Characterization of nerve growth factor-dependent herpes simplex virus latency in neurons in vitro.
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A comparison of herpes simplex virus type 1 and varicella-zoster virus latency and reactivationThe molecular basis of herpes simplex virus latencyA cultured affair: HSV latency and reactivation in neuronsOcular herpes simplex virus: how are latency, reactivation, recurrent disease and therapy interrelated?Nuclear localization of the C1 factor (host cell factor) in sensory neurons correlates with reactivation of herpes simplex virus from latencyEntry of herpes simplex virus type 1 into primary sensory neurons in vitro is mediated by Nectin-1/HveCTransient reversal of episome silencing precedes VP16-dependent transcription during reactivation of latent HSV-1 in neuronsHerpes simplex virus type 1 vector-mediated expression of nerve growth factor protects dorsal root ganglion neurons from peroxide toxicityEpigenetic regulation of latent HSV-1 gene expression.Induction of varicella zoster virus DNA replication in dissociated human trigeminal gangliaLong-term transgene expression in mice infected with a herpes simplex virus type 1 mutant severely impaired for immediate-early gene expression.Transactivation of herpes simplex virus type 1 immediate-early gene expression by virion-associated factors is blocked by an inhibitor of cyclin-dependent protein kinasesModeling HSV-1 Latency in Human Embryonic Stem Cell-Derived Neurons.Inducible cyclic AMP early repressor produces reactivation of latent herpes simplex virus type 1 in neurons in vitroEntry of herpes simplex virus type 1 (HSV-1) into the distal axons of trigeminal neurons favors the onset of nonproductive, silent infectionAnalysis of HSV Viral Reactivation in Explants of Sensory Neurons.Neurons differentially control expression of a herpes simplex virus type 1 immediate-early promoter in transgenic mice.Mechanisms of herpes simplex virus type 1 reactivation.Herpes simplex virus gene expression in neurons: viral DNA synthesis is a critical regulatory event in the branch point between the lytic and latent pathways.The herpes simplex virus type 1 latency-associated transcript promoter is activated through Ras and Raf by nerve growth factor and sodium butyrate in PC12 cells.The herpes simplex virus type 1 immediate-early protein ICP0 is necessary for the efficient establishment of latent infection.Characterization of a glial cell line persistently infected with borna disease virus (BDV): influence of neurotrophic factors on BDV protein and RNA expressionEvidence for a novel regulatory pathway for herpes simplex virus gene expression in trigeminal ganglion neurons.An antigen encoded by the latency-associated transcript in neuronal cell cultures latently infected with herpes simplex virus type 1.Nerve growth factor is an autocrine factor essential for the survival of macrophages infected with HIVReactivation of latent herpes simplex virus by adenovirus recombinants encoding mutant IE-0 gene products.Efficient quiescent infection of normal human diploid fibroblasts with wild-type herpes simplex virus type 1.Human herpesviruses: a consideration of the latent state.An Immortalized Human Dorsal Root Ganglion Cell Line Provides a Novel Context To Study Herpes Simplex Virus 1 Latency and Reactivation.Neurotrophic Factors NGF, GDNF and NTN Selectively Modulate HSV1 and HSV2 Lytic Infection and Reactivation in Primary Adult Sensory and Autonomic NeuronsInduction of Transcription Factor Early Growth Response Protein 1 during HSV-1 Infection Promotes Viral Replication in Corneal Cells.Herpes Simplex Virus Establishment, Maintenance, and Reactivation: In Vitro Modeling of Latency.Human corneal cells and other fibroblasts can stimulate the appearance of herpes simplex virus from quiescently infected PC12 cells.Herpes simplex virus type 1 promoter activity during latency establishment, maintenance, and reactivation in primary dorsal root neurons in vitro.Regulation and cell-type-specific activity of a promoter located upstream of the latency-associated transcript of herpes simplex virus type 1.Nature and duration of growth factor signaling through receptor tyrosine kinases regulates HSV-1 latency in neurons.The herpes simplex virus protein Vmw65 can trans-activate both viral and cellular promoters in neuronal cells.Inhibition of H3K27me3-specific histone demethylases JMJD3 and UTX blocks reactivation of herpes simplex virus 1 in trigeminal ganglion neurons.The loss of binary: Pushing the herpesvirus latency paradigm.Silencing of Viral Elements: An Available Cure for Schizophrenia?
P2860
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P2860
Characterization of nerve growth factor-dependent herpes simplex virus latency in neurons in vitro.
description
1988 nî lūn-bûn
@nan
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
1988年论文
@zh
1988年论文
@zh-cn
name
Characterization of nerve grow ...... s latency in neurons in vitro.
@en
type
label
Characterization of nerve grow ...... s latency in neurons in vitro.
@en
prefLabel
Characterization of nerve grow ...... s latency in neurons in vitro.
@en
P2860
P1433
P1476
Characterization of nerve grow ...... s latency in neurons in vitro.
@en
P2093
C L Wilcox
E M Johnson
P2860
P304
P407
P577
1988-02-01T00:00:00Z