The Angiotensin Converting Enzyme Inhibitor Lisinopril Improves Muscle Histopathology but not Contractile Function in a Mouse Model of Duchenne Muscular Dystrophy.
about
Mineralocorticoid receptors are present in skeletal muscle and represent a potential therapeutic target.Similar efficacy from specific and non-specific mineralocorticoid receptor antagonist treatment of muscular dystrophy mice.Gene expression effects of glucocorticoid and mineralocorticoid receptor agonists and antagonists on normal human skeletal muscle.Renin-angiotensin-aldosterone system inhibitors improve membrane stability and change gene-expression profiles in dystrophic skeletal muscles.Myeloid cells are capable of synthesizing aldosterone to exacerbate damage in muscular dystrophy.
P2860
The Angiotensin Converting Enzyme Inhibitor Lisinopril Improves Muscle Histopathology but not Contractile Function in a Mouse Model of Duchenne Muscular Dystrophy.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
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2015年论文
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name
The Angiotensin Converting Enz ...... f Duchenne Muscular Dystrophy.
@ast
The Angiotensin Converting Enz ...... f Duchenne Muscular Dystrophy.
@en
type
label
The Angiotensin Converting Enz ...... f Duchenne Muscular Dystrophy.
@ast
The Angiotensin Converting Enz ...... f Duchenne Muscular Dystrophy.
@en
prefLabel
The Angiotensin Converting Enz ...... f Duchenne Muscular Dystrophy.
@ast
The Angiotensin Converting Enz ...... f Duchenne Muscular Dystrophy.
@en
P2093
P2860
P356
P1476
The Angiotensin Converting Enz ...... f Duchenne Muscular Dystrophy.
@en
P2093
Andrew J Wodarcyk
Eric J Schultz
Jeovanna Lowe
Jessica A Chadwick
Jill A Rafael-Fortney
Kyle T Floyd
Neha Rastogi
Paul M L Janssen
Sarah A Swager
Subha V Raman
P2860
P304
P356
10.3233/JND-150099
P577
2015-09-02T00:00:00Z