Cardioprotective PKG-independent NO signaling at reperfusion - Wikidata - awgldk - TriplyDB

Cardioprotective PKG-independent NO signaling at reperfusion

Cardioprotective PKG-independent NO signaling at reperfusion

http://www.wikidata.org/entity/Q34426209

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Mitochondria selective S-nitrosation by mitochondria-targeted S-nitrosothiol protects against post-infarct heart failure in mouse hearts.Cardioprotection by S-nitrosation of a cysteine switch on mitochondrial complex I.Adenosine A1 receptor activation increases myocardial protein S-nitrosothiols and elicits protection from ischemia-reperfusion injury in male and female hearts Probing biased/partial agonism at the G protein-coupled A(2B) adenosine receptor.A2B adenosine receptors inhibit superoxide production from mitochondrial complex I in rabbit cardiomyocytes via a mechanism sensitive to Pertussis toxin Overexpression of cGMP-dependent protein kinase I (PKG-I) attenuates ischemia-reperfusion-induced kidney injury.The alpha1 isoform of soluble guanylate cyclase regulates cardiac contractility but is not required for ischemic preconditioning Hydrogen sulfide and PKG in ischemia-reperfusion injury: sources, signaling, accelerators and brakes.Essential role of nitric oxide in acute ischemic preconditioning: S-nitros(yl)ation versus sGC/cGMP/PKG signaling?Additive cardioprotection by pharmacological postconditioning with hydrogen sulfide and nitric oxide donors in mouse heart: S-sulfhydration vs. S-nitrosylation.Soluble guanylate cyclase activation during ischemic injury in mice protects against postischemic inflammation at the mitochondrial level.Preconditioning with soluble guanylate cyclase activation prevents postischemic inflammation and reduces nitrate tolerance in heme oxygenase-1 knockout mice.Postconditioning leads to an increase in protein S-nitrosylation.Novel approaches and opportunities for cardioprotective signaling through 3',5'-cyclic guanosine monophosphate manipulation.The cGMP/PKG pathway as a common mediator of cardioprotection: translatability and mechanism.NO Better Way to Protect the Heart during Ischemia-Reperfusion: To be in the Right Place at the Right Time.Treatment of Myocardial Ischemia/Reperfusion Injury by Ischemic and Pharmacological Postconditioning.ATP-induced cardioprotection against myocardial ischemia/reperfusion injury is mediated through the RISK pathway.NO-independent stimulation or activation of soluble guanylyl cyclase during early reperfusion limits infarct size.Comparison of soluble guanylate cyclase stimulators and activators in models of cardiovascular disease associated with oxidative stress.Combined hypoxia and sodium nitrite pretreatment for cardiomyocyte protection in vitro.

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Cardioprotective PKG-independent NO signaling at reperfusion

http://www.wikidata.org/entity/Q34426209

description

2010 nî lūn-bûn

@nan

2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Cardioprotective PKG-independent NO signaling at reperfusion

@ast

Cardioprotective PKG-independent NO signaling at reperfusion

@en

Cardioprotective PKG-independent NO signaling at reperfusion

@nl

type

label

Cardioprotective PKG-independent NO signaling at reperfusion

@ast

Cardioprotective PKG-independent NO signaling at reperfusion

@en

Cardioprotective PKG-independent NO signaling at reperfusion

@nl

prefLabel

Cardioprotective PKG-independent NO signaling at reperfusion

@ast

Cardioprotective PKG-independent NO signaling at reperfusion

@en

Cardioprotective PKG-independent NO signaling at reperfusion

@nl

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AJPHEART.00527.2010

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American Journal of Physiology - Heart and Circulatory Physiology

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Cardioprotective PKG-independent NO signaling at reperfusion

@en

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James M Downey

http://www.w3.org/2001/XMLSchema#string

Michael V Cohen

http://www.w3.org/2001/XMLSchema#string

Nataliya V Solenkova

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Xi-Ming Yang

http://www.w3.org/2001/XMLSchema#string

Yanping Liu

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P2860

Intramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPT

Akt-mediated activation of HIF-1 in pulmonary vascular endothelial cells by S-nitrosoglutathione

Upregulation of endothelial nitric oxide synthase by HMG CoA reductase inhibitors.

Postconditioning protects rabbit hearts through a protein kinase C-adenosine A2b receptor cascade.

Sildenafil and vardenafil but not nitroglycerin limit myocardial infarction through opening of mitochondrial K(ATP) channels when administered at reperfusion following ischemia in rabbits

GSK3beta inhibition and K(ATP) channel opening mediate acute opioid-induced cardioprotection at reperfusion.

Nitrite augments tolerance to ischemia/reperfusion injury via the modulation of mitochondrial electron transfer.

Nitric oxide is a preconditioning mimetic and cardioprotectant and is the basis of many available infarct-sparing strategies.

Vardenafil protects isolated rat hearts at reperfusion dependent on GC and PKG

Infarct limitation by a protein kinase G activator at reperfusion in rabbit hearts is dependent on sensitizing the heart to A2b agonists by protein kinase C

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H2028-36

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scholarly article

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10.1152/AJPHEART.00527.2010

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6

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2010-09-17T00:00:00Z

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20852051

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3006290

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