Group B Streptococcus CovR regulation modulates host immune signalling pathways to promote vaginal colonization - Wikidata - awgldk - TriplyDB

Group B Streptococcus CovR regulation modulates host immune signalling pathways to promote vaginal colonization

Group B Streptococcus CovR regulation modulates host immune signalling pathways to promote vaginal colonization

http://www.wikidata.org/entity/Q36853324

about

Group B Streptococcus vaccine development: present status and future considerations, with emphasis on perspectives for low and middle income countries A streptococcal lipid toxin induces membrane permeabilization and pyroptosis leading to fetal injury Two novel functions of hyaluronidase from Streptococcus agalactiae are enhanced intracellular survival and inhibition of proinflammatory cytokine expression.Identification of a group B streptococcal fibronectin binding protein, SfbA, that contributes to invasion of brain endothelium and development of meningitis.Analysis of two-component systems in group B Streptococcus shows that RgfAC and the novel FspSR modulate virulence and bacterial fitness.Group B Streptococcus β-hemolysin/cytolysin breaches maternal-fetal barriers to cause preterm birth and intrauterine fetal demise in vivo Group B streptococcal serine-rich repeat proteins promote interaction with fibrinogen and vaginal colonization Interaction of Streptococcus agalactiae and Cellular Innate Immunity in Colonization and Disease Group B streptococcal haemolysin and pigment, a tale of twins.The sensor histidine kinase RgfC affects group B streptococcal virulence factor expression independent of its response regulator RgfA.Preventive effect of Lactobacillus reuteri CRL1324 on Group B Streptococcus vaginal colonization in an experimental mouse model.Streptococcus salivarius K12 Limits Group B Streptococcus Vaginal Colonization.Mast cell degranulation by a hemolytic lipid toxin decreases GBS colonization and infection Characterization of host immunity during persistent vaginal colonization by Group B Streptococcus.A novel C5a-derived immunobiotic peptide reduces Streptococcus agalactiae colonization through targeted bacterial killing.BsaB, a novel adherence factor of group B Streptococcus.Bacterial Stress Responses during Host Infection Perinatal Group B Streptococcal Infections: Virulence Factors, Immunity, and Prevention Strategies.A Murine Model of Group B Streptococcus Vaginal Colonization.PbsP, a cell wall-anchored protein that binds plasminogen to promote hematogenous dissemination of group B Streptococcus.Immunomodulation of Lactobacillus reuteri CRL1324 on Group B Streptococcus Vaginal Colonization in a Murine Experimental Model.Acid Stress Response Mechanisms of Group B Streptococci.Mast Cell Chymase Decreases The Severity Of Group B Streptococcus Infections.Quantitative assessment of the blood-brain barrier opening caused by Streptococcus agalactiae hyaluronidase in a BALB/c mouse model.A vaginal tract signal detected by the GBS SaeRS system elicits transcriptomic changes and enhances murine colonization.Protective effect of Group B Streptococcus type-III polysaccharide conjugates against maternal colonization, ascending infection and neonatal transmission in rodent models.Group B Streptococcal Colonization, Molecular Characteristics, and Epidemiology.Group B Streptococcal Maternal Colonization and Neonatal Disease: Molecular Mechanisms and Preventative Approaches.Characterization of a two-component system transcriptional regulator LtdR that impacts Group B Streptococcal colonization and disease.Stable Expression of Modified Green Fluorescent Protein in Group B Streptococci To Enable Visualization in Experimental Systems The plasminogen binding protein PbsP is required for brain invasion by hypervirulent CC17 Group B streptococci

