LPS decreases fatty acid oxidation and nuclear hormone receptors in the kidney.
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Therapeutic Effects of Procainamide on Endotoxin-Induced Rhabdomyolysis in RatsGene network and pathway analysis of bovine mammary tissue challenged with Streptococcus uberis reveals induction of cell proliferation and inhibition of PPARgamma signaling as potential mechanism for the negative relationships between immune responPostprandial lysophospholipid suppresses hepatic fatty acid oxidation: the molecular link between group 1B phospholipase A2 and diet-induced obesity.Integrative "omic" analysis of experimental bacteremia identifies a metabolic signature that distinguishes human sepsis from systemic inflammatory response syndromesDynamic molecular and histopathological changes in the extracellular matrix and inflammation in the transition to heart failure in isolated volume overloadToll-like receptor-mediated inflammatory signaling reprograms cardiac energy metabolism by repressing peroxisome proliferator-activated receptor γ coactivator-1 signaling.PGC-1α promotes recovery after acute kidney injury during systemic inflammation in mice.Inhibition of c-Jun-N-terminal kinase increases cardiac peroxisome proliferator-activated receptor alpha expression and fatty acid oxidation and prevents lipopolysaccharide-induced heart dysfunction.MicroRNA-155 Deficiency Attenuates Liver Steatosis and Fibrosis without Reducing Inflammation in a Mouse Model of Steatohepatitis.Mitochondrial function and disturbances in the septic kidneyLipopolysaccharide significantly influences the hepatic triglyceride metabolism in growing pigs.The Tm7sf2 Gene Deficiency Protects Mice against Endotoxin-Induced Acute Kidney Injury.Effect of lipopolysaccharides on adipogenic potential and premature senescence of adipocyte progenitors.FGF21 is increased by inflammatory stimuli and protects leptin-deficient ob/ob mice from the toxicity of sepsisTargeted metabolomics for discrimination of systemic inflammatory disorders in critically ill patientsEffects of Tributyrin on Intestinal Energy Status, Antioxidative Capacity and Immune Response to Lipopolysaccharide Challenge in Broilers.Molecular mechanisms responsible for the reduced expression of cholesterol transporters from macrophages by low-dose endotoxinEffects of the PPAR-β/δ agonist GW0742 during resuscitated porcine septic shock.Nuclear control of the inflammatory response in mammals by peroxisome proliferator-activated receptorsInfection decreases fatty acid oxidation and nuclear hormone receptors in the diaphragmIRAK-1 contributes to lipopolysaccharide-induced reactive oxygen species generation in macrophages by inducing NOX-1 transcription and Rac1 activation and suppressing the expression of antioxidative enzymes.An integrated clinico-metabolomic model improves prediction of death in sepsis.Altered renal lipid metabolism and renal lipid accumulation in human diabetic nephropathy.Nonthyroidal illness and the cardiorenal syndrome.Acute rhabdomyolysis and inflammation.Structure, function and disease relevance of Omega-class glutathione transferases.Fuzhisan, a chinese herbal medicine, suppresses beta-secretase gene transcription via upregulation of SIRT1 expression in N2a-APP695 cells.Molecular mechanism underlying the suppression of lipid oxidation during endotoxemia.Inflammatory stress promotes the development of obesity-related chronic kidney disease via CD36 in mice.Toll-like Receptor (TLR) Signaling Interacts with CREBH to Modulate High-density Lipoprotein (HDL) in Response to Bacterial Endotoxin.Inflammation inhibits the expression of phosphoenolpyruvate carboxykinase in liver and adipose tissue.Toll-like receptor-4 (TLR4) down-regulates microRNA-107, increasing macrophage adhesion via cyclin-dependent kinase 6.Gene expression-based in vivo and in vitro prediction of liver toxicity allows compound selection at an early stage of drug development.Transcriptional control of enterohepatic lipid regulatory targets in response to early cholesterol and phytosterol exposure in apoE-/- mice.PGC1α in the kidney.Effect of Maternal Obesity on Placental Lipid Metabolism.Argan oil prevents down-regulation induced by endotoxin on liver fatty acid oxidation and gluconeogenesis and on peroxisome proliferator-activated receptor gamma coactivator-1α, (PGC-1α), peroxisome proliferator-activated receptor α (PPARα) and estrCREBH Regulates Systemic Glucose and Lipid Metabolism.Pivotal Roles of Peroxisome Proliferator-Activated Receptors (PPARs) and Their Signal Cascade for Cellular and Whole-Body Energy Homeostasis.Association between 4-year all-cause mortality and carnitine profile in maintenance hemodialysis patients
P2860
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P2860
LPS decreases fatty acid oxidation and nuclear hormone receptors in the kidney.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
LPS decreases fatty acid oxidation and nuclear hormone receptors in the kidney.
@en
type
label
LPS decreases fatty acid oxidation and nuclear hormone receptors in the kidney.
@en
prefLabel
LPS decreases fatty acid oxidation and nuclear hormone receptors in the kidney.
@en
P2093
P2860
P1476
LPS decreases fatty acid oxidation and nuclear hormone receptors in the kidney.
@en
P2093
Arthur Moser
Carl Grunfeld
Judy K Shigenaga
Kenneth R Feingold
Yuwei Wang
P2860
P304
P356
10.1194/JLR.M800233-JLR200
P577
2008-06-23T00:00:00Z