopenic purpura.Coming of Age--CC Chemokine Ligand 18 in ANCA-Associated Vasculitis.Identification of a major epitope recognized by PLA2R autoantibodies in primary membranous nephropathy.

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P2860

Epitope specificity determines pathogenicity and detectability in ANCA-associated vasculitis.

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Epitope specificity determines ...... in ANCA-associated vasculitis.

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A Richard Kitching

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Aleeza J Roth

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Caroline E Poulton

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Coen A Stegeman

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Elisabeth A Berg

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Gloria A Preston

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J Charles Jennette

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Jacob J Hess

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John Niles

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Joshua D Ooi

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P2860

Antineutrophil cytoplasmic autoantibodies specific for myeloperoxidase cause glomerulonephritis and vasculitis in mice

Chemical evidence that proteolytic cleavage causes the heterogeneity present in human ceruloplasmin preparations

Nomenclature of systemic vasculitides. Proposal of an international consensus conference

MALDI/MS-based epitope mapping of antigens bound to immobilized antibodies.

Occurrence of autoantibodies to human leucocyte elastase in Wegener's granulomatosis and other inflammatory disorders.

Anti-neutrophil cytoplasmic autoantibodies induce neutrophils to degranulate and produce oxygen radicals in vitro

Addendum to the International Consensus Statement on testing and reporting of antineutrophil cytoplasmic antibodies. Quality control guidelines, comments, and recommendations for testing in other autoimmune diseases.

Asymptomatic autoantibodies associate with future anti-glomerular basement membrane disease

The immunodominant myeloperoxidase T-cell epitope induces local cell-mediated injury in antimyeloperoxidase glomerulonephritis.

IL-23, not IL-12, directs autoimmunity to the Goodpasture antigen.

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10.1172/JCI65292

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4

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123

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3613913

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