about
The triterpenoid CDDO-Me inhibits bleomycin-induced lung inflammation and fibrosisResolvin D1 attenuates polyinosinic-polycytidylic acid-induced inflammatory signaling in human airway epithelial cells via TAK1Specialized proresolving mediators (SPMs) inhibit human B-cell IgE productionNeu-164 and Neu-107, two novel antioxidant and anti-myeloperoxidase compounds, inhibit acute cigarette smoke-induced lung inflammationCigarette smoke exposure exacerbates lung inflammation and compromises immunity to bacterial infection.PPAR-γ ligands repress TGFβ-induced myofibroblast differentiation by targeting the PI3K/Akt pathway: implications for therapy of fibrosis.Inhibition of transglutaminase 2, a novel target for pulmonary fibrosis, by two small electrophilic molecules.A novel anti-inflammatory and pro-resolving role for resolvin D1 in acute cigarette smoke-induced lung inflammationLung-targeted overexpression of the NF-κB member RelB inhibits cigarette smoke-induced inflammationElectrophilic PPARγ Ligands Attenuate IL-1β and Silica-Induced Inflammatory Mediator Production in Human Lung Fibroblasts via a PPARγ-Independent Mechanism.Transglutaminase 2 and its role in pulmonary fibrosis.Thy1 (CD90) controls adipogenesis by regulating activity of the Src family kinase, FynNormal Human Lung Epithelial Cells Inhibit Transforming Growth Factor-β Induced Myofibroblast Differentiation via Prostaglandin E2Aryl hydrocarbon receptor-deficient mice develop heightened inflammatory responses to cigarette smoke and endotoxin associated with rapid loss of the nuclear factor-kappaB component RelB.The Role of PPARs in Lung Fibrosis.Emerging PPARγ-Independent Role of PPARγ Ligands in Lung Diseases.Ionizing radiation induces myofibroblast differentiation via lactate dehydrogenaseResolvins attenuate inflammation and promote resolution in cigarette smoke-exposed human macrophages.Pharmacologic inhibition of lactate production prevents myofibroblast differentiation.Peroxisome proliferator-activated receptor γ B cell-specific-deficient mice have an impaired antibody response.Resolvin D1 Reduces Emphysema and Chronic Inflammation.Resolvin D1 Dampens Pulmonary Inflammation and Promotes Clearance of Nontypeable Haemophilus influenzaeRole of CXCR2 in cigarette smoke-induced lung inflammation.High-dose but not low-dose mainstream cigarette smoke suppresses allergic airway inflammation by inhibiting T cell functionAttenuation of inflammatory mediator production by the NF-κB member RelB is mediated by microRNA-146a in lung fibroblasts.Electrophilic peroxisome proliferator-activated receptor-gamma ligands have potent antifibrotic effects in human lung fibroblasts.Dung biomass smoke activates inflammatory signaling pathways in human small airway epithelial cells.Human lung fibroblasts produce proresolving peroxisome proliferator-activated receptor-γ ligands in a cyclooxygenase-2-dependent manner.Crystalline and amorphous silica differentially regulate the cyclooxygenase-prostaglandin pathway in pulmonary fibroblasts: implications for pulmonary fibrosis.PPARgamma agonists inhibit TGF-beta induced pulmonary myofibroblast differentiation and collagen production: implications for therapy of lung fibrosis.Cigarette Smoke Dampens Anti-viral Signaling in Small Airway Epithelial Cells by Disrupting TLR3 Cleavage.Endogenous ligands of the aryl hydrocarbon receptor regulate lung dendritic cell function.Sensitivity to bleomycin-induced lung injury is not moderated by an antigen-limited T-cell repertoire.Autologous T-lymphocytes stimulate proliferation of orbital fibroblasts derived from patients with Graves' ophthalmopathy.Secondhand Smoke Induces Inflammation and Impairs Immunity to Respiratory Infections.Activated Human Lung Fibroblasts Produce Extracellular Vesicles with Anti-Fibrotic ProstaglandinsLipoxin B Enhances Human Memory B Cell Antibody Production via Upregulating Cyclooxygenase-2 ExpressionIncreasing the frequency of T-cell precursors specific for a cryptic epitope of hen-egg lysozyme converts it to an immunodominant epitopeBacterial protease treatment of natural rubber latex alters its primary immunogenicity in a mouse model of sensitizationOropharyngeal aspiration of a silica suspension produces a superior model of silicosis in the mouse when compared to intratracheal instillation
P50
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P50
description
hulumtues
@sq
researcher
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wetenschapper
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հետազոտող
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name
Thomas Thatcher
@ast
Thomas Thatcher
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Thomas Thatcher
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Thomas Thatcher
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Thomas Thatcher
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Томас Тэтчер
@ru
type
label
Thomas Thatcher
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Thomas Thatcher
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Thomas Thatcher
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Thomas Thatcher
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Thomas Thatcher
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Томас Тэтчер
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prefLabel
Thomas Thatcher
@ast
Thomas Thatcher
@en
Thomas Thatcher
@es
Thomas Thatcher
@nl
Thomas Thatcher
@sl
Томас Тэтчер
@ru
P106
P1153
7003794881
P21
P31
P496
0000-0001-7177-3041