Vardenafil protects isolated rat hearts at reperfusion dependent on GC and PKG - Wikidata - awgldk - TriplyDB

Vardenafil protects isolated rat hearts at reperfusion dependent on GC and PKG

Vardenafil protects isolated rat hearts at reperfusion dependent on GC and PKG

http://www.wikidata.org/entity/Q36735813

about

Riociguat reduces infarct size and post-infarct heart failure in mouse hearts: insights from MRI/PET imaging Evidence for an intracellular localization of the adenosine A2B receptor in rat cardiomyocytes.Phospholemman Ser69 phosphorylation contributes to sildenafil-induced cardioprotection against reperfusion injury.Cardioprotective PKG-independent NO signaling at reperfusion Hydrogen sulfide and PKG in ischemia-reperfusion injury: sources, signaling, accelerators and brakes.Infarct limitation by a protein kinase G activator at reperfusion in rabbit hearts is dependent on sensitizing the heart to A2b agonists by protein kinase C Phosphodiesterase type-5 inhibitor use in type 2 diabetes is associated with a reduction in all-cause mortality.Phosphodiesterase 5 inhibitors: are they cardioprotective?Protein kinase G-dependent cardioprotective mechanism of phosphodiesterase-5 inhibition involves phosphorylation of ERK and GSK3beta.Anti-necrotic and cardioprotective effects of a cytosolic renin isoform under ischemia-related conditions.Pharmacological postconditioning by bolus injection of phosphodiesterase-5 inhibitors vardenafil and sildenafil.BAY 58-2667, a nitric oxide-independent guanylyl cyclase activator, pharmacologically post-conditions rabbit and rat hearts.

P2860

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P2860

Vardenafil protects isolated rat hearts at reperfusion dependent on GC and PKG

http://www.wikidata.org/entity/Q36735813

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Vardenafil protects isolated rat hearts at reperfusion dependent on GC and PKG

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Vardenafil protects isolated rat hearts at reperfusion dependent on GC and PKG

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Vardenafil protects isolated rat hearts at reperfusion dependent on GC and PKG

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Vardenafil protects isolated rat hearts at reperfusion dependent on GC and PKG

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prefLabel

Vardenafil protects isolated rat hearts at reperfusion dependent on GC and PKG

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Vardenafil protects isolated rat hearts at reperfusion dependent on GC and PKG

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British Journal of Pharmacology

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Vardenafil protects isolated rat hearts at reperfusion dependent on GC and PKG

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P2093

H K Kroemer

http://www.w3.org/2001/XMLSchema#string

M Frenzel

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O Maas

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S B Felix

http://www.w3.org/2001/XMLSchema#string

T Rütz

http://www.w3.org/2001/XMLSchema#string

U Donat

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P2860

Potency, selectivity, and consequences of nonselectivity of PDE inhibition

Isoflurane inhibits cardiac myocyte apoptosis during oxidative and inflammatory stress by activating Akt and enhancing Bcl-2 expression

HL-1 cells: a cardiac muscle cell line that contracts and retains phenotypic characteristics of the adult cardiomyocyte

Vardenafil preclinical trial data: potency, pharmacodynamics, pharmacokinetics, and adverse events.

Highly specific, membrane-permeant peptide blockers of cGMP-dependent protein kinase Ialpha inhibit NO-induced cerebral dilation.

Sildenafil and vardenafil but not nitroglycerin limit myocardial infarction through opening of mitochondrial K(ATP) channels when administered at reperfusion following ischemia in rabbits

Cyclic guanosine monophosphate compartmentation in rat cardiac myocytes

Cyclic GMP and protein kinase-G in myocardial ischaemia-reperfusion: opportunities and obstacles for survival signaling.

Species- and tissue-dependent effects of NO and cyclic GMP on cardiac ion channels.

Natriuretic peptides and nitric oxide stimulate cGMP synthesis in different cellular compartments.

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25-31

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10.1038/BJP.2008.71

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1

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154

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5519094

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18332860

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2438966

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