A noninverting genome of a viable herpes simplex virus 1: presence of head-to-tail linkages in packaged genomes and requirements for circularization after infection
about
The molecular biology of human herpesvirus-6 latency and telomere integrationThe herpes simplex virus type 1 alkaline nuclease and single-stranded DNA binding protein mediate strand exchange in vitroMacrophages and cytokines in the early defence against herpes simplex virusCloning human herpes virus 6A genome into bacterial artificial chromosomes and study of DNA replication intermediatesRecruitment of cellular recombination and repair proteins to sites of herpes simplex virus type 1 DNA replication is dependent on the composition of viral proteins within prereplicative sites and correlates with the induction of the DNA damage respoInhibition of B virus (Macacine herpesvirus 1) by conventional and experimental antiviral compounds.Herpes simplex virus genome isomerization: origins of adjacent long segments in concatemeric viral DNA.In vitro processing of herpes simplex virus type 1 DNA replication intermediates by the viral alkaline nuclease, UL12Construction and properties of a viable herpes simplex virus 1 recombinant lacking coding sequences of the alpha 47 geneAn early regulatory function required in a cell type-dependent manner is expressed by the genomic but not the cDNA copy of the herpes simplex virus 1 gene encoding infected cell protein 0.Properties of an equine herpesvirus 1 mutant devoid of the internal inverted repeat sequence of the genomic short region.Recombination promoted by DNA viruses: phage λ to herpes simplex virus.Evidence that the immediate-early gene product ICP4 is necessary for the genome of the herpes simplex virus type 1 ICP4 deletion mutant strain d120 to circularize in infected cellsReplication of the herpes simplex virus genome: does it really go around in circles?Relationship of herpes simplex virus genome configuration to productive and persistent infections.Role of ICP0 in the strategy of conquest of the host cell by herpes simplex virus 1The a sequence is dispensable for isomerization of the herpes simplex virus type 1 genome.Cathepsin B mediates cleavage of herpes simplex virus type 1 origin binding protein (OBP) to yield OBPC-1, and cleavage is dependent upon viral DNA replication.Gamma-herpesvirus kinase actively initiates a DNA damage response by inducing phosphorylation of H2AX to foster viral replicationIsolation of a herpes simplex virus cDNA encoding the DNA repair enzyme uracil-DNA glycosylaseRequirement for double-strand breaks but not for specific DNA sequences in herpes simplex virus type 1 genome isomerization events.Immediate-early transcription over covalently joined genome ends of bovine herpesvirus 1: the circ gene.Herpes simplex virus type 1 latency-associated transcript (LAT) promoter deletion mutants can express a 2-kilobase transcript mapping to the LAT region.Construction and properties of a recombinant herpes simplex virus 1 lacking both S-component origins of DNA synthesis.Herpes simplex virus type 1 mutant strain in1814 establishes a unique, slowly progressing infection in SCID mice.The herpes simplex virus type 1 (HSV-1) a sequence serves as a cleavage/packaging signal but does not drive recombinational genome isomerization when it is inserted into the HSV-2 genome.Expression of the herpes simplex virus 1 alpha transinducing factor (VP16) does not induce reactivation of latent virus or prevent the establishment of latency in mice.Herpes simplex virus origin-binding protein (UL9) loops and distorts the viral replication originCharacterization of DNA sequence-common and sequence-specific proteins binding to cis-acting sites for cleavage of the terminal a sequence of the herpes simplex virus 1 genome.Visualizing the replicating HSV-1 virus using STED super-resolution microscopy.Sequence requirements for DNA rearrangements induced by the terminal repeat of herpes simplex virus type 1 KOS DNARecombinogenic properties of herpes simplex virus type 1 DNA sequences resident in simian virus 40 minichromosomes.A herpes simplex virus type 1 mutant containing a nontransinducing Vmw65 protein establishes latent infection in vivo in the absence of viral replication and reactivates efficiently from explanted trigeminal ganglia.Entry of herpes simplex virus 1 in BJ cells that constitutively express viral glycoprotein D is by endocytosis and results in degradation of the virusInversion and circularization of the varicella-zoster virus genomeHerpes simplex virus amplicon: cleavage of concatemeric DNA is linked to packaging and involves amplification of the terminally reiterated a sequence.Generation of an inverting herpes simplex virus 1 mutant lacking the L-S junction a sequences, an origin of DNA synthesis, and several genes including those specifying glycoprotein E and the alpha 47 geneHerpes simplex virus 1 recombinants with noninverting genomes frozen in different isomeric arrangements are capable of independent replicationFunctional domains within the a sequence involved in the cleavage-packaging of herpes simplex virus DNA.Characterization of major recognition sequences for a herpes simplex virus type 1 origin-binding protein.
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P2860
A noninverting genome of a viable herpes simplex virus 1: presence of head-to-tail linkages in packaged genomes and requirements for circularization after infection
description
1985 nî lūn-bûn
@nan
1985年の論文
@ja
1985年論文
@yue
1985年論文
@zh-hant
1985年論文
@zh-hk
1985年論文
@zh-mo
1985年論文
@zh-tw
1985年论文
@wuu
1985年论文
@zh
1985年论文
@zh-cn
name
A noninverting genome of a via ...... ircularization after infection
@en
type
label
A noninverting genome of a via ...... ircularization after infection
@en
prefLabel
A noninverting genome of a via ...... ircularization after infection
@en
P2860
P1433
P1476
A noninverting genome of a via ...... ircularization after infection
@en
P2093
K L Poffenberger
P2860
P304
P407
P577
1985-02-01T00:00:00Z