Hyperactive somatostatin interneurons contribute to excitotoxicity in neurodegenerative disorders.
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Somatostatin-Expressing Inhibitory Interneurons in Cortical CircuitsCortical synaptic and dendritic spine abnormalities in a presymptomatic TDP-43 model of amyotrophic lateral sclerosis.Early Cognitive/Social Deficits and Late Motor Phenotype in Conditional Wild-Type TDP-43 Transgenic Mice.Tdp-43 cryptic exons are highly variable between cell types.Calretinin and Neuropeptide Y interneurons are differentially altered in the motor cortex of the SOD1G93A mouse model of ALS.Impairments in Motor Neurons, Interneurons and Astrocytes Contribute to Hyperexcitability in ALS: Underlying Mechanisms and Paths to Therapy.Transcranial Magnetic Stimulation for the Assessment of Neurodegenerative Disease.Prefrontal Cortex GABAergic Deficits and Circuit Dysfunction in the Pathophysiology and Treatment of Chronic Stress and Depression.Synapse Dysfunction of Layer V Pyramidal Neurons Precedes Neurodegeneration in a Mouse Model of TDP-43 Proteinopathies.Distinct Activity Profiles of Somatostatin-Expressing Interneurons in the Neocortex.Single-Cell Profiling of an In Vitro Model of Human Interneuron Development Reveals Temporal Dynamics of Cell Type Production and Maturation.TDP-43 accelerates age-dependent degeneration of interneurons.Protein kinase Mζ in medial prefrontal cortex mediates depressive-like behavior and antidepressant response.Artificial intelligence in neurodegenerative disease research: use of IBM Watson to identify additional RNA-binding proteins altered in amyotrophic lateral sclerosis.Current Advances and Limitations in Modeling ALS/FTD in a Dish Using Induced Pluripotent Stem Cells.PV+ Cells Enhance Temporal Population Codes but not Stimulus-Related Timing in Auditory Cortex.Brain functional connectome abnormalities in amyotrophic lateral sclerosis are associated with disability and cortical hyperexcitability.Changes in the Excitability of Neocortical Neurons in a Mouse Model of Amyotrophic Lateral Sclerosis Are Not Specific to Corticospinal Neurons and Are Modulated by Advancing Disease.Cortical hyperexcitability and disease spread in amyotrophic lateral sclerosis.In vivo calcium imaging and Parkinson's disease.Synaptic dysfunction and altered excitability in C9ORF72 ALS/FTD.[Intern(euron)al affairs : The role of specific neocortical interneuron classes in the interaction between acetylcholine and GABAergic anesthetics].Human iPSC-Derived Endothelial Cells and Microengineered Organ-Chip Enhance Neuronal Development.Microglia activation contributes to quinolinic acid-induced neuronal excitotoxicity through TNF-α.Reduced Excitability and Increased Neurite Complexity of Cortical Interneurons in a Familial Mouse Model of Amyotrophic Lateral Sclerosis
P2860
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P2860
Hyperactive somatostatin interneurons contribute to excitotoxicity in neurodegenerative disorders.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Hyperactive somatostatin inter ...... n neurodegenerative disorders.
@ast
Hyperactive somatostatin inter ...... n neurodegenerative disorders.
@en
type
label
Hyperactive somatostatin inter ...... n neurodegenerative disorders.
@ast
Hyperactive somatostatin inter ...... n neurodegenerative disorders.
@en
prefLabel
Hyperactive somatostatin inter ...... n neurodegenerative disorders.
@ast
Hyperactive somatostatin inter ...... n neurodegenerative disorders.
@en
P2093
P2860
P356
P1433
P1476
Hyperactive somatostatin inter ...... in neurodegenerative disorders
@en
P2093
Da-Ting Lin
David Schroeder
Gregory A Cox
Lifeng Zhang
Zhong-Wei Zhang
P2860
P2888
P304
P356
10.1038/NN.4257
P407
P50
P577
2016-02-22T00:00:00Z