Impaired opsonization with C3b and phagocytosis of Streptococcus pneumoniae in sera from subjects with defects in the classical complement pathway - Wikidata - awgldk - TriplyDB

Impaired opsonization with C3b and phagocytosis of Streptococcus pneumoniae in sera from subjects with defects in the classical complement pathway

Impaired opsonization with C3b and phagocytosis of Streptococcus pneumoniae in sera from subjects with defects in the classical complement pathway

http://www.wikidata.org/entity/Q36804267

about

Streptococcus pneumoniae capsular serotype 19F is more resistant to C3 deposition and less sensitive to opsonophagocytosis than serotype 6B PSGL-1 on Leukocytes is a Critical Component of the Host Immune Response against Invasive Pneumococcal Disease The lectin pathway of complement activation is a critical component of the innate immune response to pneumococcal infection Effects of Streptococcus pneumoniae strain background on complement resistance Protective contributions against invasive Streptococcus pneumoniae pneumonia of antibody and Th17-cell responses to nasopharyngeal colonisation C3b/iC3b deposition on Streptococcus pneumoniae is not affected by HIV infection Enhanced in vivo activity of cefditoren in pre-immunized mice against penicillin-resistant S. pneumoniae (serotypes 6B, 19F and 23F) in a sepsis model Recombinant human complement component C2 produced in a human cell line restores the classical complement pathway activity in-vitro: an alternative treatment for C2 deficiency diseases Enhanced susceptibility to acute pneumococcal otitis media in mice deficient in complement C1qa, factor B, and factor B/C2.Nasopharyngeal colonization and invasive disease are enhanced by the cell wall hydrolases LytB and LytC of Streptococcus pneumoniae.The capsular serotype of Streptococcus pneumoniae is more important than the genetic background for resistance to complement.Cefditoren and ceftriaxone enhance complement-mediated immunity in the presence of specific antibodies against antibiotic-resistant pneumococcal strains The Streptococcus pneumoniae capsule inhibits complement activity and neutrophil phagocytosis by multiple mechanisms Importance of bacterial replication and alveolar macrophage-independent clearance mechanisms during early lung infection with Streptococcus pneumoniae.RrgB321, a fusion protein of the three variants of the pneumococcal pilus backbone RrgB, is protective in vivo and elicits opsonic antibodies.Emergence of Amoxicillin-Resistant Variants of Spain9V-ST156 Pneumococci Expressing Serotype 11A Correlates with Their Ability to Evade the Host Immune Response Death is associated with complement C3 depletion in cerebrospinal fluid of patients with pneumococcal meningitis.Generation and Improvement of Effector Function of a Novel Broadly Reactive and Protective Monoclonal Antibody against Pneumococcal Surface Protein A of Streptococcus pneumoniae.Naturally Acquired Human Immunity to Pneumococcus Is Dependent on Antibody to Protein Antigens.Role of the alternative and classical complement activation pathway in complement mediated killing against Streptococcus pneumoniae colony opacity variants during acute pneumococcal otitis media in mice.PspK of Streptococcus pneumoniae increases adherence to epithelial cells and enhances nasopharyngeal colonization Streptococcus pneumoniae capsular serotype invasiveness correlates with the degree of factor H binding and opsonization with C3b/iC3b Proteomic analysis of cerebrospinal fluid in pneumococcal meningitis reveals potential biomarkers associated with survival.Anatomical site-specific contributions of pneumococcal virulence determinants.CovR Regulates Streptococcus mutans Susceptibility To Complement Immunity and Survival in Blood.Immune ageing and susceptibility to Streptococcus pneumoniae.Role of Streptococcus pneumoniae Proteins in Evasion of Complement-Mediated Immunity.The two-component system VicRK regulates functions associated with Streptococcus mutans resistance to complement immunity.Three surface exoglycosidases from Streptococcus pneumoniae, NanA, BgaA, and StrH, promote resistance to opsonophagocytic killing by human neutrophils.Biofilm formation avoids complement immunity and phagocytosis of Streptococcus pneumoniae.Spectrum and management of complement immunodeficiencies (excluding hereditary angioedema) across Europe.Macrolides and β-lactam antibiotics enhance C3b deposition on the surface of multidrug-resistant Streptococcus pneumoniae strains by a LytA autolysin-dependent mechanism.Pleiotropic effects of cell wall amidase LytA on Streptococcus pneumoniae sensitivity to the host immune response The effects of PspC on complement-mediated immunity to Streptococcus pneumoniae vary with strain background and capsular serotype.Streptococcus pneumoniae resistance to complement-mediated immunity is dependent on the capsular serotype Human and pneumococcal cell surface glyceraldehyde-3-phosphate dehydrogenase (GAPDH) proteins are both ligands of human C1q protein.Priming innate immune responses to infection by cyclooxygenase inhibition kills antibiotic-susceptible and -resistant bacteria.The Surface-Exposed Protein SntA Contributes to Complement Evasion in Zoonotic Streptococcus suis.

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P2860

Impaired opsonization with C3b and phagocytosis of Streptococcus pneumoniae in sera from subjects with defects in the classical complement pathway

http://www.wikidata.org/entity/Q36804267

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Impaired opsonization with C3b ...... e classical complement pathway

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Impaired opsonization with C3b ...... e classical complement pathway

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Impaired opsonization with C3b ...... e classical complement pathway

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Impaired opsonization with C3b ...... e classical complement pathway

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Impaired opsonization with C3b ...... e classical complement pathway

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Infection and Immunity

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Impaired opsonization with C3b ...... e classical complement pathway

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P2093

Ashwin Sen

http://www.w3.org/2001/XMLSchema#string

Catherine Hyams

http://www.w3.org/2001/XMLSchema#string

Fiona Goldblatt

http://www.w3.org/2001/XMLSchema#string

Göran Jönsson

http://www.w3.org/2001/XMLSchema#string

Helen E Baxendale

http://www.w3.org/2001/XMLSchema#string

Jeremy S Brown

http://www.w3.org/2001/XMLSchema#string

Lennart Truedsson

http://www.w3.org/2001/XMLSchema#string

Liang-Seah Tay

http://www.w3.org/2001/XMLSchema#string

Marina Botto

http://www.w3.org/2001/XMLSchema#string

P2860

Study of community acquired pneumonia aetiology (SCAPA) in adults admitted to hospital: implications for management guidelines

Are anticapsular antibodies the primary mechanism of protection against invasive pneumococcal disease?

Complement. First of two parts

Complement activation in factor D-deficient mice

Abrogation of the alternative complement pathway by targeted deletion of murine factor B

Targeted gene disruption reveals a role for natural secretory IgM in the maturation of the primary immune response

Serum amyloid P aids complement-mediated immunity to Streptococcus pneumoniae.

Studies of group B streptococcal infection in mice deficient in complement component C3 or C4 demonstrate an essential role for complement in both innate and acquired immunity.

Pneumococcal surface protein A inhibits complement activation by Streptococcus pneumoniae.

Intracellular trafficking and killing of Streptococcus pneumoniae by human alveolar macrophages are influenced by opsonins

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3761-3770

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10.1128/IAI.00291-08

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8

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76

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18541650

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2493217

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