Interaction between cardiac sympathetic afferent reflex and chemoreflex is mediated by the NTS AT1 receptors in heart failure - Wikidata - awgldk - TriplyDB

Interaction between cardiac sympathetic afferent reflex and chemoreflex is mediated by the NTS AT1 receptors in heart failure

Interaction between cardiac sympathetic afferent reflex and chemoreflex is mediated by the NTS AT1 receptors in heart failure

http://www.wikidata.org/entity/Q36893410

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Angiotensin II-superoxide-NFκB signaling and aortic baroreceptor dysfunction in chronic heart failure Autonomic, locomotor and cardiac abnormalities in a mouse model of muscular dystrophy: targeting the renin-angiotensin system Temporal profile and mechanisms of the prompt sympathoexcitation following coronary ligation in Wistar rats Evolution and physiology of neural oxygen sensing Inhibition of cardiac sympathetic afferent reflex and sympathetic activity by baroreceptor and vagal afferent inputs in chronic heart failure Exercise training prevents the exaggerated exercise pressor reflex in rats with chronic heart failure.Angiotensin II-induced upregulation of AT(1) receptor expression: sequential activation of NF-kappaB and Elk-1 in neurons.Role of apoptosis signal-regulating kinase-1-c-Jun NH2-terminal kinase-p38 signaling in voltage-gated K+ channel remodeling of the failing heart: regulation by thioredoxin.Effects of combined aging and heart failure on visceral sympathetic nerve and cardiovascular responses to progressive hyperthermia in F344 rats.Cardiovascular responses elicited by a new endogenous angiotensin in the nucleus tractus solitarius of the rat.Disruption of cardiovascular circadian rhythms in mice post myocardial infarction: relationship with central angiotensin II receptor expression.Endogenous reactive oxygen species modulates voltage-gated sodium channels in dorsal root ganglia of rats.Angiotensin peptides and central autonomic regulation Central angiotensin (1-7) enhances baroreflex gain in conscious rabbits with heart failure Neurohumoral stimulation.Angiotensin receptors alter myocardial infarction-induced remodeling of the guinea pig cardiac plexus Responses of Nucleus Tractus Solitarius (NTS) early and late neurons to blood pressure changes in anesthetized F344 rats.Effect of angiotensin II on voltage-gated sodium currents in aortic baroreceptor neurons and arterial baroreflex sensitivity in heart failure rats Relative contributions of the thalamus and the paraventricular nucleus of the hypothalamus to the cardiac sympathetic afferent reflex Pharmacological treatment for heart failure: a view from the brain.Cardiac autonomic neural remodeling and susceptibility to sudden cardiac death: effect of endurance exercise training.Dynamic remodeling of the guinea pig intrinsic cardiac plexus induced by chronic myocardial infarction.Contribution of Autonomic Reflexes to the Hyperadrenergic State in Heart Failure Central nervous system circuits modified in heart failure: pathophysiology and therapeutic implications.Cardiac sympathetic afferent reflex and its implications for sympathetic activation in chronic heart failure and hypertension.Brain mechanisms of sympathetic activation in heart failure: Roles of the renin‑angiotensin system, nitric oxide and pro‑inflammatory cytokines (Review)Translational neurocardiology: preclinical models and cardioneural integrative aspects.Cardiac sympathetic afferent reflex control of cardiac function in normal and chronic heart failure states.NF-κB and CREB are required for angiotensin II type 1 receptor upregulation in neurons.Influence of brain-derived neurotrophic factor-tyrosine receptor kinase B signalling in the nucleus tractus solitarius on baroreflex sensitivity in rats with chronic heart failure.Parallel changes in neuronal AT1R and GRK5 expression following exercise training in heart failure.H₂O₂ induces delayed hyperexcitability in nucleus tractus solitarii neurons.Alteration in skeletal muscle afferents in rats with chronic heart failure.Oxidative stress in carotid body contributes to enhanced chemoreflex in heart failure: focus on "Elevated mitochondrial superoxide contributes to enhanced chemoreflex in heart failure rabbits".Glutathione homeostasis in ventricular myocytes from rat hearts with chronic myocardial infarction.Experimental Evidences Supporting the Benefits of Exercise Training in Heart Failure.Role of hypothalamic angiotensin type 1 receptors in pressure overload-induced mineralocorticoid receptor activation and salt-induced sympathoexcitation.Exercise training attenuates the pressor response evoked by peripheral chemoreflex in rats with heart failure.Dysregulation of the Renin-Angiotensin System and the Vasopressinergic System Interactions in Cardiovascular Disorders.

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Interaction between cardiac sympathetic afferent reflex and chemoreflex is mediated by the NTS AT1 receptors in heart failure

http://www.wikidata.org/entity/Q36893410

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2008 nî lūn-bûn

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2008年论文

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Interaction between cardiac sy ...... AT1 receptors in heart failure

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Interaction between cardiac sy ...... AT1 receptors in heart failure

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American Journal of Physiology - Heart and Circulatory Physiology

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Interaction between cardiac sy ...... AT1 receptors in heart failure

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Han-Jun Wang

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Irving H Zucker

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Lie Gao

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Wei Wang

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Wei-Zhong Wang

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P2860

The nucleus of the solitary tract: an integrating station for nociceptive and cardiorespiratory afferents.

The regulation of sympathetic outflow in heart failure. The roles of angiotensin II, nitric oxide, and exercise training.

Novel mechanisms of sympatho-excitation in chronic heart failure.

The origin of sympathetic outflow in heart failure: the roles of angiotensin II and nitric oxide.

Differential effects of cardiac sympathetic afferent stimulation on neurons in the nucleus tractus solitarius.

Reflex cardiovascular control in congestive heart failure.

Novel mechanisms of sympathetic regulation in chronic heart failure.

The relationship of the sympathetic nervous system and the renin-angiotensin system in congestive heart failure.

Annual review prize lecture. Central nervous mechanisms contributing to cardiovascular control.

Sensory innervation of the viscera: peripheral basis of visceral pain.

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