Murine Borrelia arthritis is highly dependent on ASC and caspase-1, but independent of NLRP3.
about
Mechanisms of Borrelia burgdorferi internalization and intracellular innate immune signalingNLRP3 inflammasome mediates albumin-induced renal tubular injury through impaired mitochondrial function.Innate immunity networks during infection with Borrelia burgdorferi.Activation of autophagy and nucleotide-binding domain leucine-rich repeat-containing-like receptor family, pyrin domain-containing 3 inflammasome during Leishmania infantum-associated glomerulonephritisγδ T Cells and dendritic cells in refractory Lyme arthritis.NLRP3 inflammasome and host protection against bacterial infectionMouse and pig models for studies of natural and vaccine-induced immunity to Bordetella pertussis.Deviant Behavior: Tick-Borne Pathogens and Inflammasome Signaling.
P2860
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P2860
Murine Borrelia arthritis is highly dependent on ASC and caspase-1, but independent of NLRP3.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Murine Borrelia arthritis is h ...... e-1, but independent of NLRP3.
@en
type
label
Murine Borrelia arthritis is h ...... e-1, but independent of NLRP3.
@en
prefLabel
Murine Borrelia arthritis is h ...... e-1, but independent of NLRP3.
@en
P2093
P2860
P50
P356
P1476
Murine Borrelia arthritis is h ...... se-1, but independent of NLRP3
@en
P2093
Frank L van de Veerdonk
Ineke Verschueren
Jos W M van der Meer
Kathrin Buffen
Leo A B Joosten
Marije Oosting
Mihai G Netea
Patrick Sturm
P2860
P2888
P356
10.1186/AR4090
P577
2012-11-13T00:00:00Z