P2860

Q28075884-633754F6-7A72-4C0D-9363-0A9AF2DCC0BF Q28258475-7A697D35-79A7-4A94-8F05-213C8AFC1C94 Q33603044-CDF4A49D-F67E-438A-8770-E6A43A7DD6FA Q33603050-DC5A14BD-394E-4180-9B4A-AEC0FD49DC09 Q33647953-93F6B8A2-BC61-4F97-9236-AB5B38565563 Q33883219-81629A21-347E-478F-8399-215373B0C821 Q34315633-A41F6E7E-D72E-4857-8FBA-390091D4686E Q34419556-EB3A65D7-35FB-4153-B482-6D1B7BB0B4DF Q35045034-22D15C82-7378-4F60-81DD-B09BA8A7D223 Q35098179-6B1A2C61-A5BD-4020-A4CC-9D6FC602DE0B Q35580560-10F121CF-8B41-404E-A42F-EA8ADB8F0798 Q35947443-A150D518-1994-4C63-A3A3-638C824053E2 Q36096819-C8D8A96E-C7C9-4E6E-B8C8-21D38CEE898F Q36137438-7204B5C3-4E10-4F9C-BFC2-67F1AD1EE9BE Q37263526-EC020711-94EE-40E4-A7F1-B8E01A6E5773 Q37643871-BE3DD57B-7E63-45A1-92D0-438ACE5091A7 Q38924600-6982B562-22EB-4D37-B261-EB2996203B17 Q39386700-952AB5FA-3298-483E-9AEA-8CD6A019EC03 Q40426394-513DE800-BC83-4F53-A32B-D2A2EF6B9694 Q40787080-CCACF230-C483-4751-A587-824F16ED4407 Q40918656-97D0BFF7-4616-433E-83DF-EC222D5B4A17 Q41311562-59D03BAA-BC85-46F2-96D7-764D33907A82 Q41997583-DB52B7F3-EE3B-4085-B980-61A56CE9C856 Q42659909-6927CA2D-47F2-409C-88AD-9669ACA48134 Q49597974-1F8EECA4-9905-4E4E-822B-09FA18BA2DAA Q50129993-337C9C11-9C58-4C8B-8B7C-5C278115B95D Q52624533-8A9F2715-40D2-4773-BCF2-37F72EA938B3 Q52665560-811FF8DC-9D55-46CF-B5A4-C9D427B1B1F0 Q52691718-64F825E7-15B8-4768-8EC4-2E4A39BE27CF Q58053827-8A3A71D4-E0C1-4445-A932-365C2EEF3AF3 Q58698635-437EA0C4-4554-4BA4-9816-A9A32FFB8841

P2860

Group B Streptococcus CovR regulation modulates host immune signalling pathways to promote vaginal colonization

