High-dose but not low-dose mainstream cigarette smoke suppresses allergic airway inflammation by inhibiting T cell function
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The triterpenoid CDDO-Me inhibits bleomycin-induced lung inflammation and fibrosisToxic elements in tobacco and in cigarette smoke: inflammation and sensitizationMouse models to unravel the role of inhaled pollutants on allergic sensitization and airway inflammationCigarette smoke exposure exacerbates lung inflammation and compromises immunity to bacterial infection.Inhalation exposure to nanosized and fine TiO2 particles inhibits features of allergic asthma in a murine model.Exposure to cigarette smoke inhibits the pulmonary T-cell response to influenza virus and Mycobacterium tuberculosis.Exposure to cigarette smoke impacts myeloid-derived regulatory cell function and exacerbates airway hyper-responsivenessA novel anti-inflammatory and pro-resolving role for resolvin D1 in acute cigarette smoke-induced lung inflammationCigarette smoke exacerbates mouse allergic asthma through Smad proteins expressed in mast cells.Substance p regulates environmental tobacco smoke-enhanced tracheal smooth muscle responsiveness in mice.Inhalation of the reactive aldehyde acrolein promotes antigen sensitization to ovalbumin and enhances neutrophilic inflammation.Perturbation of cellular immune functions in cigarette smokers and protection by palm oil vitamin E supplementationAndrographolide protects against cigarette smoke-induced oxidative lung injury via augmentation of Nrf2 activity.Tobacco use increases susceptibility to bacterial infection.Acrolein exposure suppresses antigen-induced pulmonary inflammation.The mode of lymphoblastoid cell death in response to gas phase cigarette smoke is dose-dependent.Cigarette Smoke Increases CD8α+ Dendritic Cells in an Ovalbumin-Induced Airway Inflammation.Acrolein and thiol-reactive electrophiles suppress allergen-induced innate airway epithelial responses by inhibition of DUOX1 and EGFREndogenous ligands of the aryl hydrocarbon receptor regulate lung dendritic cell function.Effects of conventional tobacco smoke and nicotine-free cigarette smoke on airway inflammation, airway remodelling and lung function in a triple allergen model of severe asthma.Secondhand Smoke Induces Inflammation and Impairs Immunity to Respiratory Infections.Modelling the asthma phenotype: impact of cigarette smoke exposure.
P2860
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P2860
High-dose but not low-dose mainstream cigarette smoke suppresses allergic airway inflammation by inhibiting T cell function
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
High-dose but not low-dose mai ...... by inhibiting T cell function
@en
type
label
High-dose but not low-dose mai ...... by inhibiting T cell function
@en
prefLabel
High-dose but not low-dose mai ...... by inhibiting T cell function
@en
P2093
P2860
P1476
High-dose but not low-dose mai ...... by inhibiting T cell function
@en
P2093
Patricia J Sime
Randi P Benson
Richard P Phipps
P2860
P304
P356
10.1152/AJPLUNG.00392.2007
P577
2008-06-20T00:00:00Z