Mitochondrial ROS Produced via Reverse Electron Transport Extend Animal Lifespan - Wikidata - awgldk - TriplyDB

Mitochondrial ROS Produced via Reverse Electron Transport Extend Animal Lifespan

Mitochondrial ROS Produced via Reverse Electron Transport Extend Animal Lifespan

http://www.wikidata.org/entity/Q36809576

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Reactive Oxygen Species Regulate T Cell Immune Response in the Tumor Microenvironment Being right on Q: shaping eukaryotic evolution Mitochondria and oxidative stress in heart aging Electron tomographic analysis reveals ultrastructural features of mitochondrial cristae architecture which reflect energetic state and aging.Regulation of the H+-ATP synthase by IF1: a role in mitohormesis.Arsenite Effects on Mitochondrial Bioenergetics in Human and Mouse Primary Hepatocytes Follow a Nonlinear Dose Response.Improved health-span and lifespan in mtDNA mutator mice treated with the mitochondrially targeted antioxidant SkQ1.The 4E-BP growth pathway regulates the effect of ambient temperature on Drosophila metabolism and lifespan.Evolution of peroxisomes illustrates symbiogenesis.Role of Mitochondrial Reverse Electron Transport in ROS Signaling: Potential Roles in Health and Disease.Subcellular Redox Targeting: Bridging in Vitro and in Vivo Chemical Biology.Mitochondria are the powerhouses of immunity.Reactive Oxygen and Nitrogen Species in the Development of Pulmonary Hypertension.Krebs cycle rewired for macrophage and dendritic cell effector functions.UCP1 deficiency causes brown fat respiratory chain depletion and sensitizes mitochondria to calcium overload-induced dysfunction Aging: Molecular Pathways and Implications on the Cardiovascular System.Honokiol induces superoxide production by targeting mitochondrial respiratory chain complex I in Candida albicans Suppressors of Superoxide-H2O2 Production at Site IQ of Mitochondrial Complex I Protect against Stem Cell Hyperplasia and Ischemia-Reperfusion Injury.The bright side of reactive oxygen species: lifespan extension without cellular demise.The Combination of Physical Exercise with Muscle-Directed Antioxidants to Counteract Sarcopenia: A Biomedical Rationale for Pleiotropic Treatment with Creatine and Coenzyme Q10.The role of mitochondrial ROS in the aging brain.Synthesis and Radioprotective Activity of Mitochondria Targeted Dihydropyridines In Vitro.Reverse electron transfer results in a loss of flavin from mitochondrial complex I: Potential mechanism for brain ischemia reperfusion injury.Reactive oxygen species extend insect life span using components of the insulin-signaling pathway.Mitochondria targeting by environmental stressors: Implications for redox cellular signaling.The Putative Drp1 Inhibitor mdivi-1 Is a Reversible Mitochondrial Complex I Inhibitor that Modulates Reactive Oxygen Species.Succinate Dehydrogenase Supports Metabolic Repurposing of Mitochondria to Drive Inflammatory Macrophages.EXD2 governs germ stem cell homeostasis and lifespan by promoting mitoribosome integrity and translation.New roles for old actors, ROS and PRMT1.Coenzyme Q10 Supplementation in Aging and Disease.Activation of DAF-16/FOXO by reactive oxygen species contributes to longevity in long-lived mitochondrial mutants in Caenorhabditis elegans.Influence of anaerobic and aerobic exercise on age-related pathways in skeletal muscle.Mitochondrial oxidative stress causes insulin resistance without disrupting oxidative phosphorylation.Metabolic Modulation in Macrophage Effector Function.Adaptations in energy metabolism and gene family expansions revealed by comparative transcriptomics of three Chagas disease triatomine vectors.The Good, the Bad, and the Ugly of ROS: New Insights on Aging and Aging-Related Diseases from Eukaryotic and Prokaryotic Model Organisms.Superoxide dismutating molecules rescue the toxic effects of PINK1 and parkin loss.Mitochondrial Complex I activity signals antioxidant response through ERK5.Control of mitochondrial superoxide production by reverse electron transport at complex I.Perturbed Redox Signaling Exacerbates a Mitochondrial Myopathy

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Mitochondrial ROS Produced via Reverse Electron Transport Extend Animal Lifespan

http://www.wikidata.org/entity/Q36809576

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2016年论文

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Mitochondrial ROS Produced via Reverse Electron Transport Extend Animal Lifespan

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Mitochondrial ROS Produced via Reverse Electron Transport Extend Animal Lifespan

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Mitochondrial ROS Produced via Reverse Electron Transport Extend Animal Lifespan

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Mitochondrial ROS Produced via Reverse Electron Transport Extend Animal Lifespan

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Mitochondrial ROS Produced via Reverse Electron Transport Extend Animal Lifespan

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J.CMET.2016.03.009

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Cell Metabolism

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Mitochondrial ROS Produced via Reverse Electron Transport Extend Animal Lifespan

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Angela Logan

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Ashwin Sriram

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Helen M Cooper

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Madis Lõhmus

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Plácido Navas

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P2860

A mitochondrial superoxide signal triggers increased longevity in Caenorhabditis elegans

The hallmarks of aging

Extending life span by increasing oxidative stress

Glucose restriction extends Caenorhabditis elegans life span by inducing mitochondrial respiration and increasing oxidative stress

Activation of AMPK by the putative dietary restriction mimetic metformin is insufficient to extend lifespan in Drosophila

PINK1 loss-of-function mutations affect mitochondrial complex I activity via NdufA10 ubiquinone uncoupling

Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants

Metformin promotes lifespan through mitohormesis via the peroxiredoxin PRDX-2

Neuronal expression of a single-subunit yeast NADH-ubiquinone oxidoreductase (Ndi1) extends Drosophila lifespan.

Expression of the yeast NADH dehydrogenase Ndi1 in Drosophila confers increased lifespan independently of dietary restriction.

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4

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Michael P. Murphy

Daniel José Moreno Fernández Ayala

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Filippo Scialò

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