Pharmacological treatment for heart failure: a view from the brain. - Wikidata - awgldk - TriplyDB

Pharmacological treatment for heart failure: a view from the brain.

Pharmacological treatment for heart failure: a view from the brain.

http://www.wikidata.org/entity/Q37530428

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Autonomic-Immune-Vascular Interaction: An Emerging Concept for Neurogenic Hypertension Brain inflammation and hypertension: the chicken or the egg?Endogenous angiotensin II-induced p44/42 mitogen-activated protein kinase activation mediates sodium appetite but not thirst or neurohypophysial secretion in male rats.Centrally administered lipopolysaccharide elicits sympathetic excitation via NAD(P)H oxidase-dependent mitogen-activated protein kinase signaling.Mineralocorticoid receptors in the brain and cardiovascular regulation: minority rule?Central regulation of blood pressure by the mineralocorticoid receptor EP₃ receptors mediate PGE₂-induced hypothalamic paraventricular nucleus excitation and sympathetic activation.Peroxisome proliferator-activated receptor-γ regulates inflammation and renin-angiotensin system activity in the hypothalamic paraventricular nucleus and ameliorates peripheral manifestations of heart failure.Aldosterone-induced brain MAPK signaling and sympathetic excitation are angiotensin II type-1 receptor dependent Vasopressin and oxytocin in control of the cardiovascular system Neuroinflammatory and autonomic mechanisms in diabetes and hypertension.Central nervous system circuits modified in heart failure: pathophysiology and therapeutic implications.Brain mechanisms of sympathetic activation in heart failure: Roles of the renin‑angiotensin system, nitric oxide and pro‑inflammatory cytokines (Review)Hypothalamic and inflammatory basis of hypertension.Brain perivascular macrophages and the sympathetic response to inflammation in rats after myocardial infarction.In vivo bioluminescence imaging reveals redox-regulated activator protein-1 activation in paraventricular nucleus of mice with renovascular hypertension.Central mechanisms of abnormal sympathoexcitation in chronic heart failure.Inhibition of brain angiotensin III attenuates sympathetic hyperactivity and cardiac dysfunction in rats post-myocardial infarction.Oral simvastatin reduces the hypertensive response to air-jet stress.SOD1 overexpression in paraventricular nucleus improves post-infarct myocardial remodeling and ventricular function.The cardiovascular actions of fractalkine/CX3CL1 in the hypothalamic paraventricular nucleus are attenuated in rats with heart failure.

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Pharmacological treatment for heart failure: a view from the brain.

http://www.wikidata.org/entity/Q37530428

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 24 June 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Pharmacological treatment for heart failure: a view from the brain.

@en

Pharmacological treatment for heart failure: a view from the brain.

@nl

type

label

Pharmacological treatment for heart failure: a view from the brain.

@en

Pharmacological treatment for heart failure: a view from the brain.

@nl

prefLabel

Pharmacological treatment for heart failure: a view from the brain.

@en

Pharmacological treatment for heart failure: a view from the brain.

@nl

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Clinical Pharmacology & Therapeutics

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Pharmacological treatment for heart failure: a view from the brain.

@en

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R B Felder

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S-G Wei

http://www.w3.org/2001/XMLSchema#string

Y Yu

http://www.w3.org/2001/XMLSchema#string

Z-H Zhang

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P2860

Nanotechnology approaches to crossing the blood-brain barrier and drug delivery to the CNS

Cardiovascular and renal sympathetic activation by blood-borne TNF-alpha in rat: the role of central prostaglandins

Mitogen-activated protein kinases mediate upregulation of hypothalamic angiotensin II type 1 receptors in heart failure rats

Prostaglandins as inflammatory messengers across the blood-brain barrier.

Transvascular delivery of small interfering RNA to the central nervous system.

Signaling the brain in systemic inflammation: the role of perivascular cells.

The new biology of aldosterone.

Inhibition of brain proinflammatory cytokine synthesis reduces hypothalamic excitation in rats with ischemia-induced heart failure.

Interaction between cardiac sympathetic afferent reflex and chemoreflex is mediated by the NTS AT1 receptors in heart failure

Cytokine blockade attenuates sympathoexcitation in heart failure: cross-talk between nNOS, AT-1R and cytokines in the hypothalamic paraventricular nucleus.

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216-220

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scholarly article

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10.1038/CLPT.2009.117

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2

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86

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2009-06-24T00:00:00Z

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1000786663

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19553933

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2786214

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