SCO2 Mediates Oxidative Stress-Induced Glycolysis to Oxidative Phosphorylation Switch in Hematopoietic Stem Cells

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The sweet trap in tumors: aerobic glycolysis and potential targets for therapy Metabolic regulation of hematopoietic and leukemic stem/progenitor cells under homeostatic and stress conditions.Cell-Cycle-Specific Function of p53 in Fanconi Anemia Hematopoietic Stem and Progenitor Cell Proliferation.

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SCO2 Mediates Oxidative Stress-Induced Glycolysis to Oxidative Phosphorylation Switch in Hematopoietic Stem Cells

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Andrew F Wilson

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Qishen Pang

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Surya Amarachintha

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Wei Du

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P2860

Fatal infantile cardioencephalomyopathy with COX deficiency and mutations in SCO2, a COX assembly gene

Molecular pathogenesis and clinical management of Fanconi anemia

Regulation of glucose metabolism by p53: emerging new roles for the tumor suppressor

Surfing the p53 network

TIGAR, a p53-inducible regulator of glycolysis and apoptosis

Targeted disruption of exons 1 to 6 of the Fanconi Anemia group A gene leads to growth retardation, strain-specific microphthalmia, meiotic defects and primordial germ cell hypoplasia

p53 regulates mitochondrial respiration

Inactivation of Fac in mice produces inducible chromosomal instability and reduced fertility reminiscent of Fanconi anaemia

Fanconi anaemia and the repair of Watson and Crick DNA crosslinks

Glycolytic enzymes can modulate cellular life span

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scholarly article

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10.1002/STEM.2260

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SCO2 Mediates Oxidative Stress-Induced Glycolysis to Oxidative Phosphorylation Switch in Hematopoietic Stem Cells

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P2860

SCO2 Mediates Oxidative Stress-Induced Glycolysis to Oxidative Phosphorylation Switch in Hematopoietic Stem Cells

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P2860

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P577

P698

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P1476

P2093

P2860

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P356

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P478

P577

P698

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