GSK-3beta in mouse fibroblasts controls wound healing and fibrosis through an endothelin-1-dependent mechanism - Wikidata - awgldk - TriplyDB

GSK-3beta in mouse fibroblasts controls wound healing and fibrosis through an endothelin-1-dependent mechanism

GSK-3beta in mouse fibroblasts controls wound healing and fibrosis through an endothelin-1-dependent mechanism

http://www.wikidata.org/entity/Q36890830

about

Signaling in Fibrosis: TGF-β, WNT, and YAP/TAZ Converge The GSK-3 family as therapeutic target for myocardial diseases Fibroblasts in fibrosis: novel roles and mediators Cutaneous wound healing: recruiting developmental pathways for regeneration Topical insulin accelerates wound healing in diabetes by enhancing the AKT and ERK pathways: a double-blind placebo-controlled clinical trial Rib fractures and death from deletion of osteoblast βcatenin in adult mice is rescued by corticosteroids A proteomics approach to identifying key protein targets involved in VEGF inhibitor mediated attenuation of bleomycin-induced pulmonary fibrosis Endoglin haploinsufficiency promotes fibroblast accumulation during wound healing through Akt activation Beneficial compaction of spinal cord lesion by migrating astrocytes through glycogen synthase kinase-3 inhibition.ROCK1 functions as a suppressor of inflammatory cell migration by regulating PTEN phosphorylation and stability.PI3K-dependent GSK3ß(Ser9)-phosphorylation is implicated in the intestinal epithelial cell wound-healing response.The canonical Wnt signaling activator, R-spondin2, regulates craniofacial patterning and morphogenesis within the branchial arch through ectodermal-mesenchymal interaction Wnt1/βcatenin injury response activates the epicardium and cardiac fibroblasts to promote cardiac repair.The Effect of Control-released Basic Fibroblast Growth Factor in Wound Healing: Histological Analyses and Clinical Application.The four-herb Chinese medicine ANBP enhances wound healing and inhibits scar formation via bidirectional regulation of transformation growth factor pathway.Ha-Ras stabilization mediates pro-fibrotic signals in dermal fibroblasts.Wnt and Notch signaling pathway involved in wound healing by targeting c-Myc and Hes1 separately.Angiotensin Converting Enzyme Regulates Cell Proliferation and Migration.Biochemical changes induced by grape seed extract and low level laser therapy administration during intraoral wound healing in rat liver: an experimental and in silico study.Direct action of endothelin-1 on podocytes promotes diabetic glomerulosclerosis Scar management in burn injuries using drug delivery and molecular signaling: Current treatments and future directions.Secreted frizzled related proteins inhibit fibrosis in vitro but appear redundant in vivo.β-catenin and Smad3 regulate the activity and stability of myocardin-related transcription factor during epithelial-myofibroblast transition.Evaluation of intracellular signalling pathways in response to insulin-like growth factor I in apoptotic-resistant activated human hepatic stellate cells.Wound-healing properties of nut oil from Pouteria lucuma.Loss of GSK-3 Causes Abnormal Astrogenesis and Behavior in Mice.Bothered and bewildered, but not bewitched.Fibroblast-Specific Deletion of Hypoxia Inducible Factor-1 Critically Impairs Murine Cutaneous Neovascularization and Wound Healing.

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P2860

GSK-3beta in mouse fibroblasts controls wound healing and fibrosis through an endothelin-1-dependent mechanism

http://www.wikidata.org/entity/Q36890830

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

@zh-mo

2008年論文

@zh-tw

2008年论文

@wuu

2008年论文

@zh

2008年论文

@zh-cn

name

GSK-3beta in mouse fibroblasts ...... dothelin-1-dependent mechanism

@en

type

label

GSK-3beta in mouse fibroblasts ...... dothelin-1-dependent mechanism

@en

prefLabel

GSK-3beta in mouse fibroblasts ...... dothelin-1-dependent mechanism

@en

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Journal of Clinical Investigation

P1476

GSK-3beta in mouse fibroblasts ...... dothelin-1-dependent mechanism

@en

P2093

Andrew Leask

http://www.w3.org/2001/XMLSchema#string

Christopher P Denton

http://www.w3.org/2001/XMLSchema#string

David J Abraham

http://www.w3.org/2001/XMLSchema#string

James R Woodgett

http://www.w3.org/2001/XMLSchema#string

Kun Huh

http://www.w3.org/2001/XMLSchema#string

Matthew McCann

http://www.w3.org/2001/XMLSchema#string

Mohit Kapoor

http://www.w3.org/2001/XMLSchema#string

Shangxi Liu

http://www.w3.org/2001/XMLSchema#string

Xu Shi-wen

http://www.w3.org/2001/XMLSchema#string

P2860

GSK-3: tricks of the trade for a multi-tasking kinase

Requirement for glycogen synthase kinase-3beta in cell survival and NF-kappaB activation

Functional interaction of beta-catenin with the transcription factor LEF-1

beta-catenin is a target for the ubiquitin-proteasome pathway

Wound healing--aiming for perfect skin regeneration

Convergence of Wnt, beta-catenin, and cadherin pathways

Genetic deficiency of glycogen synthase kinase-3beta corrects diabetes in mouse models of insulin resistance

Interactions of fibroblasts with the extracellular matrix: implications for the understanding of fibrosis.

beta-Catenin stabilization dysregulates mesenchymal cell proliferation, motility, and invasiveness and causes aggressive fibromatosis and hyperplastic cutaneous wounds

The Akt pathway: molecular targets for anti-cancer drug development.

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3279-3290

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scholarly article

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10.1172/JCI35381

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10

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118

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2008-10-01T00:00:00Z

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18802478

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2542848

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