Mechanism of recovery from acute virus infection: treatment of lymphocytic choriomeningitis virus-infected mice with monoclonal antibodies reveals that Lyt-2+ T lymphocytes mediate clearance of virus and regulate the antiviral antibody response.
about
Impaired antibody response causes persistence of prototypic T cell-contained virusSecondary rearrangements and hypermutation generate sufficient B cell diversity to mount protective antiviral immunoglobulin responses.Dynamic regulation of functionally distinct virus-specific T cells.Enhanced virus clearance by early inducible lymphocytic choriomeningitis virus-neutralizing antibodies in immunoglobulin-transgenic miceCD40 ligand-mediated interactions are involved in the generation of memory CD8(+) cytotoxic T lymphocytes (CTL) but are not required for the maintenance of CTL memory following virus infection.Herpes simplex virus type 1-specific cytotoxic T-lymphocyte arming occurs within lymph nodes draining the site of cutaneous infectionAdditive effect of neutralizing antibody and antiviral drug treatment in preventing virus escape and persistence.Virus-induced delayed-type hypersensitivity reaction is sequentially mediated by CD8+ and CD4+ T lymphocytes.Host factors influencing viral persistence.Role of T lymphocyte subsets in the pathogenesis of primary infection and rechallenge with respiratory syncytial virus in mice.T cells in the central nervous system: the delicate balance between viral clearance and diseaseThe virus-encoded chemokine vMIP-II inhibits virus-induced Tc1-driven inflammationExogenous interleukin-12 protects against lethal infection with coxsackievirus B4.Antiviral CD4 and CD8 T-cell memory: differences in the size of the response and activation requirements.An intermediate dose of LCMV clone 13 causes prolonged morbidity that is maintained by CD4+ T cells.Host response to Sendai virus in mice lacking class II major histocompatibility complex glycoproteins.Bone marrow is a major site of long-term antibody production after acute viral infectionImmunobiology of cytotoxic T-cell escape mutants of lymphocytic choriomeningitis virusDNA vaccination against persistent viral infection.Antiviral protective immunity induced by major histocompatibility complex class I molecule-restricted viral T-lymphocyte epitopes inserted in various positions in immunologically self and nonself proteins.CD40 ligand-deficient mice generate a normal primary cytotoxic T-lymphocyte response but a defective humoral response to a viral infection.Uncovering subdominant cytotoxic T-lymphocyte responses in lymphocytic choriomeningitis virus-infected BALB/c mice.Virus-specific, CD8+ major histocompatibility complex class I-restricted cytotoxic T lymphocytes in lymphocytic choriomeningitis virus-infected beta2-microglobulin-deficient mice.Immune response against lymphocytic choriomeningitis virus infection in mice without CD8 expression.Early antibodies specific for the neutralizing epitope on the receptor binding subunit of the lymphocytic choriomeningitis virus glycoprotein fail to neutralize the virus.Genetic basis of viral persistence: single amino acid change in the viral glycoprotein affects ability of lymphocytic choriomeningitis virus to persist in adult mice.CD8 T cell memory in B cell-deficient mice.Two roads diverged: interferon alpha/beta- and interleukin 12-mediated pathways in promoting T cell interferon gamma responses during viral infectionCharacterization of CD8+ T cell function and immunodominance generated with an H2O2-inactivated whole-virus vaccine.Novel plant virus-based vaccine induces protective cytotoxic T-lymphocyte-mediated antiviral immunity through dendritic cell maturation.Enhanced establishment of a virus carrier state in adult CD4+ T-cell-deficient mice.CD4+ T cells are required to sustain CD8+ cytotoxic T-cell responses during chronic viral infection.Antiviral immune responses of lymphocytic choriomeningitis virus-infected mice lacking CD8+ T lymphocytes because of disruption of the beta 2-microglobulin gene.Cross-protection against lymphocytic choriomeningitis virus mediated by a CD4+ T-cell clone specific for an envelope glycoprotein epitope of Lassa virus.Central neuropathogenesis of vesicular stomatitis virus infection of immunodeficient miceAntiviral immune responses in gene-targeted mice expressing the immunoglobulin heavy chain of virus-neutralizing antibodiesAntiviral antibodies attenuate T-cell-mediated immunopathology following acute lymphocytic choriomeningitis virus infectionSuppression of virus-specific antibody production by CD8+ class I-restricted antiviral cytotoxic T cells in vivo.Efficacious control of cytomegalovirus infection after long-term depletion of CD8+ T lymphocytes.Trojan horse lymphocytes: a vesicular stomatitis virus-specific T-cell clone lyses target cells by carrying virus.
P2860
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P2860
Mechanism of recovery from acute virus infection: treatment of lymphocytic choriomeningitis virus-infected mice with monoclonal antibodies reveals that Lyt-2+ T lymphocytes mediate clearance of virus and regulate the antiviral antibody response.
description
1987 nî lūn-bûn
@nan
1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
1987年论文
@zh
1987年论文
@zh-cn
name
Mechanism of recovery from acu ...... e antiviral antibody response.
@en
type
label
Mechanism of recovery from acu ...... e antiviral antibody response.
@en
prefLabel
Mechanism of recovery from acu ...... e antiviral antibody response.
@en
P2093
P2860
P1433
P1476
Mechanism of recovery from acu ...... e antiviral antibody response.
@en
P2093
Cobbold SP
Lehmann-Grube F
Moskophidis D
P2860
P304
P407
P577
1987-06-01T00:00:00Z