Differentiation and DNA contact points of host proteins binding at the cis site for virion-mediated induction of alpha genes of herpes simplex virus 1.
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The molecular basis of herpes simplex virus latencyThe octamer-binding proteins form multi-protein--DNA complexes with the HSV alpha TIF regulatory proteinProperties of two 5'-coterminal RNAs transcribed part way and across the S component origin of DNA synthesis of the herpes simplex virus 1 genome.Mutational analysis of the promoter region of the alpha 27 gene of herpes simplex virus 1 within the context of the viral genome.Binding of the herpes simplex virus major regulatory protein to viral DNA.Transcription factors interacting with herpes simplex virus alpha gene promoters in sensory neuronsHerpes simplex virus type 1 and bovine herpesvirus 1 latency.The number of alphaherpesvirus particles infecting axons and the axonal protein repertoire determines the outcome of neuronal infectionThe promoter and transcriptional unit of a novel herpes simplex virus 1 alpha gene are contained in, and encode a protein in frame with, the open reading frame of the alpha 22 geneDirect combinatorial interaction between a herpes simplex virus regulatory protein and a cellular octamer-binding factor mediates specific induction of virus immediate-early gene expressionMammalian alphaherpesvirus miRNAs.An 85-kilodalton herpes simplex virus type 1 alpha trans-induction factor (VP16)-VP13/14 fusion protein retains the transactivation and structural properties of the wild-type molecule during virus infection.Role of herpes simplex virus type 1 UL46 and UL47 in alpha TIF-mediated transcriptional induction: characterization of three viral deletion mutants.EBNA-2 transactivates a lymphoid-specific enhancer in the BamHI C promoter of Epstein-Barr virus.Expression of the herpes simplex virus 1 alpha transinducing factor (VP16) does not induce reactivation of latent virus or prevent the establishment of latency in mice.Role of alpha-transinducing factor (VP16) in the induction of alpha genes within the context of viral genomes.A cellular factor binds to the herpes simplex virus type 1 transactivator Vmw65 and is required for Vmw65-dependent protein-DNA complex assembly with Oct-1Activation of human papillomavirus type 18 gene expression by herpes simplex virus type 1 viral transactivators and a phorbol ester.Reactivation of latent herpes simplex virus by adenovirus recombinants encoding mutant IE-0 gene products.A herpes simplex virus type 1 mutant containing a nontransinducing Vmw65 protein establishes latent infection in vivo in the absence of viral replication and reactivates efficiently from explanted trigeminal ganglia.Specific effect of interferon on the herpes simplex virus type 1 transactivation event.Virus infections in the nervous systemConstruction and characterization of a herpes simplex virus type 1 mutant unable to transinduce immediate-early gene expressionDifferential dependence of herpes simplex virus immediate-early gene expression on de novo-infected cell protein synthesis.Overlapping octamer and TAATGARAT motifs in the VF65-response elements in herpes simplex virus immediate-early promoters represent independent binding sites for cellular nuclear factor IIIAdenovirus vector expressing functional herpes simplex virus ICP0.Structural requirements in the herpes simplex virus type 1 transactivator Vmw65 for interaction with the cellular octamer-binding protein and target TAATGARAT sequences.The 3 facets of regulation of herpes simplex virus gene expression: A critical inquiry.Identification of a promoter mapping within the reiterated sequences that flank the herpes simplex virus type 1 UL region.Deletion of the VP16 open reading frame of herpes simplex virus type 1.Separation of requirements for protein-DNA complex assembly from those for functional activity in the herpes simplex virus regulatory protein Vmw65.Direct interactions of Kaposi's sarcoma-associated herpesvirus/human herpesvirus 8 ORF50/Rta protein with the cellular protein octamer-1 and DNA are critical for specifying transactivation of a delayed-early promoter and stimulating viral reactivatiThe C-terminal 79 amino acids of the herpes simplex virus regulatory protein, Vmw65, efficiently activate transcription in yeast and mammalian cells in chimeric DNA-binding proteins.
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P2860
Differentiation and DNA contact points of host proteins binding at the cis site for virion-mediated induction of alpha genes of herpes simplex virus 1.
description
1988 nî lūn-bûn
@nan
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
1988年论文
@zh
1988年论文
@zh-cn
name
Differentiation and DNA contac ...... nes of herpes simplex virus 1.
@en
type
label
Differentiation and DNA contac ...... nes of herpes simplex virus 1.
@en
prefLabel
Differentiation and DNA contac ...... nes of herpes simplex virus 1.
@en
P2860
P1433
P1476
Differentiation and DNA contac ...... nes of herpes simplex virus 1.
@en
P2093
T M Kristie
P2860
P304
P407
P577
1988-04-01T00:00:00Z