http://www.wikidata.org/entity/Q36853324

description

2013 nî lūn-bûn

@nan

2013年の論文

@ja

2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

2013年论文

@zh

2013年论文

@zh-cn

name

Group B Streptococcus CovR reg ...... o promote vaginal colonization

@en

type

label

Group B Streptococcus CovR reg ...... o promote vaginal colonization

@en

prefLabel

Group B Streptococcus CovR reg ...... o promote vaginal colonization

@en

P1433

Q36853324-ED0440FF-0C03-4E06-9DB3-E48BC9F49152

P1476

Q36853324-45351CD0-9B89-4472-B1CD-91391232E570

P2093

Q36853324-081463DC-EE76-4C3A-9381-C8CB2E172CC5

Q36853324-48C6B505-05FD-4145-8F04-CEDEB684EEAC

Q36853324-7B753870-9605-463A-84B7-793A2DC542D4

Q36853324-9D807978-32F1-401C-93A2-89AC62397670

Q36853324-9FF137BC-296C-4F36-B042-CF696504BAA7

Q36853324-A7B88836-67E1-49FF-861E-8EC6785822FD

Q36853324-AAF2869C-795F-4BC0-819B-B35EFAF77C63

Q36853324-C246AB81-C1CF-45B0-B02B-65B09C8161F0

Q36853324-C471FC95-2C32-4835-B746-D02B6B1ACF9F

Q36853324-D423B98D-32D8-41B3-A9B0-9E63A3EF8161

P2860

Q36853324-013DE8BD-61CE-40AC-945B-D1F316B771A0

Q36853324-052D85EE-2E19-480D-BACE-27C1F1D08BA0

Q36853324-06EBAE42-8148-4B7E-B15D-F5A24742128F

Q36853324-084162DF-229A-4F14-B330-79DDB1F6408A

Q36853324-0955C5CB-2CE0-45D7-A2EC-6583C9946E13

Q36853324-0A153C18-B82F-45E7-AD6B-9C7E7A2C3C78

Q36853324-0E36DF63-20EA-47BD-A01E-4808810ACE1C

Q36853324-11B29CF9-B951-40DD-9162-6D88837BAFF7

Q36853324-155BB8D8-050B-4589-AF55-86F686DD912D

Q36853324-1566275E-3597-4A6D-B978-6635633C6787

P304

Q36853324-330B0932-0F06-4508-9CB5-5590E7D37928

P31

Q36853324-56ECDBC7-3012-496A-A44C-17B341B9A4E3

P356

Q36853324-62E5D69D-2D34-48CA-8AAC-3D9CF524592A

P433

Q36853324-9BF9D66B-5A46-4896-B8CF-F729049F3E7A

P478

Q36853324-BBA72CC1-50CB-4BD2-BCD4-B54A87B0FA6E

P577

Q36853324-EC0B75B0-585F-4699-9B85-849C9F275D7C

P698

Q36853324-FDF8698B-BFE7-4CEE-975C-A6342B0CF70B

P932

Q36853324-2C87E87C-FB88-4FDC-80B1-A708FC0AFA7E

P356

P1433

Cellular Microbiology

P1476

Group B Streptococcus CovR reg ...... o promote vaginal colonization

@en

P2093

Alyssa Jimenez

http://www.w3.org/2001/XMLSchema#string

Courtney K Cavaco

http://www.w3.org/2001/XMLSchema#string

Erin M Fletcher

http://www.w3.org/2001/XMLSchema#string

Joshua Fierer

http://www.w3.org/2001/XMLSchema#string

Kathryn A Patras

http://www.w3.org/2001/XMLSchema#string

Kelly S Doran

http://www.w3.org/2001/XMLSchema#string

Lakshmi Rajagopal

http://www.w3.org/2001/XMLSchema#string

Mansi Garg

http://www.w3.org/2001/XMLSchema#string

Nai-Yu Wang

http://www.w3.org/2001/XMLSchema#string

Tamsin R Sheen

http://www.w3.org/2001/XMLSchema#string

P2860

Genome-wide analysis of group a streptococci reveals a mutation that modulates global phenotype and disease specificity.

Recent advances in understanding the molecular basis of group B Streptococcus virulence

Lectin-like oxidized LDL receptor-1 is a cell-adhesion molecule involved in endotoxin-induced inflammation

CovS/CovR of group B streptococcus: a two-component global regulatory system involved in virulence.

Regulation of CovR expression in Group B Streptococcus impacts blood-brain barrier penetration.

Chemokines and chemokine receptors in leukocyte trafficking.

Blood-brain barrier invasion by group B Streptococcus depends upon proper cell-surface anchoring of lipoteichoic acid.

Anthrax toxins inhibit neutrophil signaling pathways in brain endothelium and contribute to the pathogenesis of meningitis

Removal of group B streptococci colonizing the vagina and oropharynx of mice with a bacteriophage lytic enzyme

Adherence to and invasion of human brain microvascular endothelial cells are promoted by fibrinogen-binding protein FbsA of Streptococcus agalactiae.

P304

1154-1167

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1111/CMI.12105

http://www.w3.org/2001/XMLSchema#string

P433

7

http://www.w3.org/2001/XMLSchema#string

P478

15

http://www.w3.org/2001/XMLSchema#string

P577

2013-01-30T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

23298320

http://www.w3.org/2001/XMLSchema#string

P932

3657335

http://www.w3.org/2001/XMLSchema#string